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Cell (Cambridge), ISSN 0092-8674, 2010, Volume 140, Issue 2, pp. 197 - 208
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2011, Volume 108, Issue 1, pp. 308 - 313
Journal Article
Journal Article
Nature (London), ISSN 1476-4687, 2012, Volume 491, Issue 7423, pp. 254 - 258
...(5), referred to as 'tumour-elicited inflammation'(6). Although infiltrating CD4(+) T(H)1 cells and CD8(+) cytotoxic T cells constitute a positive prognostic sign in colorectal cancer... 
COLITIS | PROMOTES COLON TUMORIGENESIS | COLORECTAL-CANCER | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | HELPER-CELLS | MOUSE MODEL | APC(MIN/+) MICE | RECEPTOR | DIFFERENTIATION | CUTTING EDGE | Inflammation - pathology | Adenoma - genetics | Colorectal Neoplasms - genetics | Humans | Colitis - complications | Interleukin-17 - immunology | Interleukin-23 - deficiency | Tumor Microenvironment | Adenoma - immunology | Adenoma - microbiology | Genes, APC | Interleukin-23 - genetics | Myeloid Cells - immunology | Cell Division | Myeloid Differentiation Factor 88 - immunology | Toll-Like Receptors - metabolism | Disease Models, Animal | Bacteria - metabolism | Inflammation - microbiology | Signal Transduction | Mice, Inbred C57BL | Toll-Like Receptors - immunology | Interleukin-17 - genetics | Colorectal Neoplasms - microbiology | Inflammation - immunology | beta Catenin - metabolism | Interleukin-23 - immunology | Disease-Free Survival | Animals | Colorectal Neoplasms - immunology | Adenoma - pathology | Inflammation - genetics | Myeloid Cells - metabolism | Bacteria - pathogenicity | Mice | Cell Transformation, Neoplastic - pathology | Colorectal Neoplasms - pathology | Myeloid Differentiation Factor 88 - metabolism | Development and progression | Cellular signal transduction | Research | Interleukins | Properties | Tumors
Journal Article
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2012, Volume 143, Issue 3, pp. 765 - 776.e3
Background & Aims Interleukin (IL)-17 signaling has been implicated in lung and skin fibrosis. We examined the role of IL-17 signaling in the pathogenesis of... 
Gastroenterology and Hepatology | Immune Response | Myofibroblast | Bone Marrow-Derived Macrophages | Mouse Model | RAT-LIVER | TARGET | IL-17 RECEPTOR | HEPATOCYTES | FAMILY-MEMBERS | STAT3 | TRANSDUCTION | PATHWAY | DISEASE | GENE-EXPRESSION | GASTROENTEROLOGY & HEPATOLOGY | Kupffer Cells - pathology | Liver - pathology | Carbon Tetrachloride | Humans | Transforming Growth Factor beta1 - metabolism | Hepatic Stellate Cells - metabolism | Receptors, Interleukin-17 - genetics | Interleukin-23 - genetics | Receptors, Interleukin-17 - deficiency | Interleukins - genetics | Liver Cirrhosis, Experimental - prevention & control | Liver - immunology | Time Factors | Bone Marrow Transplantation | Liver Cirrhosis, Alcoholic - immunology | Interleukin-6 - metabolism | STAT3 Transcription Factor - genetics | Interleukin-17 - administration & dosage | Liver Cirrhosis, Experimental - immunology | Signal Transduction | Liver Cirrhosis, Alcoholic - pathology | Liver - metabolism | Interleukin-17 - genetics | Genotype | Bile Ducts - surgery | Disease Progression | Mice, Knockout | Interleukin-17 - metabolism | Phenotype | Interleukins - deficiency | Mice | Liver Cirrhosis, Experimental - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation Mediators - administration & dosage | Liver Cirrhosis, Experimental - etiology | Interleukin-23 - deficiency | Ligation | Hepatic Stellate Cells - immunology | Inflammation Mediators - metabolism | STAT3 Transcription Factor - deficiency | Liver Cirrhosis, Experimental - pathology | Kupffer Cells - metabolism | Hepatic Stellate Cells - pathology | Cell Line | Interleukin-1 - metabolism | Collagen Type I - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Interleukin-17 - deficiency | Liver Cirrhosis, Experimental - genetics | Animals | Interleukins - administration & dosage | Kupffer Cells - immunology | Medical colleges | Liver diseases | Liver | Carbon tetrachloride | Transforming growth factors | Muscle proteins | Macrophages | Endothelium | Messenger RNA | Interleukins | Actin | Analysis | Collagen | Fibrosis | Liver cirrhosis | Index Medicus | Abridged Index Medicus
Journal Article
PLoS Neglected Tropical Diseases, ISSN 1935-2727, 05/2017, Volume 11, Issue 5, p. e0005595
Journal Article
Gastroenterology, ISSN 0016-5085, 2010, Volume 139, Issue 3, pp. 987 - 998
Background & Aims Chronic injury changes the fate of certain cellular populations, inducing epithelial cells to generate fibroblasts by... 
Gastroenterology and Hepatology | EMT | MET | Mesenchymal-to-Epithelial Transition | RAT-LIVER | CELLS | HEPATOCYTES | FIBROGENESIS | HEPATIC-FIBROSIS | ORGANS | DISEASE | FIBROBLASTS DERIVE | GASTROENTEROLOGY & HEPATOLOGY | CONTRIBUTES | Immunohistochemistry | Glial Fibrillary Acidic Protein - genetics | Liver - pathology | S100 Proteins | Epithelial Cells - metabolism | S100 Calcium-Binding Protein A4 | Carbon Tetrachloride | Cell Transdifferentiation - genetics | Luminescent Proteins - biosynthesis | Liver Regeneration - genetics | Ligation | Liver Cirrhosis - metabolism | Chemical and Drug Induced Liver Injury - pathology | Liver Cirrhosis - genetics | Genes, Reporter | Hepatic Stellate Cells - pathology | Disease Models, Animal | Fibroblasts - metabolism | Biomarkers - metabolism | Promoter Regions, Genetic | Liver - metabolism | RNA, Untranslated | Epithelial Cells - pathology | Mice, Transgenic | Bile Ducts - surgery | Chemical and Drug Induced Liver Injury - genetics | Fibroblasts - pathology | Collagen Type II - genetics | Keratin-19 - genetics | Proteins - genetics | Cell Lineage | Animals | Chemical and Drug Induced Liver Injury - metabolism | Liver Cirrhosis - pathology | Luminescent Proteins - genetics | Mice | Calcium-Binding Proteins - genetics | Crosses, Genetic | Keratin | Liver diseases | Developmental biology | Actin | Collagen | Liver | Stem cells | Fibrosis | Genetic research | Fluorescence | Muscle proteins | Liver fibrosis | cholangiocytes
Journal Article
Hepatology (Baltimore, Md.), ISSN 0270-9139, 2011, Volume 53, Issue 5, pp. 1730 - 1741
Nicotinamide adenine dinucleotide phosphate oxidase (NOX) is a multicomponent enzyme that mediates electron transfer from nicotinamide adenine dinucleotide... 
Liver cirrhosis | Rodents | hepatic stellate cells | hepatic fibrosis | NADPH oxidase | oxidative stress
Journal Article