X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (127) 127
index medicus (107) 107
female (79) 79
cancer (55) 55
cell biology (53) 53
animals (41) 41
oncology (41) 41
proteins (40) 40
biology (39) 39
middle aged (39) 39
aged (35) 35
cell line, tumor (35) 35
expression (33) 33
male (33) 33
prognosis (32) 32
article (31) 31
binding (31) 31
breast neoplasms - metabolism (31) 31
adult (30) 30
breast neoplasms - pathology (30) 30
cell (30) 30
immunohistochemistry (30) 30
bioscience (29) 29
calcium (29) 29
cycle (29) 29
landes (29) 29
mice (29) 29
organogenesis (29) 29
breast cancer (28) 28
pancreatic cancer (28) 28
pathology (28) 28
caveolin-1 (26) 26
breast-cancer (24) 24
gene expression (20) 20
gene expression regulation, neoplastic (20) 20
analysis (19) 19
pancreatic neoplasms - metabolism (19) 19
pancreatic neoplasms - pathology (19) 19
progression (19) 19
growth (18) 18
tumor stroma (18) 18
aged, 80 and over (17) 17
metastasis (17) 17
pancreatic neoplasms - genetics (17) 17
surgery (17) 17
tumors (17) 17
breast neoplasms - genetics (16) 16
cells (16) 16
disease progression (16) 16
oxidative stress (16) 16
abridged index medicus (15) 15
carcinoma (15) 15
autophagy (14) 14
biochemistry & molecular biology (14) 14
biomarkers, tumor - metabolism (14) 14
caveolin 1 - genetics (14) 14
caveolin 1 - metabolism (14) 14
kaplan-meier estimate (14) 14
research (14) 14
retinoblastoma protein - metabolism (14) 14
therapy (14) 14
carcinoma, pancreatic ductal - genetics (13) 13
carcinoma, pancreatic ductal - metabolism (13) 13
cdk4 (13) 13
cyclin-dependent kinase 4 - antagonists & inhibitors (13) 13
cyclin-dependent kinase 6 - antagonists & inhibitors (13) 13
development and progression (13) 13
protein (13) 13
tumor microenvironment (13) 13
adenocarcinoma (12) 12
e2f (12) 12
fibroblasts (12) 12
fibroblasts - metabolism (12) 12
neoplasms (12) 12
report (12) 12
stromal cells - metabolism (12) 12
carcinoma, pancreatic ductal - pathology (11) 11
protein kinase inhibitors - pharmacology (11) 11
research paper (11) 11
retinoblastoma protein - genetics (11) 11
carcinoma, intraductal, noninfiltrating - pathology (10) 10
cardiovascular system (10) 10
cell proliferation (10) 10
gene expression profiling (10) 10
genetic aspects (10) 10
glycolysis (10) 10
subtypes (10) 10
angiogenesis (9) 9
biomarkers (9) 9
breast neoplasms - mortality (9) 9
cancer-associated fibroblasts (9) 9
cell cycle (9) 9
chemotherapy (9) 9
gene-expression (9) 9
phenotype (9) 9
phosphorylation (9) 9
stromal cells - pathology (9) 9
survival (9) 9
survival analysis (9) 9
apoptosis (8) 8
more...
Library Location Library Location
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Cell Cycle, ISSN 1538-4101, 12/2011, Volume 10, Issue 23, pp. 4047 - 4064
We have recently proposed a new mechanism for explaining energy transfer in cancer metabolism.  In this scenario, cancer cells behave as metabolic parasites,... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Oxidative phosphorylation (OXPHOS) | NADH dehydrogenase | Complex I | Mitophagy | Warburg's respiratory enzyme | Autophagy | Complex IV | Mitochondria | Respiratory chain | Cancer metabolism | Cytochrome c oxidase (COX) | Aerobic glycolysis | Metformin | Electron transport | Warburg effect | RESPIRATORY-CHAIN | STROMAL CAVEOLIN-1 | CELL BIOLOGY | BREAST-CANCER | LACTATE | electron transport | STRESS | aerobic glycolysis | complex IV | autophagy | mitochondria | metformin | FIBROBLASTS | cancer metabolism | mitophagy | MICROENVIRONMENT | complex I | ENERGY-METABOLISM | cytochrome c oxidase (COX) | EXPRESSION | respiratory chain | oxidative phosphorylation (OXPHOS) | Oxidative Stress | Epithelial Cells - drug effects | Humans | Gene Expression Regulation, Neoplastic | Tumor Microenvironment | Cell Line, Tumor - drug effects | Malonates - pharmacology | Mitochondrial Proteins - genetics | Breast Neoplasms - metabolism | Electron Transport Complex IV - metabolism | Neoplasm Metastasis | Mitochondria - genetics | Membrane Transport Proteins - genetics | Mitochondrial Proteins - metabolism | Biomarkers, Tumor - metabolism | Female | Membrane Transport Proteins - metabolism | Metformin - pharmacology | Computational Biology | Enzyme Inhibitors - pharmacology | Oxidative Phosphorylation | Receptors, Cell Surface - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Energy Metabolism | Glycolysis | Enzyme Activation | Receptors, Cell Surface - genetics | Warburg respiratory enzyme | Report
Journal Article
SCIENTIFIC REPORTS, ISSN 2045-2322, 09/2019, Volume 9, Issue 1, pp. 12766 - 15
Genetic alterations are essential for cancer initiation and progression. However, differentiating mutations that drive the tumor phenotype from mutations that... 
PATHWAYS | READ ALIGNMENT | COMBINATIONS | PROGNOSIS | ANNOTATION | MULTIDISCIPLINARY SCIENCES | DRIVER | MULTIPLE MUTATIONS | SOMATIC MUTATIONS | DISCOVERY | PREDICTION | Cell culture | Phenotypes | Alleles | Genomes | Mutation | Ribonucleic acid--RNA | Cell differentiation | Cancer | Fitness
Journal Article
Cell Cycle, ISSN 1538-4101, 09/2010, Volume 9, Issue 17, pp. 3515 - 3533
Recently, using a co-culture system, we demonstrated that MCF7 epithelial cancer cells induce oxidative stress in adjacent cancer-associated fibroblasts,... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Caveolin-1 | Oxidative stress | TIGAR | NFκB | BNIP3 | Tumor stroma | Cancer associated fibroblasts | Mitophagy | Autophagy | HIF1 | Hypoxia | LC3 | Predictive biomarker | hypoxia | autophagy | ONCOGENICALLY TRANSFORMED-CELLS | NITRIC-OXIDE SYNTHASE | DOWN-REGULATION | INDUCIBLE FACTOR INDUCTION | cancer associated fibroblasts | CELL BIOLOGY | BREAST-CANCER | caveolin-1 | mitophagy | predictive biomarker | tumor stroma | CAROTID-BODY | EXPRESSION | NF kappa B | oxidative stress | MITOCHONDRIAL COMPLEX-III | Up-Regulation | Oxidative Stress | Microtubule-Associated Proteins - metabolism | Coculture Techniques | Humans | Tumor Microenvironment | NF-kappa B - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Breast Neoplasms - metabolism | Cell Hypoxia | Chloroquine - pharmacology | RNA Interference | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Female | Glutathione Synthase - metabolism | Membrane Proteins - metabolism | Antirheumatic Agents - pharmacology | Glutathione Synthase - antagonists & inhibitors | Fibroblasts - metabolism | Proto-Oncogene Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Paracrine Communication | Cell Survival | Stromal Cells - metabolism | Caveolin 1 - genetics | Mice, Knockout | Caveolin 1 - metabolism | Animals | Breast Neoplasms - pathology | Cell Line, Tumor | Mice | RNA, Small Interfering - metabolism | Report | cancer-associated fibroblasts
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 04/2015, Volume 6, Issue 1, p. 6744
Pancreatic ductal adenocarcinoma (PDA) has a dismal prognosis and insights into both disease etiology and targeted intervention are needed. A total of 109... 
SIGNATURES | MODELS | DUCTAL ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | TUMOR | SOMATIC MUTATION | COPY-NUMBER ALTERATION | GENOMIC CHARACTERIZATION | CARCINOMA | PROGRESSION | DISCOVERY | Cell Cycle - genetics | RNA-Binding Proteins - genetics | Prognosis | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Middle Aged | Carcinoma, Adenosquamous - drug therapy | Male | Antineoplastic Agents - therapeutic use | DNA Repair - genetics | Molecular Targeted Therapy | Tumor Suppressor Protein p53 - genetics | Carcinoma, Pancreatic Ductal - genetics | DNA Copy Number Variations | Genetic Variation | Pancreatic Neoplasms - drug therapy | Aged, 80 and over | Adult | Female | Nuclear Proteins - genetics | Carcinoma, Adenosquamous - pathology | Pancreatic Neoplasms - pathology | Kaplan-Meier Estimate | Pancreatic Neoplasms - genetics | Carcinoma, Adenosquamous - genetics | Transcription Factors - genetics | Carcinoma, Pancreatic Ductal - pathology | Sequence Analysis, DNA | Carcinoma, Pancreatic Ductal - drug therapy | Drug Resistance, Neoplasm - genetics | Exome - genetics | Gene Amplification | Sulfonamides - therapeutic use | Wnt Signaling Pathway - genetics | Proto-Oncogene Proteins B-raf - genetics | Retinoblastoma Protein - genetics | Genes, myc - genetics | Indoles - therapeutic use | Aged | BRCA2 Protein - genetics
Journal Article
Cell Cycle, ISSN 1538-4101, 08/2011, Volume 10, Issue 15, pp. 2497 - 2503
The CDKN2A gene is a tumor suppressor that encodes the CDK4/6 inhibitor p16ink4a. Loss of this tumor suppressor contributes to the bypass of critical senescent... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Therapy | p16 | CDKN2a | Radiation | Cyclin | E2F | CDK | cyclin | radiation | therapy | Review
Journal Article
Cell Cycle, ISSN 1538-4101, 12/2009, Volume 8, Issue 23, pp. 3984 - 4001
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2013, Volume 123, Issue 5, pp. 2155 - 2168
Journal Article
Journal Article
Gut, ISSN 0017-5749, 03/2018, Volume 67, Issue 3, pp. 508 - 520
ObjectivePancreatic ductal adenocarcinoma (PDAC) is a therapy recalcitrant disease with the worst survival rate of common solid tumours. Preclinical models... 
Cancer Genetics | Oncogenes | Pancreatic Cancer | HETEROGENEITY | THERAPY | GENOMIC ANALYSES | MODELS | DUCTAL ADENOCARCINOMA | SUBTYPES | LANDSCAPE | TUMOR | GASTROENTEROLOGY & HEPATOLOGY | PROGRESSION | XENOGRAFTS | Pancreatic Neoplasms - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Middle Aged | Carcinoma, Pancreatic Ductal - metabolism | Male | Tumor Suppressor Protein p53 - genetics | Carcinoma, Pancreatic Ductal - genetics | RNA, Neoplasm - analysis | Heterografts | Smad4 Protein - genetics | DNA Mutational Analysis | Aged, 80 and over | Adult | Female | DNA, Neoplasm - analysis | Gene Expression | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - pathology | Whole Exome Sequencing | Phenotype | Animals | Sequence Analysis, RNA | Cell Line, Tumor | Aged | Mice | Polymorphism, Single Nucleotide | Adenocarcinoma | Cell culture | Pipelines | Biology | Biodiversity | Cancer therapies | Metastases | Cell cycle | Genetic analysis | Xenografts | Evolution | Preservation | Breast cancer | Genetic diversity | Tumor cell lines | Gene expression | Ribonucleic acid--RNA | Patients | Genetic variance | Chemotherapy | Pancreatic cancer | Medical prognosis | Cell lines | Mutation | Solid tumors | Evolution & development | Tumors | ONCOGENES | CANCER GENETICS | Pancreas | PANCREATIC CANCER | 1506
Journal Article