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Diabetes/Metabolism: Research and Reviews, ISSN 1520-7552, 02/2014, Volume 30, Issue 2, p. 120
Journal Article
Diabetes/Metabolism Research and Reviews, ISSN 1520-7552, 02/2014, Volume 30, Issue 2, pp. 120 - 121
Journal Article
Journal Article
Journal of Endocrinology, ISSN 0022-0795, 11/2011, Volume 211, Issue 2, pp. 177 - 185
Journal Article
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 01/2018, Volume 233, Issue 1, pp. 486 - 496
Our data suggest that short‐term early life protein malnourishment induces reduction in redox GIIS signaling only at extreme oxidant challenges, elicited here... 
antioxidant enzymes | insulin secretion | protein malnutrition | pancreatic islets | reactive oxygen species | OXIDATIVE STRESS | PHYSIOLOGY | CELL BIOLOGY | OXYGEN | SUPPLEMENTATION | PKA-ALPHA EXPRESSION | SUPEROXIDE-DISMUTASE ACTIVITY | DIET | HYDROGEN-PEROXIDE | BETA-CELL MASS | TAURINE | MALNOURISHED RATS | Islets of Langerhans - drug effects | Rats, Wistar | Glutathione - metabolism | Protein-Energy Malnutrition - physiopathology | Male | Insulin - blood | RNA, Messenger - metabolism | Time Factors | Islets of Langerhans - metabolism | Superoxide Dismutase-1 - metabolism | Protein-Energy Malnutrition - blood | Nutritional Status | Insulin Secretion | Disease Models, Animal | Glutathione Peroxidase - metabolism | Oxidation-Reduction | Catalase - genetics | RNA, Messenger - genetics | Protein-Energy Malnutrition - genetics | Antioxidants - pharmacology | Glutathione Peroxidase - genetics | Hydrogen Peroxide - metabolism | Catalase - metabolism | Diet, Protein-Restricted | Gene Expression Regulation, Enzymologic | Insulin - metabolism | Animals | Animal Nutritional Physiological Phenomena | Superoxide Dismutase-1 - genetics | Protein-Energy Malnutrition - metabolism | Oxidative Stress - drug effects | Blood Glucose - metabolism | Insulin | Dextrose | Glucose | Glutathione peroxidase | Hydrogen peroxide | Secretion | Weaning | Rats | Acetylcysteine | Superoxide dismutase | Superoxide | Gene expression | Proteins | Antioxidants | Catalase | Rodents | Peroxidase | Malnutrition | Pancreas | Glutathione | Index Medicus
Journal Article
Canadian Journal of Physiology and Pharmacology, ISSN 0008-4212, 2014, Volume 92, Issue 10, pp. 867 - 878
The disruption to glucose homeostasis upon glucocorticoid (GC) treatment in adult male rats has not been fully characterized in older rats or in females. Thus,... 
dexamethasone | insulin sensitivity | dexaméthasone | sécrétion d’insuline | femelle | insulin secretion | female | glucose tolerance | tolérance au glucose | sensibilité à l’insuline | Glucose tolerance | Female | Dexamethasone | Insulin sensitivity | Insulin secretion | PHYSIOLOGY | ELDERLY SUBJECTS | SENSITIVITY | INTOLERANCE | INDUCED INSULIN-RESISTANCE | ENDOCRINE PANCREAS | WOMEN | NORMAL MEN | BETA-CELL DYSFUNCTION | MODEL ASSESSMENT | PHARMACOLOGY & PHARMACY | Adrenal Glands - pathology | Age Factors | Rats, Wistar | Cholesterol - blood | Body Weight - drug effects | Homeostasis | Male | Anti-Inflammatory Agents - adverse effects | Dexamethasone - pharmacology | Glucose Intolerance - chemically induced | Adrenal Glands - drug effects | Insulin Secretion | Glucocorticoids - adverse effects | Liver - metabolism | Eating - drug effects | Hyperinsulinism - chemically induced | Insulin - metabolism | Animals | Insulin-Secreting Cells - drug effects | Sex Factors | Triglycerides - blood | Glucose - metabolism | Immunosuppressive Agents - adverse effects | Insulin-Secreting Cells - pathology | Blood Glucose - metabolism | Medical research | Corticosteroids | Physiological aspects | Medicine, Experimental | Dosage and administration | Glucose | Research | Dextrose | Insulin | Rodents | Medical treatment | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2014, Volume 9, Issue 4, pp. e93531 - e93531
Glucocorticoid (GC)-based therapies can cause insulin resistance (IR), glucose intolerance, hyperglycemia and, occasionally, overt diabetes. Understanding the... 
RESPONSIVENESS | 11-BETA-HYDROXYSTEROID DEHYDROGENASE TYPE-1 | GLUCAGON-LIKE PEPTIDE-1 | MULTIDISCIPLINARY SCIENCES | MASS | RESISTANCE | MECHANISMS | SECRETION | RECEPTORS | ISLET CELLS | PREDNISOLONE | Glucocorticoids - administration & dosage | Dexamethasone - administration & dosage | Drug Administration Schedule | Rats, Wistar | Glucagon-Secreting Cells - pathology | Cells, Cultured | Injections, Intraperitoneal | Insulin Resistance | Rats | Dexamethasone - adverse effects | Male | Blood Glucose - drug effects | Glucagon-Secreting Cells - drug effects | Hyperinsulinism - chemically induced | Insulin - metabolism | Animals | Glucagon-Secreting Cells - physiology | Homeostasis - drug effects | Blood Glucose - metabolism | Hyperinsulinism - blood | Insulin Secretion | Glucocorticoids - adverse effects | Glucose metabolism | Hyperglycemia | Care and treatment | Dexamethasone | Analysis | Homeostasis | Diagnosis | Health aspects | Bioengineering | Hyperinsulinemia | Biomedical research | Dehydrogenases | Glucagon | Glucocorticoids | Biology | Glucose | Guanylate cyclase | Blood | Blockage | Secretory vesicles | Rodents | Physiology | Pancreas | Obesity | Secretion | Diabetes mellitus | Metabolism | Insulin | Glucose tolerance | Intolerance | Insulin resistance | Diabetes | Metabolic disorders | Endocrinology | Index Medicus
Journal Article
Journal Article