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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2009, Volume 106, Issue 34, pp. 14385 - 14390
Journal Article
New England Journal of Medicine, ISSN 0028-4793, 05/2010, Volume 362, Issue 20, p. 1943
Journal Article
Kidney International, ISSN 0085-2538, 05/2013, Volume 83, Issue 5, pp. 785 - 787
A recent clinical trial in patients with chronic kidney disease (CKD) and diabetic nephropathy demonstrated that bardoxolone methyl (CDDO-ME) increases... 
UROLOGY & NEPHROLOGY | NRF2 | KIDNEY | BARDOXOLONE METHYL | Cell Shape - drug effects | Angiotensin II - pharmacology | Glomerular Filtration Rate - drug effects | Animals | Humans | Oleanolic Acid - analogs & derivatives | Oleanolic Acid - pharmacology | Male | Mesangial Cells - drug effects | Index Medicus
Journal Article
by Fuchsberger, Christian and Flannick, Jason and Teslovich, Tanya M and Mahajan, Anubha and Agarwala, Vineeta and Gaulton, Kyle J and Ma, Clement and Fontanillas, Pierre and Moutsianas, Loukas and McCarthy, Davis J and Rivas, Manuel A and Perry, John R. B and Sim, Xueling and Blackwell, Thomas W and Robertson, Neil R and Rayner, N. William and Cingolani, Pablo and Locke, Adam E and Fernandez Tajes, Juan and Highland, Heather M and Dupuis, Josee and Chines, Peter S and Lindgren, Cecilia M and Hartl, Christopher and Jackson, Anne U and Chen, Han and Huyghe, Jeroen R and van de Bunt, Martijn and Pearson, Richard D and Kumar, Ashish and Müller-Nurasyid, Martina and Grarup, Niels and Stringham, Heather M and Gamazon, Eric R and Lee, Jaehoon and Chen, Yuhui and Scott, Robert A and Below, Jennifer E and Chen, Peng and Huang, Jinyan and Go, Min Jin and Stitzel, Michael L and Pasko, Dorota and Parker, Stephen C. J and Varga, Tibor V and Green, Todd and Beer, Nicola L and Day-Williams, Aaron G and Ferreira, Teresa and Fingerlin, Tasha and Horikoshi, Momoko and Hu, Cheng and Huh, Iksoo and Ikram, Mohammad Kamran and Kim, Bong-Jo and Kim, Yongkang and Kim, Young Jin and Kwon, Min-Seok and Lee, Juyoung and Lee, Selyeong and Lin, Keng-Han and Maxwell, Taylor J and Nagai, Yoshihiko and Wang, Xu and Welch, Ryan P and Yoon, Joon and Zhang, Weihua and Barzilai, Nir and Voight, Benjamin F and Han, Bok-Ghee and Jenkinson, Christopher P and Kuulasmaa, Teemu and Kuusisto, Johanna and Manning, Alisa and Ng, Maggie C. Y and Palmer, Nicholette D and Balkau, Beverley and Stancáková, Alena and Abboud, Hanna E and Boeing, Heiner and Gieaitis, Vilmantas and Prabhakaran, Dorairaj and Gottesman, Omri and Scott, James and Carey, Jason and Kwan, Phoenix and Grant, George and Smith, Joshua D and Neale, Benjamin M and Purcell, Shaun and Butterworth, Adam S and Howson, Joanna M. M and Lee, Heung Man and Lu, Yingchang and Kwak, Soo-Heon and Zhao, Wei and Danesh, John and Lam, Vincent K. L and Park, Kyong Soo and Saleheen, Danish and ... and Medicinska fakulteten and Institutionen för folkhälsa och klinisk medicin and Allmänmedicin and Umeå universitet and Medicin
Nature, ISSN 0028-0836, 2016, Volume 536, Issue 7614, pp. 41 - +
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 01/2010, Volume 21, Issue 1, pp. 93 - 102
TGF-β1 expression closely associates with activation and conversion of fibroblasts to a myofibroblast phenotype and synthesis of an alternatively spliced... 
Basic Research
Journal Article
Journal Article
Journal Article
Cancer Science, ISSN 1347-9032, 08/2016, Volume 107, Issue 8, pp. 1092 - 1100
Reactive oxygen species ( ROS ) are an important endogenous source of DNA damage and oxidative stress in all cell types. Deficiency in tuberin resulted in... 
tumorigenesis | Kidney | renal cells | tumor suppressor gene | reactive oxygen species | Reactive oxygen species | Tumorigenesis | Renal cells | Tumor suppressor gene | NADPH-OXIDASE | SUPEROXIDE | ACTIVATION | OXIDATIVE DNA-DAMAGE | DIABETIC-NEPHROPATHY | ONCOLOGY | ENDOTHELIAL-CELLS | PROSTATE-CANCER | DISEASE | NOX4 | EXPRESSION | Reactive Oxygen Species - metabolism | TOR Serine-Threonine Kinases - metabolism | Kidney Cortex - metabolism | Humans | NADPH Oxidases - metabolism | Male | RNA, Messenger - metabolism | TOR Serine-Threonine Kinases - antagonists & inhibitors | Tumor Suppressor Proteins - deficiency | NADPH Oxidases - genetics | Fibroblasts - metabolism | Kidney Tubules, Proximal - cytology | Tumor Suppressor Proteins - metabolism | RNA, Messenger - genetics | Cells, Cultured | Rats | NADPH Oxidase 1 | Animals | Tuberous Sclerosis Complex 2 Protein | Tuberous Sclerosis - metabolism | Embryo, Mammalian - cytology | Kidney Tubules, Proximal - metabolism | Fibroblasts - cytology | Mice | Kidney Cortex - enzymology | Oxidases | Tuberous sclerosis | RNA | Protein kinases | Cell culture | Oxidative stress | Protein kinase C | DNA damage | Kinases | NAD(P)H oxidase | Nox protein | Sclerosis | Proteins | Cell growth | NOX4 protein | CYBB protein | Fibroblasts | Deoxyribonucleic acid--DNA | Enzymes | Oxygen | Rapamycin | Embryo fibroblasts | Gene expression | Embryos | Studies | Tuberin | Bisindolylmaleimide I | Isoforms | Protein expression | Mutation | Prostate | Renal cortex | Binding sites | Cancer | Index Medicus | Original
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 02/2012, Volume 302, Issue 3, pp. C597 - C604
Maalouf RM, Eid AA, Gorin YC, Block K, Escobar GP, Bailey S, Abboud HE. Nox4-derived reactive oxygen species mediate cardiomyocyte injury in early type 1... 
Cardiac injury | NADPH oxidases | Oxidative stress | NADPH OXIDASE 4 | APOPTOSIS | ACTIVATION | PHYSIOLOGY | cardiac injury | CELL BIOLOGY | NAD(P)H OXIDASE | FAILING HEART | NOX FAMILY | MESANGIAL CELLS | MYOFIBROBLASTS | EXPRESSION | oxidative stress | REVEALS | Fibronectins - biosynthesis | Diabetic Cardiomyopathies - metabolism | Reactive Oxygen Species - metabolism | Oxidative Stress | Ventricular Myosins - biosynthesis | Humans | Membrane Glycoproteins - biosynthesis | NADPH Oxidases - metabolism | Diabetes Mellitus, Type 1 - metabolism | Male | Diabetes Mellitus, Type 1 - complications | Collagen - biosynthesis | Diabetes Mellitus, Experimental - complications | NADH, NADPH Oxidoreductases - biosynthesis | NADPH Oxidases - biosynthesis | Diabetes Mellitus, Experimental - metabolism | Actins - biosynthesis | Oligonucleotides, Antisense - pharmacology | Cells, Cultured | Rats | Glucose - pharmacology | NADPH Oxidase 4 | NADPH Oxidase 2 | Rats, Sprague-Dawley | NADPH Oxidase 1 | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - metabolism | Diabetic Cardiomyopathies - pathology | Heart Ventricles - metabolism | Complications and side effects | Type 1 diabetes | Cardiomyopathy | Heart cells | NADP (Coenzyme) | Physiological aspects | Development and progression | Research | Health aspects | Heart diseases | Diabetes | Glucose | Rodents | Cells | Index Medicus | Receptors and Signal Transduction
Journal Article
PLoS Genetics, ISSN 1553-7390, 08/2015, Volume 11, Issue 8, pp. e1005352 - e1005352
Journal Article
Diabetes, ISSN 0012-1797, 05/2009, Volume 58, Issue 5, pp. 1201 - 1211
Journal Article