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No shinkei geka. Neurological surgery, ISSN 0301-2603, 01/2019, Volume 47, Issue 1, p. 49
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2015, Volume 66, Issue 3, pp. 273 - 277
  [...]regulated post-translational modification of cellular targets by ROS may be achieved only in highly compartmentalized environments or in the presence of... 
Cardiovascular | Internal Medicine | Cells | Heart failure | Short term | Enzymes | Phosphorylation | Cardiomyocytes | Kinases | Phosphatase | Investigations | Proteins | Ischemia | Transgenic animals | Rodents | Physiology | Deoxyribonucleic acid--DNA
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2010, Volume 107, Issue 35, pp. 15565 - 15570
NAD(P)H oxidases (Noxs) produce O 2 − and play an important role in cardiovascular pathophysiology. The Nox4 isoform is expressed primarily in the mitochondria... 
Heart | Oxidases | Oxidative stress | Cardiomegaly | Up regulation | Mitochondria | Transcriptional regulatory elements | Reactive oxygen species | Fibrosis | Apoptosis | Cardiac hypertrophy | Superoxide | NAD(P)H oxidase | MYOCARDIUM | PRESSURE-OVERLOAD | MYOCYTES | mitochondria | MULTIDISCIPLINARY SCIENCES | FAILURE | MITOCHONDRIAL PERMEABILITY TRANSITION | CARDIAC-HYPERTROPHY | RADICALS | cardiac hypertrophy | superoxide | REVEALS | Reactive Oxygen Species - metabolism | Oxidative Stress | NADPH Oxidases - metabolism | Immunoblotting | Male | Isoenzymes - metabolism | Myocardium - metabolism | NADPH Oxidases - genetics | Female | Heart - physiopathology | In Situ Nick-End Labeling | Echocardiography | Isoenzymes - genetics | Mice, Inbred C57BL | Cardiomegaly - physiopathology | Heart Failure - genetics | Myocardium - pathology | Heart Failure - metabolism | Mitochondria - metabolism | Heart Failure - pathology | NADPH Oxidase 4 | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - metabolism | Mice | Cardiomegaly - genetics | Mitochondria - physiology | Cardiomegaly - metabolism | Heart failure | Composition | Genetic aspects | Health aspects | Proteins | Genotype & phenotype | Rodents | Cardiomyocytes | Physiology | Gene expression | Index Medicus | Biological Sciences
Journal Article
Circulation Research, ISSN 0009-7330, 04/2010, Volume 106, Issue 7, pp. 1253 - 1264
RATIONALE:NADPH oxidases are a major source of superoxide (O2) in the cardiovascular system. The function of Nox4, a member of the Nox family of NADPH... 
Aging | Oxidative stress | Reactive oxygen species | Superoxide | Apoptosis | Hypertrophy | CELLS | PRESSURE-OVERLOAD | CARDIAC & CARDIOVASCULAR SYSTEMS | hypertrophy | PHOSPHORYLATION | apoptosis | ANGIOTENSIN-II | FAMILY NADPH OXIDASES | FREE-RADICALS | NAD(P)H OXIDASE | reactive oxygen species | PERIPHERAL VASCULAR DISEASE | GENERATION | aging | superoxide | HEMATOLOGY | oxidative stress | Aconitate Hydratase - metabolism | Up-Regulation | Cysteine | Cell Proliferation | Uncoupling Agents - pharmacology | Oxidative Stress | Rats, Wistar | Ventricular Function, Left | Apoptosis - drug effects | Mitochondria, Heart - pathology | Humans | NADPH Oxidases - metabolism | Cardiomegaly - pathology | Mitochondria, Heart - drug effects | Myocytes, Cardiac - enzymology | Transfection | Ventricular Dysfunction, Left - genetics | Rotenone - pharmacology | Superoxides - metabolism | Ventricular Dysfunction, Left - pathology | NADPH Oxidases - genetics | Ventricular Dysfunction, Left - enzymology | Disease Models, Animal | Oxidation-Reduction | NADPH Oxidases - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Mitochondria, Heart - enzymology | Cardiomegaly - physiopathology | Rats | Genotype | Mice, Transgenic | Cardiomegaly - enzymology | NADPH Oxidase 4 | Onium Compounds - pharmacology | NADH Dehydrogenase - metabolism | Ventricular Dysfunction, Left - physiopathology | Aging - pathology | Myocytes, Cardiac - pathology | Phenotype | Animals | Myocytes, Cardiac - drug effects | Fibrosis | Mice | Cardiomegaly - genetics | Aging - metabolism | Index Medicus
Journal Article
by Malik, Rainer and Chauhan, Ganesh and Traylor, Matthew and Sargurupremraj, Muralidharan and Okada, Yukinori and Mishra, Aniket and Rutten-Jacobs, Loes and Giese, Anne-Katrin and Van Der Laan, Sander W and Gretarsdottir, Solveig and Anderson, Christopher D and Chong, Michael and Adams, Hieab H. H and Ago, Tetsuro and Almgren, Peter and Amouyel, Philippe and Ay, Hakan and Bartz, Traci M and Benavente, Oscar R and Bevan, Steve and Boncoraglio, Giorgio B and Brown, Robert D and Butterworth, Adam S and Carrera, Caty and Carty, Cara L and Chasman, Daniel I and Chen, Wei-Min and Cole, John W and Correa, Adolfo and Cotlarciuc, Ioana and Cruchaga, Carlos and Danesh, John and De Bakker, Paul I. W and Destefano, Anita L and Den Hoed, Marcel and Duan, Qing and Engelter, Stefan T and Falcone, Guido J and Gottesman, Rebecca F and Grewal, Raji P and Gudnason, Vilmundur and Gustafsson, Stefan and Haessler, Jeffrey and Harris, Tamara B and Hassan, Ahamad and Havulinna, Aki S and Heckbert, Susan R and Holliday, Elizabeth G and Howard, George and Hsu, Fang-Chi and Hyacinth, Hyacinth I and Ikram, M. Arfan and Ingelsson, Erik and Irvin, Marguerite R and Jian, Xueqiu and Jiménez-Conde, Jordi and Johnson, Julie A and Jukema, J. Wouter and Kanai, Masahiro and Keene, Keith L and Kissela, Brett M and Kleindorfer, Dawn O and Kooperberg, Charles and Kubo, Michiaki and Lange, Leslie A and Langefeld, Carl D and Langenberg, Claudia and Launer, Lenore J and Lee, Jin-Moo and Lemmens, Robin and Leys, Didier and Lewis, Cathryn M and Lin, Wei-Yu and Lindgren, Arne G and Lorentzen, Erik and Magnusson, Patrik K and Maguire, Jane and Manichaikul, Ani and McArdle, Patrick F and Meschia, James F and Mitchell, Braxton D and Mosley, Thomas H and Nalls, Michael A and Ninomiya, Toshiharu and O'Donnell, Martin J and Psaty, Bruce M and Pulit, Sara L and Rannikmäe, Kristiina and Reiner, Alexander P and Rexrode, Kathryn M and Rice, Kenneth and Rich, Stephen S and Ridker, Paul M and Rost, Natalia S and Rothwell, Peter M and Rotter, Jerome I and Rundek, Tatjana and Sacco, Ralph L and Sakaue, Saori and Sale, Michele M and ... and AFGen Consortium and UK Young Lacunar DNA Study and EPIC-CVD Consortium and Int Stroke Genetics Consortium ISG and INVENT Consortium and Int Genomics Blood Pressure iGEN and MEGASTROKE Consortium and COMPASS Consortium and METASTROKE Consortium and EPIC-InterAct Consortium and Cohorts Heart Aging Res Genomic and BioBank Japan Cooperative Hosp Grp and NINDS Stroke Genetics Network Si and STARNET and Neurology Working Grp Charge Con and Cohorts for Heart and Aging Research in Genomic Epidemiology (CHARGE) Consortium and International Stroke Genetics Consortium (ISGC) and NINDS Stroke Genetics Network (SiGN) and International Genomics of Blood Pressure (iGEN-BP) Consortium and BioBank Japan Cooperative Hospital Group and Neurology Working Group of the CHARGE Consortium and Science for Life Laboratory, SciLifeLab and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Institutionen för immunologi, genetik och patologi and Medicinsk genetik och genomik and Geriatrik and Kardiovaskulär epidemiologi and Uppsala universitet and Institutionen för medicinska vetenskaper and Molekylär epidemiologi and Institutionen för folkhälso- och vårdvetenskap
Nature Genetics, ISSN 1061-4036, 04/2018, Volume 50, Issue 4, pp. 524 - 537
Journal Article
Journal Article
Stroke, ISSN 0039-2499, 11/2017, Volume 48, Issue 11, pp. 3049 - 3056
Journal Article
Fukuoka igaku zasshi = Hukuoka acta medica, ISSN 0016-254X, 06/2014, Volume 105, Issue 6, pp. 125 - 130
Journal Article
Journal Article