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prostatic neoplasms - metabolism (5) 5
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antigens, nuclear - metabolism (4) 4
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Cancer Research, ISSN 0008-5472, 07/2018, Volume 78, Issue 13 Supplement, pp. 4265 - 4265
Journal Article
Cancer Research, ISSN 0008-5472, 07/2017, Volume 77, Issue 13 Supplement, pp. 4533 - 4533
Journal Article
Cancer Research, ISSN 0008-5472, 07/2016, Volume 76, Issue 14 Supplement, pp. 2707 - 2707
The purpose of this study is to quantitatively investigate the mechanism of androgen receptor (AR) and non-homologous end joining (NHEJ) interaction after and... 
Journal Article
Methods in Molecular Biology, ISSN 1064-3745, 2015, Volume 1219, pp. 1 - 9
Apoptosis can be measured by number of methods by taking advantage of the morphological, biochemical, and molecular changes undergoing in a cell during this... 
Immunohistochemistry | Irradiation | Flow cytometry | Caspase-3 | PARP-1 | Apoptosis | Flow Cytometry - methods | Humans | Caspase 3 - metabolism | Immunoblotting - methods | Immunohistochemistry - methods | Animals | Fluorescein | Caspase 3 - analysis | Mice | DNA Damage | Enzyme Activation | Fluorescent Dyes | Index Medicus
Journal Article
Cancer Biology & Therapy, ISSN 1538-4047, 09/2018, Volume 19, Issue 9, pp. 755 - 762
Apo2 ligand (Apo2L)/tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) is unique to selectively induce apoptosis in tumor cells while sparing... 
prostate cancer | autophagy | flux | p62/SQSTM1 | Apo2L/TRAIL | autophagosome | Apoptosis | ACTIVATION | TRAIL | CANCER-CELL-DEATH | CRYSTAL-STRUCTURE | RECEPTOR 5 | p62 | TNF FAMILY | BREAST-CANCER | Apo2L | APOPTOSIS-INDUCING LIGAND | NECROSIS | ONCOLOGY | TRAIL-INDUCED APOPTOSIS | CASPASE-8 | SQSTM1
Journal Article
by Klionsky, Daniel J and Abdalla, Fabio C and Abeliovich, Hagai and Abraham, Robert T and Acevedo-Arozena, Abraham and Adeli, Khosrow and Agholme, Lotta and Agnello, Maria and Agostinis, Patrizia and Aguirre-Ghiso, Julio A and Ahn, Hyung Jun and Ait-Mohamed, Ouardia and Ait-Si-Ali, Slimane and Akematsu, Takahiko and Akira, Shizuo and Al-Younes, Hesham M and Al-Zeer, Munir A and Albert, Matthew L and Albin, Roger L and Alegre-Abarrategui, Javier and Aleo, Maria Francesca and Alirezaei, Mehrdad and Almasan, Alexandru and Almonte-Becerril, Maylin and Amano, Atsuo and Amaravadi, Ravi K and Amarnath, Shoba and Amer, Amal O and Andrieu-Abadie, Nathalie and Anantharam, Vellareddy and Ann, David K and Anoopkumar-Dukie, Shailendra and Aoki, Hiroshi and Apostolova, Nadezda and Arancia, Giuseppe and Aris, John P and Asanuma, Katsuhiko and Asare, Nana Y.O and Ashida, Hisashi and Askanas, Valerie and Askew, David S and Auberger, Patrick and Baba, Misuzu and Backues, Steven K and Baehrecke, Eric H and Bahr, Ben A and Bai, Xue-Yuan and Bailly, Yannick and Baiocchi, Robert and Baldini, Giulia and Balduini, Walter and Ballabio, Andrea and Bamber, Bruce A and Bampton, Edward T.W and Juhász, Gábor and Bartholomew, Clinton R and Bassham, Diane C and Bast, Robert C and Batoko, Henri and Bay, Boon-Huat and Beau, Isabelle and Béchet, Daniel M and Begley, Thomas J and Behl, Christian and Behrends, Christian and Bekri, Soumeya and Bellaire, Bryan and Bendall, Linda J and Benetti, Luca and Berliocchi, Laura and Bernardi, Henri and Bernassola, Francesca and Besteiro, Sébastien and Bhatia-Kissova, Ingrid and Bi, Xiaoning and Biard-Piechaczyk, Martine and Blum, Janice S and Boise, Lawrence H and Bonaldo, Paolo and Boone, David L and Bornhauser, Beat C and Bortoluci, Karina R and Bossis, Ioannis and Bost, Frédéric and Bourquin, Jean-Pierre and Boya, Patricia and Boyer-Guittaut, Michaël and Bozhkov, Peter V and Brady, Nathan R and Brancolini, Claudio and Brech, Andreas and Brenman, Jay E and Brennand, Ana and Bresnick, Emery H and Brest, Patrick and Bridges, Dave and Bristol, Molly L and Brookes, Paul S and Brown, Eric J and Brumell, John H and ... and Linköpings universitet and Institutionen för klinisk och experimentell medicin and Geriatrik and Institutionen för medicin och hälsa and Farmakologi and Experimentell patologi and Hälsouniversitetet and Cellbiologi
Autophagy, ISSN 1554-8627, 04/2012, Volume 8, Issue 4, pp. 445 - 544
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, pp. e60408 - e60408
Exposure to genotoxic agents, such as irradiation produces DNA damage, the toxicity of which is augmented when the DNA repair is impaired. Poly (ADP-ribose)... 
CARCINOMA-CELLS | LYMPHOMA-CELLS | AG014699 | GENOTOXIC STRESS | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | HOMOLOGOUS RECOMBINATION | IONIZING-RADIATION | TUMORS | PROGRESSION | POLY(ADP-RIBOSE) POLYMERASE INHIBITOR | Transcriptional Regulator ERG | Cell Line | Cell Survival - drug effects | PTEN Phosphohydrolase - genetics | Prostatic Neoplasms - metabolism | Humans | Trans-Activators | Cellular Senescence - drug effects | Morpholines - pharmacology | PTEN Phosphohydrolase - metabolism | Male | Cell Survival - radiation effects | Recombinant Fusion Proteins | Poly(ADP-ribose) Polymerases - metabolism | Poly(ADP-ribose) Polymerases - genetics | Cellular Senescence - radiation effects | Fluorescent Antibody Technique | Cell Line, Tumor | Indoles - pharmacology | Serine Endopeptidases | Chromones - pharmacology | DNA damage | Analysis | Genetic aspects | Research | DNA repair | Prostate cancer | Cancer | Cell culture | Protein kinase C | Radiosensitivity | Homologous recombination | Lung cancer | Genotoxicity | Radiation | Galactosidase | Phosphatase | ADP | Mimicry | Proteins | Fusion protein | Deoxyribonucleic acid--DNA | Sensitivity analysis | BRCA1 protein | Gene expression | Survival | Radiation dosage | ERG gene | Inhibitors | Irradiation | Sensitivity enhancement | Lymphomas | Mutation | Radiation damage | ETS protein | Senescence | Toxicity | Homology | Biology | Activation | Ribose | Rodents | Cell cycle | Brachytherapy | BRCA2 protein | Cell survival | Poly(ADP-ribose) polymerase | Breast cancer | Radiation therapy | Polymerase | Sensitivity | DNA-dependent protein kinase | Cell lines | Prostate | PTEN protein | Tumors | Apoptosis | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
NATURE COMMUNICATIONS, ISSN 2041-1723, 08/2019, Volume 10, Issue 1, pp. 1 - 15
Breast cancer stem cells (BCSCs) are unique in their ability to undergo unlimited self-renewal, an essential process in breast cancer recurrence following... 
EPITHELIAL-MESENCHYMAL TRANSITION | GLYCOLYSIS | BREAST-CANCER CELLS | STEM-CELLS | MULTIDISCIPLINARY SCIENCES | GROWTH | MECHANISMS | FRUCTOSE 2,6-BISPHOSPHATE | CHLOROQUINE | CHEMOTHERAPY | PROGRESSION | Activation | Breast cancer | Metastasis | Kinases | Gene expression | Autophagy | Fructose | Metastases | Dormancy | Depletion | DNA microarrays | Stem cells | Breast | Disruption | Lesions | 6-Phosphofructo-2-kinase | Phagocytosis | Index Medicus
Journal Article
Journal Article
Journal Article
Methods in Molecular Biology, ISSN 1064-3745, 12/2007, Volume 414, pp. 13 - 21
Conference Proceeding
PLoS ONE, ISSN 1932-6203, 11/2014, Volume 9, Issue 11, pp. e111113 - e111113
Journal Article