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Nature, ISSN 0028-0836, 2012, Volume 482, Issue 7386, pp. 529 - U254
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2011, Volume 17, Issue 10, pp. 1275 - 1282
Journal Article
Nature Communications, ISSN 2041-1723, 08/2016, Volume 7, Issue 1, pp. 12425 - 12425
Poly (ADP-ribose) polymerase (PARP) inhibitor (PARPi) olaparib has been approved for treatment of advanced ovarian cancer associated with BRCA1 and BRCA2... 
POLY(ADP-RIBOSE) POLYMERASE | REPAIR | REPLICATION FORKS | LETHALITY | MUTANT-CELLS | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | PARP | HOMOLOGOUS RECOMBINATION | OVARIAN-CANCER | OLAPARIB MAINTENANCE THERAPY | Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 09/2017, Volume 121, Issue 8, pp. 923 - 929
RATIONALE:Duchenne muscular dystrophy is a severe inherited form of muscular dystrophy caused by mutations in the reading frame of the dystrophin gene... 
Cardiomyopathies | Mice | Dystrophin | Gene editing | Muscular dystrophy | FIBROSIS | CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | CARDIOMYOPATHY | dystrophin | gene editing | mice | DELIVERY | NUCLEASES | muscular dystrophy | THERAPY | MOUSE MODEL | MUSCLE FUNCTION | PERIPHERAL VASCULAR DISEASE | DUCHENNE MUSCULAR-DYSTROPHY | CAS9 | HEMATOLOGY | cardiomyopathies | Dependovirus - genetics | RNA, Guide - genetics | Exons | RNA, Guide - metabolism | Utrophin - genetics | Recovery of Function | Cardiomyopathies - genetics | Cardiomyopathies - physiopathology | Muscular Dystrophy, Duchenne - physiopathology | Clustered Regularly Interspaced Short Palindromic Repeats | Mice, Inbred mdx | Papillary Muscles - metabolism | Muscular Dystrophy, Duchenne - therapy | Dystrophin - metabolism | Disease Models, Animal | Myocardial Contraction | Genetic Predisposition to Disease | CRISPR-Associated Proteins - genetics | Gene Expression Regulation | Cardiomyopathies - therapy | Papillary Muscles - physiopathology | Phenotype | Animals | Dystrophin - genetics | CRISPR-Associated Proteins - metabolism | Cardiomyopathies - metabolism | CRISPR-Cas Systems | Fibrosis | Gene Editing - methods | High-Throughput Nucleotide Sequencing | Muscular Dystrophy, Duchenne - metabolism | Muscular Dystrophy, Duchenne - genetics | Mutation | Genetic Vectors | Papillary Muscles - pathology | Genetic Therapy - methods | Neonates | CRISPR | Cardiac muscle | Cardiomyopathy | Contractility | Genomes | Ribonucleic acid--RNA | Muscle contraction | Proteins | Rodents | Duchenne's muscular dystrophy | Protein expression | Dystrophy | Immunofluorescence | Heart diseases | genome editing | AAV | gene therapy | Duchenne muscular dystrophy cardiomyopathy | cardiomyopathy
Journal Article
Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6026, pp. 238 - 240
Journal Article
Nature Biotechnology, ISSN 1087-0156, 03/2009, Volume 27, Issue 3, pp. 264 - 274
Journal Article
BioEssays, ISSN 0265-9247, 04/2013, Volume 35, Issue 4, pp. 397 - 407
In this essay, we discuss new insights into the wide‐ranging impacts of mammalian transposable elements (TE) on gene expression and function. Nearly half of... 
transposition | retrotransposon | gene regulation | transcriptional variation | gene expression | Gene expression | Genomics | Genomes
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 513, Issue 7519, pp. 512 - 516
Mutations that deregulate Notch1 and Ras/phosphoinositide 3 kinase (PI3K)/Akt signalling are prevalent in T-cell acute lymphoblastic leukaemia (T-ALL), and... 
ACTIVATION | MULTIDISCIPLINARY SCIENCES | ACUTE LYMPHOBLASTIC-LEUKEMIA | FREQUENCY | PTEN | MUTATIONS | CANCER | Precursor T-Cell Lymphoblastic Leukemia-Lymphoma - pathology | Clone Cells - drug effects | Diphenylamine - pharmacology | Receptor, Notch1 - chemistry | Male | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Diphenylamine - analogs & derivatives | Receptor, Notch1 - deficiency | Benzamides - therapeutic use | Benzamides - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Precursor T-Cell Lymphoblastic Leukemia-Lymphoma - metabolism | Genes, ras - genetics | Protein Structure, Tertiary | Precursor T-Cell Lymphoblastic Leukemia-Lymphoma - drug therapy | Diphenylamine - therapeutic use | Mice, Inbred C57BL | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Receptor, Notch1 - metabolism | Down-Regulation - drug effects | Sulfonamides - pharmacology | Clone Cells - metabolism | Drug Synergism | Indazoles - pharmacology | Drug Resistance, Neoplasm - genetics | Animals | Clone Cells - pathology | Signal Transduction - drug effects | Sulfonamides - therapeutic use | Precursor T-Cell Lymphoblastic Leukemia-Lymphoma - genetics | Protein Kinase Inhibitors - therapeutic use | Mice | Protein Kinase Inhibitors - pharmacology | Receptor, Notch1 - genetics | Indazoles - therapeutic use | Drug Resistance, Neoplasm - drug effects | Studies | Rodents | Leukemia | Cloning | Medical prognosis | Mutation | Drug resistance | Gene expression | Drug dosages | Mass spectrometry | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2015, Volume 112, Issue 19, pp. 6128 - 6133
Journal Article