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Journal Article
The American Journal of Pathology, ISSN 0002-9440, 2002, Volume 160, Issue 5, pp. 1567 - 1572
Journal Article
Science, ISSN 0036-8075, 1/2003, Volume 299, Issue 5607, pp. 708 - 710
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 11/2017, Volume 23, Issue 21, pp. 6733 - 6743
Purpose: Patients who inherit a pathogenic loss-of-function genetic variant involving one of the four succinate dehydrogenase (SDH) subunit genes have up to an... 
MITOCHONDRIAL COMPLEX II | ONCOLOGY | SDHD GENE-MUTATIONS | B GENE | GERMLINE SDHA | YEAST MODEL | PARAGANGLIOMA | FLAVOPROTEIN SUBUNIT | GASTROINTESTINAL STROMAL TUMORS | SACCHAROMYCES-CEREVISIAE | DEFICIENCY | Electron Transport Complex II - genetics | Early Detection of Cancer | Saccharomyces cerevisiae - genetics | Humans | Neoplasms - enzymology | Succinate Dehydrogenase - chemistry | Saccharomyces cerevisiae Proteins - genetics | Mutation - genetics | Succinate Dehydrogenase - genetics | Neoplasms - genetics | Electron Transport Complex II - metabolism | Gene Expression Regulation, Enzymologic - genetics | Saccharomyces cerevisiae Proteins - metabolism | Saccharomyces cerevisiae - enzymology | Succinate Dehydrogenase - metabolism | Neoplasms - pathology | Electron Transport Complex II - chemistry | Gene Expression Regulation, Neoplastic - genetics | Saccharomyces cerevisiae Proteins - chemistry | Pathogens | Succinate dehydrogenase | Yeast | Dehydrogenases | Risk groups | Genes | Dehydrogenase | Medical screening | Patients | Reclassification | Pathogenicity | Genetic variance | Mitochondria | Alterations | Computation | Experimental design | Computer applications | Mutation | Protein structure | Tumors | Cancer | mutation | variants of unknown significance | GI stromal tumor | succinate dehydrogenase | paraganglioma
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 05/2008, Volume 14, Issue 9, pp. 2717 - 2725
Purpose: To evaluate the activity of imatinib in treating advanced, life-threatening malignancies expressing one or more imatinib-sensitive tyrosine kinases.... 
imatinib | platelet-derived growth factor | sarcoma | KIT | tyrosine kinase | METASTATIC MELANOMA | IN-VITRO | MYELOPROLIFERATIVE DISORDERS | ONCOLOGY | CLINICAL-EFFICACY | C-KIT | DERMATOFIBROSARCOMA PROTUBERANS | MYELOID-LEUKEMIA | ADENOID CYSTIC CARCINOMA | SALIVARY-GLANDS | GASTROINTESTINAL STROMAL TUMORS | Piperazines - administration & dosage | Protein-Tyrosine Kinases - metabolism | Humans | Antineoplastic Agents - therapeutic use | Hematologic Neoplasms - enzymology | Antineoplastic Agents - administration & dosage | Proto-Oncogene Proteins c-kit - metabolism | Receptor, Platelet-Derived Growth Factor beta - genetics | Neoplasms - genetics | Antineoplastic Agents - adverse effects | Proto-Oncogene Proteins c-kit - genetics | Receptor, Platelet-Derived Growth Factor beta - metabolism | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Pyrimidines - administration & dosage | Kaplan-Meier Estimate | Neoplasms - enzymology | Piperazines - therapeutic use | Imatinib Mesylate | Piperazines - adverse effects | Neoplasms - drug therapy | Pyrimidines - therapeutic use | Receptor, Platelet-Derived Growth Factor alpha - genetics | Pyrimidines - adverse effects | Hematologic Neoplasms - drug therapy | Hematologic Neoplasms - genetics | Benzamides | Mutation | Protein-Tyrosine Kinases - antagonists & inhibitors | Index Medicus
Journal Article
Blood, ISSN 0006-4971, 05/2003, Volume 101, Issue 9, pp. 3597 - 3605
FLT3 (fms-related tyrosine kinase/Flk2/ Stk-2) is a receptor tyrosine kinase (RTK) primarily expressed on hematopoietic cells. In blasts from acute myelogenous... 
INTERNAL TANDEM DUPLICATION | GENE | RISK GROUP | ACUTE MYELOGENOUS LEUKEMIA | BONE-MARROW | RECEPTOR | ACUTE MYELOID-LEUKEMIA | CELL-GROWTH | MUTATIONS | HEMATOLOGY | ENDOTHELIAL GROWTH-FACTOR | Vascular Endothelial Growth Factor A | Apoptosis - drug effects | Vascular Endothelial Growth Factors | Humans | fms-Like Tyrosine Kinase 3 | Intercellular Signaling Peptides and Proteins - biosynthesis | Neoplasm Proteins - antagonists & inhibitors | Antineoplastic Agents - therapeutic use | Tandem Repeat Sequences | Leukemia, Myeloid - pathology | Recombinant Fusion Proteins - antagonists & inhibitors | Tumor Cells, Cultured - enzymology | Transfection | Protein Processing, Post-Translational - drug effects | Bone Marrow Transplantation | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Female | Indoles - pharmacology | Antineoplastic Agents - pharmacology | Lymphokines - biosynthesis | Phosphorylation - drug effects | Neoplasm Proteins - genetics | Pyrroles - therapeutic use | Protein Structure, Tertiary | Proto-Oncogene Proteins - antagonists & inhibitors | Acute Disease | Leukemia, Myeloid - enzymology | Enzyme Inhibitors - pharmacology | Proto-Oncogene Proteins - genetics | Endothelial Growth Factors - biosynthesis | Mice, SCID | Enzyme Inhibitors - therapeutic use | Xenograft Model Antitumor Assays | Point Mutation | Pyrroles - pharmacology | Animals | Receptor Protein-Tyrosine Kinases - genetics | Signal Transduction - drug effects | Mice, Nude | Indoles - therapeutic use | Mice, Inbred NOD | Mice | Amino Acid Substitution
Journal Article
Cancer Research, ISSN 0008-5472, 04/2013, Volume 73, Issue 8 Supplement, pp. 3281 - 3281
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 05/2018, Volume 36, Issue 15_suppl, pp. 11535 - 11535
Journal Article
Cancer Research, ISSN 0008-5472, 09/2003, Volume 63, Issue 18, pp. 5761 - 5766
Journal Article
Blood, ISSN 0006-4971, 11/2013, Volume 122, Issue 21, pp. 4105 - 4105
Abstract Activating KIT mutations are a hallmark of human systemic mast cell disease and have also been identified in gastrointestinal stromal tumors, AML,... 
Journal Article