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JAMA, The Journal of the American Medical Association, ISSN 0098-7484, 01/2011, Volume 305, Issue 1, p. 95
Results of a study to evaluate the relationship between a microRNA/TP53 feedback circuitry with pathogenesis and outcome of B-cell chronic lymphocytic leukemia... 
Chronic lymphocytic leukemia | Care and treatment | Genetic aspects | MicroRNA | Diagnosis | Research
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 08/2007, Volume 67, Issue 15, pp. 7421 - 7430
Administration of a combination of yeast-derived {szligbeta}-glucan with antitumor monoclonal antibodies (mAb) has significant therapeutic efficacy in a... 
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2006, Volume 177, Issue 3, pp. 1661 - 1669
Anti-tumor mAbs hold promise for cancer therapy, but are relatively inefficient. Therefore, there is a need for agents that might amplify the effectiveness of... 
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 08/2007, Volume 67, Issue 15, pp. 7421 - 7430
Administration of a combination of yeast-derived beta-glucan with antitumor monoclonal antibodies (mAb) has significant therapeutic efficacy in a variety of... 
COMPLEMENT | T-CELL IMMUNITY | ONCOLOGY | RITUXIMAB | IN-VIVO | RECEPTOR | TUMOR-CELLS | CANCER-IMMUNOTHERAPY | CD46 | MEMBRANE COFACTOR PROTEIN | EXPRESSION
Journal Article
Conference Proceeding
Cancer Chemotherapy and Pharmacology, ISSN 0344-5704, 11/2015, Volume 76, Issue 5, pp. 949 - 955
The sequence bendamustine (B) + Irinotecan (I) followed by etoposide (E) + carboplatin (C) was hypothesized to increase progression-free survival (PFS) and... 
Chemotherapy | Medicine & Public Health | Lung cancer | Biomarkers | Oncology | Cancer Research | Pharmacology/Toxicology | Extensive small cell lung cancer | SOLID TUMORS | II TRIAL | EVERY 3 WEEKS | ETOPOSIDE | REPAIR | CISPLATIN | ONCOLOGY | ERCC1 | PHARMACOLOGY & PHARMACY | Endonucleases - deficiency | Lung Neoplasms - drug therapy | Endonucleases - genetics | Antineoplastic Combined Chemotherapy Protocols - administration & dosage | Carcinoma, Small Cell - genetics | Bendamustine Hydrochloride - adverse effects | Biomarkers, Tumor - deficiency | Humans | Middle Aged | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Male | Fatigue - chemically induced | Weight Loss - drug effects | DNA-Binding Proteins - deficiency | Carboplatin - adverse effects | Dose-Response Relationship, Drug | Bendamustine Hydrochloride - administration & dosage | Pneumonia - chemically induced | Camptothecin - administration & dosage | Female | Camptothecin - analogs & derivatives | Lung Neoplasms - genetics | Camptothecin - adverse effects | Hematologic Diseases - chemically induced | Etoposide - adverse effects | Drug Administration Schedule | Carcinoma, Small Cell - drug therapy | Kaplan-Meier Estimate | Carboplatin - administration & dosage | Etoposide - administration & dosage | Treatment Outcome | DNA-Binding Proteins - genetics | Disease-Free Survival | Maximum Tolerated Dose | Mitosis - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Bendamustine Hydrochloride - pharmacology | Multiple Organ Failure - etiology | Brain Diseases, Metabolic - etiology | Aged | Biomarkers, Tumor - genetics | Gastrointestinal Diseases - chemically induced | Immunohistochemistry | Medicine, Experimental | Medical research | Lung cancer, Small cell | Cancer | Index Medicus
Journal Article
Journal Article
Expert Opinion in Biological Therapy, ISSN 1471-2598, 05/2005, Volume 5, Issue 5, pp. 691 - 702
Beta-glucans, biological response modifiers (BRMs) derived from the cell walls of yeast and other sources, have been demonstrated to prime leukocyte... 
Journal Article
Journal of Immunology, ISSN 0022-1767, 07/2004, Volume 173, Issue 2, pp. 797 - 806
Antitumor mAb bind to tumors and activate complement, coating tumors with iC3b. Intravenously administered yeast beta-1,3; 1,6-glucan functions as an adjuvant... 
Journal Article
Folia Histochemica et Cytobiologica, ISSN 0239-8508, 04/2007, Volume 45, Issue 2, p. 107
  Beta (1-3)-D-glucans were identified almost 40 years ago as biological response modifiers that stimulated tumor rejection. In vitro studies have shown that... 
Journal Article
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