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PLoS ONE, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, pp. e9313 - e9313
Background: Lewy body disease is a heterogeneous group of neurodegenerative disorders characterized by alpha-synuclein accumulation that includes dementia with... 
WILD-TYPE | CHAPERONE-MEDIATED AUTOPHAGY | ALZHEIMERS-DISEASE | MOUSE MODEL | FRONTOTEMPORAL DEMENTIA | BIOLOGY | AGGREGATE-PRONE PROTEINS | NEURONAL EXPRESSION | PARKINSONS-DISEASE | TRANSGENIC MICE | GAUCHER-DISEASE | Pyramidal Cells - metabolism | Humans | Nerve Degeneration - genetics | Male | Autophagy | Nerve Degeneration - metabolism | Alzheimer Disease - pathology | Brain - metabolism | Lewy Body Disease - genetics | RNA Interference | Brain - ultrastructure | Aged, 80 and over | Female | alpha-Synuclein - genetics | Pyramidal Cells - ultrastructure | Disease Models, Animal | Cell Line | Ubiquitin-Activating Enzymes - genetics | Signal Transduction | Lewy Body Disease - pathology | Mice, Transgenic | Microscopy, Electron | Ubiquitin-Activating Enzymes - metabolism | Blotting, Western | Nerve Degeneration - pathology | Lewy Body Disease - metabolism | Microscopy, Confocal | Autophagy-Related Protein 7 | Animals | Alzheimer Disease - metabolism | Brain - pathology | Cell Line, Tumor | Pyramidal Cells - pathology | Aged | Mice | Alzheimer Disease - genetics | alpha-Synuclein - metabolism | TOR protein | Neurosciences | Animal models | Parkinson's disease | Pathogenesis | Toxicity | Parkinsons disease | Phagosomes | Nervous system | Synuclein | Neuromodulation | Accumulation | Modulators | Proteins | Infusion | Alterations | Neurodegeneration | Dementia disorders | Alzheimer's disease | Movement disorders | Enzymes | Neurodegenerative diseases | Neurons | Immunomodulation | Lewy body disease | Transgenic mice | Rapamycin | Patients | Lewy bodies | Studies | Pathology | Mutation | Alzheimers disease | Phagocytosis | Apoptosis | Dementia | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 4, pp. e19338 - e19338
Journal Article
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 2014, Volume 127, Issue 6, pp. 861 - 879
Immunotherapeutic approaches are currently in the spotlight for their potential as disease-modifying treatments for neurodegenerative disorders. The discovery... 
Parkinson's disease | AFFITOME | Immunotherapy | Vaccination | Alpha-synuclein | AFFITOPE | HUMAN ALPHA-SYNUCLEIN | ALZHEIMERS-DISEASE | PATHOLOGY | NEUROSCIENCES | NEURODEGENERATIVE DISEASES | CLINICAL NEUROLOGY | EXPLORATORY ACTIVITY | AFFITOME (R) | A-BETA | MICE | MOTOR COORDINATION | LEWY-BODY-DISEASE | TRANSGENIC MOUSE MODEL | AFFITOPE (R) | Parkinson Disease - therapy | Memory Disorders - physiopathology | Motor Activity - physiology | T-Lymphocytes - physiology | Putamen - pathology | Humans | Caudate Nucleus - pathology | Nerve Degeneration - physiopathology | Axons - physiology | Synapses - pathology | Caudate Nucleus - physiopathology | Lewy Body Disease - therapy | Microglia - physiology | Vaccination - methods | Putamen - physiopathology | Microglia - pathology | alpha-Synuclein - genetics | T-Lymphocytes - pathology | Memory Disorders - pathology | Disease Models, Animal | Parkinson Disease - pathology | Synapses - physiology | Mice, Inbred C57BL | Lewy Body Disease - pathology | Antibodies - cerebrospinal fluid | Mice, Transgenic | Clinical Trials as Topic | Parkinson Disease - physiopathology | Nerve Degeneration - pathology | Animals | Axons - pathology | Memory Disorders - therapy | Antibodies - blood | alpha-Synuclein - metabolism | Lewy Body Disease - physiopathology | Nerve Degeneration - therapy | Oligomers | Platelet-derived growth factor | Peptides | Clinical trials | Genetic engineering | T cells | Index Medicus
Journal Article
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 03/2009, Volume 108, Issue 6, pp. 1561 - 1574
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 8, pp. e12333 - e12333
Levels of glutathione are lower in the substantia nigra (SN) early in Parkinson's disease (PD) and this may contribute to mitochondrial dysfunction and... 
WILD-TYPE | OXIDATIVE STRESS | GROWTH-FACTOR-B | GLUTATHIONE DEFICIENCY | ENDOTHELIAL-CELLS | BIOLOGY | NEURODEGENERATIVE SYNUCLEINOPATHIES | FAMILIAL PARKINSONS-DISEASE | KAPPA-B | RAT-BRAIN | TRANSGENIC MICE | Motor Activity - physiology | Tyrosine 3-Monooxygenase - metabolism | Glutathione - metabolism | Humans | Cytosol - drug effects | Motor Activity - drug effects | Male | NF-kappa B - metabolism | Protein Transport - drug effects | Neuroprotective Agents - pharmacology | Neostriatum - drug effects | Time Factors | Neostriatum - metabolism | Parkinson Disease - metabolism | alpha-Synuclein - genetics | Acetylcysteine - administration & dosage | Neuroprotective Agents - administration & dosage | Dopamine - metabolism | Gene Expression | Parkinson Disease - pathology | Proto-Oncogene Proteins c-sis - metabolism | Administration, Oral | Mice, Transgenic | Parkinson Disease - physiopathology | Animals | Acetylcysteine - pharmacology | Cytosol - metabolism | Mice | Dietary Supplements | Brain | Oxidative stress | Neurosciences | Cysteine | Parkinson's disease | Toxicity | Substantia nigra | Acetylcysteine | Amino acids | Mitochondrial DNA | Kinases | Tyrosine 3-monooxygenase | Synuclein | Mitochondria | Neurotoxicity | Carcinogens | Analgesics | Transgenic animals | Rodents | Neostriatum | Dopamine transporter | Supplementation | Localization | Drug dosages | Deoxyribonucleic acid--DNA | Age | Movement disorders | Glutathione | Dopamine receptors | Tyrosine | Alanine | Dopamine | Neurodegenerative diseases | Neurons | Hydroxylase | Dietary supplements | Transgenic mice | Amyotrophic lateral sclerosis | Neurology | Hypotheses | Transport buildings, stations and terminals | Immunoreactivity | Position (location) | Transporter | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Journal of Neuroimmune Pharmacology, ISSN 1557-1890, 6/2009, Volume 4, Issue 2, pp. 190 - 199
Journal Article