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biochemistry & molecular biology (8) 8
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cell survival - drug effects (3) 3
cells, cultured (3) 3
diabetic retinopathy (3) 3
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dose-response relationship, drug (3) 3
expression (3) 3
in vivo (3) 3
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stem cells (3) 3
toxicology (3) 3
activation (2) 2
antibodies (2) 2
antineoplastic agents - pharmacology (2) 2
antisense rna (2) 2
apoptosis (2) 2
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arsenic compounds (2) 2
bronchi - cytology (2) 2
cell biology (2) 2
cell line (2) 2
cell proliferation - genetics (2) 2
diabetes mellitus (2) 2
dna damage (2) 2
dna damages (2) 2
dna microarrays (2) 2
endothelium (2) 2
epithelial cells - drug effects (2) 2
epithelial cells - metabolism (2) 2
epithelial-mesenchymal transition (2) 2
epithelial-mesenchymal transition - drug effects (2) 2
fluorescent antibody technique (2) 2
gene expression regulation (2) 2
genes (2) 2
genetic research (2) 2
growth (2) 2
hypoxia-inducible factor 1, alpha subunit - metabolism (2) 2
long noncoding rna (2) 2
long noncoding rnas (2) 2
mechanisms (2) 2
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pathology (2) 2
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reverse transcriptase polymerase chain reaction (2) 2
secretion (2) 2
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surgery (2) 2
transcription (2) 2
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a549 cells (1) 1
acid sensing ion channels (1) 1
acid-sensing ion channels (1) 1
actin (1) 1
adaptor proteins, signal transducing - antagonists & inhibitors (1) 1
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Journal Article
Journal Article
Experimental Cell Research, ISSN 0014-4827, 02/2015, Volume 331, Issue 1, pp. 115 - 122
In this study, we determined the effects of transforming growth factor-beta (TGF-β) and Wnt/β-catenin signaling on myofibroblast differentiation of NIH/3T3... 
Wnt signaling pathway | NSC668036 | Fibroblast | Idiopathic pulmonary fibrosis | Myofibroblast | Writ signaling pathway | CATENIN | BLEOMYCIN | PROLIFERATION | MESENCHYMAL STEM-CELLS | FIBROBLASTS | CELL BIOLOGY | INVASION | ONCOLOGY | PATHWAY | WNT | MYOFIBROBLAST DIFFERENTIATION | PULMONARY-FIBROSIS | NIH 3T3 Cells | Antibiotics, Antineoplastic - toxicity | Cell Proliferation | Cadherins - metabolism | Male | Depsipeptides - pharmacology | Phosphoproteins - antagonists & inhibitors | Phosphoproteins - metabolism | Immunoenzyme Techniques | Flow Cytometry | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | Pulmonary Fibrosis - metabolism | Cadherins - genetics | Dishevelled Proteins | PDZ Domains - drug effects | Real-Time Polymerase Chain Reaction | Transforming Growth Factor beta1 - pharmacology | Bleomycin - toxicity | Fibroblasts - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Cells, Cultured | Phosphoproteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | beta Catenin - metabolism | Blotting, Western | beta Catenin - genetics | Animals | Signal Transduction - drug effects | Fibroblasts - drug effects | Adaptor Proteins, Signal Transducing - genetics | Fluorescent Antibody Technique | Pulmonary Fibrosis - prevention & control | Pulmonary Fibrosis - chemically induced | Fibroblasts - cytology | Mice | Adaptor Proteins, Signal Transducing - metabolism | Bone morphogenetic proteins | Respiratory tract diseases | Muscle proteins | Transforming growth factors | Collagen | Analysis | Signal transduction | Transcription factors | Cellular biology | Lung diseases | FIBROSIS | MIGRATION | COLLAGEN | STIMULATION | ACTIN | BUILDUP | LUNGS | TRANSCRIPTION | SIGNALS | IN VIVO | 60 APPLIED LIFE SCIENCES | GROWTH FACTORS | DISEASES | INHIBITION | IN VITRO | GENES | MUSCLES
Journal Article
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 10/2015, Volume 465, Issue 4, pp. 678 - 684
Ocular angiogenesis is an important pathologic character of several ocular diseases, such as retinopathy of prematurity, diabetic retinopathy and age-related... 
Anti-proliferative | Lenalidomide | Ocular angiogenesis | Anti-inflammatory | DIABETIC-RETINOPATHY | SURGERY | ANGIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MITOMYCIN | THALIDOMIDE | THERAPY | BIOPHYSICS | MYELOMA | Diabetic Retinopathy - drug therapy | Humans | Macaca mulatta | Vascular Endothelial Growth Factor A - metabolism | Diabetic Retinopathy - pathology | Thalidomide - pharmacology | Retinal Vessels - pathology | Thalidomide - analogs & derivatives | Choroidal Neovascularization - drug therapy | Choroidal Neovascularization - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Inflammation Mediators - metabolism | Antineoplastic Agents - pharmacology | Endothelial Cells - physiology | Cell Line | Cell Survival - drug effects | Angiogenesis Inhibitors - pharmacology | Retinal Vessels - drug effects | Cell Movement - drug effects | Animals | Retinal Vessels - physiopathology | Mice | Retinal Neovascularization - drug therapy | Endothelial Cells - pathology | Retinal Neovascularization - pathology | Endothelial Cells - drug effects | Macular degeneration | Diabetic retinopathy | Medical colleges | Angiogenesis inhibitors | Drug therapy | Vascular endothelial growth factor | Tumors | Endothelium | INHIBITION | NEOPLASMS | IN VITRO | INFLAMMATION | DRUGS | VIABILITY | CELL PROLIFERATION | RETINA | IN VIVO | PROTEINS | 60 APPLIED LIFE SCIENCES
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 01/2013, Volume 266, Issue 2, pp. 187 - 197
The biphasic effects of arsenite, in which low levels of arsenite induce cell proliferation and high levels of arsenite induce DNA damage and apoptosis,... 
Biphasic effects | Extracellular signal-regulated kinases (ERK) | Hypoxia inducible factor-1α | Hypoxia inducible factor-2α | Arsenite | c-Jun N-terminal kinases (JNK) | C-Jun N-terminal kinases (JNK) | MOLECULAR MECHANISMS | DNA-DAMAGE | PROLIFERATION | SODIUM ARSENITE | HYPOXIA | NEOPLASTIC TRANSFORMATION | HUMAN KERATINOCYTES | INDUCIBLE FACTOR (HIF)-1-ALPHA | Hypoxia inducible factor-1 alpha | PHARMACOLOGY & PHARMACY | TOXICOLOGY | NF-KAPPA-B | EXPRESSION | Hypoxia inducible factor-2 alpha | DNA Damage - drug effects | Epithelial Cells - metabolism | Apoptosis - drug effects | Epithelial Cells - drug effects | Humans | Cells, Cultured | JNK Mitogen-Activated Protein Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Down-Regulation - drug effects | Sodium Compounds - administration & dosage | Bronchi - drug effects | Dose-Response Relationship, Drug | Arsenites - administration & dosage | Basic Helix-Loop-Helix Transcription Factors - metabolism | Signal Transduction - drug effects | Sodium Compounds - toxicity | Arsenites - toxicity | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Bronchi - metabolism | Cell Proliferation - drug effects | Bronchi - cytology | Cell proliferation | Epithelial cells | DNA damage | Hypoxia-inducible factor 1 alpha | Extracellular signal-regulated kinase | Data processing | Carcinogenesis | Degradation | Signal transduction | c-Jun amino-terminal kinase | DNA | Apoptosis | Index Medicus | APOPTOSIS | CARCINOGENESIS | ANOXIA | CELL PROLIFERATION | PHOSPHOTRANSFERASES | 60 APPLIED LIFE SCIENCES | CONCENTRATION RATIO | DNA DAMAGES
Journal Article
Journal Article
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 09/2017, Volume 491, Issue 2, pp. 388 - 395
Bones are inflexible yet ever-changing metabolic organs, and bone homeostasis is maintained through two delicately regulated processes: bone construction and... 
Pilose antler peptide | NF-κB | Osteoclasts | Inflammation | Osteoblasts | NF-kappa B | BIOPHYSICS | BIOCHEMISTRY & MOLECULAR BIOLOGY | NECROSIS-FACTOR-ALPHA | TNF-ALPHA | OSTEOPOROSIS | BONE-RESORPTION | Bone Morphogenetic Protein 2 - pharmacology | Tumor Necrosis Factor-alpha - metabolism | Transcription Factor RelA - antagonists & inhibitors | Tumor Necrosis Factor-alpha - genetics | Alkaline Phosphatase - metabolism | Anti-Inflammatory Agents, Non-Steroidal - isolation & purification | Osteoclasts - cytology | Transcription Factor RelA - genetics | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Dose-Response Relationship, Drug | Bone Density Conservation Agents - isolation & purification | Osteoblasts - cytology | Osteogenesis - genetics | Cell Survival - drug effects | Alkaline Phosphatase - genetics | Signal Transduction | Mice, Inbred C57BL | Osteoblasts - drug effects | Osteogenesis - drug effects | Gene Expression Regulation | Antlers - chemistry | Deer | Bone Density Conservation Agents - pharmacology | Peptides - pharmacology | Bone Resorption - prevention & control | Peptides - isolation & purification | Animals | Transcription Factor RelA - metabolism | Anthraquinones | Cell Differentiation - drug effects | Mice | Primary Cell Culture | Osteoblasts - metabolism | Osteoclasts - drug effects | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus | HOMEOSTASIS | NEOPLASMS | 60 APPLIED LIFE SCIENCES | SKELETON | DEER | RHEUMATIC DISEASES | ANTLERS | INHIBITION | NECROSIS | PEPTIDES | METABOLISM | IN VITRO | INFLAMMATION | GENES | MINERALIZATION | ALIZARIN | MICE | CONNECTIVE TISSUE CELLS
Journal Article
理论物理通讯:英文版, ISSN 0253-6102, 2007, Volume 48, Issue 7, pp. 107 - 111
<正> The spatial chaos of Bose-Einstein condensates in a cigar-shaped trap is studied.For a system with asteady current,we construct the general solution... 
Melnikov | condensates | chaos | Bose-Einstein | function | Chaos | Bose-Einstein condensates | Melnikov function | SOLITONS | PHYSICS, MULTIDISCIPLINARY
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 05/2008, Volume 369, Issue 2, p. 471
Chronic low-grade inflammation characterized by adipose tissue macrophage accumulation and abnormal cytokine production is a key feature of obesity and type 2... 
Genetic research | Anti-inflammatory drugs | Tumor necrosis factor | Analysis | Resveratrol | MACROPHAGES | TRANSCRIPTION | MONOCYTES | PATHOLOGY | 60 APPLIED LIFE SCIENCES | INHIBITION | LYMPHOKINES | PROMOTERS | DNA | INFLAMMATION | GENE REGULATION | METABOLIC DISEASES | SECRETION | ADIPOSE TISSUE
Journal Article