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by Prins, B.P and Abbasi, A and Wong, A and Vaez, Ahmad and Nolte, Ilja and Franceschini, Nora and Stuart, P.E and Guterriez Achury, J and Mistry, V and Bradfield, Jonathan and Valdes, Ana Maria and Bras, Jose and Shatunov, Aleksey and Lu, Chao and Han, B and Raychaudhuri, Soumya and Bevan, Steve and Mayes, M.D and Tsoi, L.C and Evangelou, Evangelos and Nair, Rajan P and Grant, Struan and Polychronakos, Constantin and Radstake, Timothy and Heel, David and Dunstan, M.L and Wood, Nicholas and Al-Chalabi, Ammar and Dehghan, Abbas and Hakonarson, Hakon and Markus, H.S and Elder, James and Knight, J and Arking, Dan and Spector, Timothy and Koeleman, Bobby and Duijn, Cornelia and Martín, Javier and Morris, Anew and Weersma, Rinse K and Wijmenga, Cisca and Munroe, Patricia and Perry, John and Pouget, J.G and Jamshidi, Yalda and Snieder, Harold and Alizadeh, Behrooz and Inflammation Working Grp CHARGE Co and Autism Spectrum Disorder Working G and PAGE Consortium and Int Consortium Blood Pressure and Int Parkinson Dis Genomics Consort and Schizophrenia Working Grp Psychiat and DIAGRAM Consortium and CARDIoGRAMplusC4d Consortium and Int Stroke Genetics Consortium and Treat OA Consortium and ALS Consortium and CKDGen Consortium and Systemic Sclerosis Consortium and GERAD1 Consortium and International Consortium for Blood Pressure and International Stroke Genetics Consortium and Treat OA consortium and ALS consortium and CKDGen consortium and Schizophrenia Working Group of the Psychiatric Genomics Consortium and Systemic Sclerosis consortium and Inflammation Working Group of the CHARGE Consortium and International Parkinson’s Disease Genomics Consortium and CARDIoGRAMplusC4D Consortium and Autism Spectrum Disorder Working Group of the Psychiatric Genomics Consortium
PLoS medicine, ISSN 1549-1277, 06/2016, Volume 13, Issue 6, p. e1001976
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 3, p. e60454
Journal Article
Nature genetics, ISSN 1061-4036, 2017, Volume 49, Issue 9, pp. 1392 - 1397
UK Biobank is among the world's largest repositories for phenotypic and genotypic information in individuals of European ancestry1. We performed a genome-wide... 
PROVIDES INSIGHTS | METAANALYSIS | GENETICS & HEREDITY | NUCLEOTIDE EXCHANGE FACTOR | SUSCEPTIBILITY LOCI | ARCHITECTURE | RISK | TRAITS | VARIANT | GENOME-WIDE ASSOCIATION | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Leukocyte Rolling - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Middle Aged | Cells, Cultured | Genotype | Logistic Models | Male | United Kingdom | Rho Guanine Nucleotide Exchange Factors - genetics | Health Information Systems | Carrier Proteins - genetics | Phenotype | Coronary Artery Disease - genetics | Coronary Artery Disease - pathology | HEK293 Cells | Insulin Resistance - genetics | Adult | Female | Aged | Polymorphism, Single Nucleotide | Transendothelial and Transepithelial Migration - genetics | Complications and side effects | Development and progression | Insulin resistance | Genetic aspects | Disease susceptibility | Research | Coronary heart disease | Repositories | Leukocyte migration | Nucleotide sequence | Coronary artery | Genomes | Leukocytes | Insulin | Coronary artery disease | Disease control | Loci | Disease resistance | Pathways | Genetic analysis | DNA methylation | Gene loci | Mutation | Heart diseases | Deoxyribonucleic acid--DNA | coronary artery disease | population genetics | gene-expression | genome-wide association studies
Journal Article
PloS one, ISSN 1932-6203, 08/2017, Volume 12, Issue 8, p. e0182999
textabstractGlatiramer acetate is used therapeutically in multiple sclerosis but also known for adverse effects including elevated coronary artery disease... 
CELLS | GROWTH-FACTOR-BETA | ACTIVATION | TGF-BETA | MULTIPLE-SCLEROSIS | MECHANISM | MULTIDISCIPLINARY SCIENCES | ATHEROSCLEROSIS | RECEPTORS | EXPRESSION | ASSOCIATION | Genetic Predisposition to Disease | Glatiramer Acetate - adverse effects | Humans | Cardiovascular Diseases - chemically induced | Polymorphism, Single Nucleotide | Cardiovascular Diseases - genetics | Case-Control Studies | Linkage Disequilibrium | Glatiramer acetate | Complications and side effects | Multiple sclerosis | Analysis | Genetic aspects | Dosage and administration | Research | Drug therapy | Health risk assessment | Coronary heart disease | Risk factors | Chromosome 19 | Drugs | Copolymer 1 | Heart attacks | Genes | Linkage disequilibrium | Risk | Cardiovascular disease | Genomes | Single-nucleotide polymorphism | Epidemiology | Consortia | Blood platelets | Atherosclerosis | Genetics | Cardiology | Heart diseases | Growth factors | Hypertension | Antigens | Transforming growth factor-b1 | Coronary artery | Health risks | Gene expression | Coronary artery disease | Studies | Side effects | Hospitals | Coronary vessels | In vivo methods and tests | Cardiovascular diseases | In vitro methods and tests | Medicinsk genetik | Basic Medicine | Medical Genetics | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Journal Article
Journal Article
by Nikpay, Majid and Goel, Anuj and Won, Hong-Hee and Hall, Leanne M and Willenborg, Christina and Kanoni, Stavroula and Saleheen, Danish and Kyriakou, Theodosios and Nelson, Christopher P and Hopewell, Jemma C and Webb, Thomas R and Zeng, Lingyao and Dehghan, Abbas and Alver, Maris and Armasu, Sebastian M and Auro, Kirsi and Bjonnes, Andrew and Chasman, Daniel I and Chen, Shufeng and Ford, Ian and Franceschini, Nora and Gieger, Christian and Grace, Christopher and Gustafsson, Stefan and Huang, Jie and Hwang, Shih-Jen and Kim, Yun Kyoung and Kleber, Marcus E and Lau, King Wai and Lu, Xiangfeng and Lu, Yingchang and Lyytikäinen, Leo-Pekka and Mihailov, Evelin and Morrison, Alanna C and Pervjakova, Natalia and Qu, Liming and Rose, Lynda M and Salfati, Elias and Saxena, Richa and Scholz, Markus and Smith, Albert V and Tikkanen, Emmi and Uitterlinden, Andre and Yang, Xueli and Zhang, Weihua and Zhao, Wei and de Andrade, Mariza and de Vries, Paul S and van Zuydam, Natalie R and Anand, Sonia S and Bertram, Lars and Beutner, Frank and Dedoussis, George and Frossard, Philippe and Gauguier, Dominique and Goodall, Alison H and Gottesman, Omri and Haber, Marc and Han, Bok-Ghee and Huang, Jianfeng and Jalilzadeh, Shapour and Kessler, Thorsten and König, Inke R and Lannfelt, Lars and Lieb, Wolfgang and Lind, Lars and Lindgren, Cecilia M and Lokki, Marja-Liisa and Magnusson, Patrik K and Mallick, Nadeem H and Mehra, Narinder and Meitinger, Thomas and Memon, Fazal-Ur-Rehman and Morris, Andrew P and Nieminen, Markku S and Pedersen, Nancy L and Peters, Annette and Rallidis, Loukianos S and Rasheed, Asif and Samuel, Maria and Shah, Svati H and Sinisalo, Juha and Stirrups, Kathleen E and Trompet, Stella and Wang, Laiyuan and Zaman, Khan S and Ardissino, Diego and Boerwinkle, Eric and Borecki, Ingrid B and Bottinger, Erwin P and Buring, Julie E and Chambers, John C and Collins, Rory and Cupples, L Adrienne and Danesh, John and Demuth, Ilja and Elosua, Roberto and Epstein, Stephen E and Esko, Tõnu and Feitosa, Mary F and ...
Nature genetics, ISSN 1546-1718, 10/2015, Volume 47, Issue 10, p. 1121
Existing knowledge of genetic variants affecting risk of coronary artery disease (CAD) is largely based on genome-wide association study (GWAS) analysis of... 
Genome-Wide Association Study | Phenotype | Genome, Human | Humans | Coronary Artery Disease - genetics
Journal Article
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