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humans (5) 5
index medicus (5) 5
animals (4) 4
female (4) 4
ovarian cancer (4) 4
endoteli (3) 3
endothelium (3) 3
factor de creixement de l'endoteli vascular (3) 3
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1. Full Text The TGFβ pathway stimulates ovarian cancer cell proliferation by increasing IGF1R levels
by Alsina‐Sanchis, Elisenda and Figueras, Agnès and Lahiguera, Álvaro and Vidal, August and Casanovas, Oriol and Graupera, Mariona and Villanueva, Alberto and Viñals, Francesc
International Journal of Cancer, ISSN 0020-7136, 10/2016, Volume 139, Issue 8, pp. 1894 - 1903
In a search for new therapeutic targets for treating epithelial ovarian cancer, we analyzed the Transforming Growth Factor Beta (TGFβ) signaling pathway in... 
TGFβ | epithelial ovarian tumors | IGF1R | Epithelial ovarian tumors | INSULIN | PATHOGENESIS | LY2109761 | TARGET | ONCOLOGY | BIOLOGY | GROWTH-FACTOR | TGF beta | EXPRESSION | Neoplasms, Glandular and Epithelial - pathology | Antibodies, Monoclonal - pharmacology | Humans | Cell Proliferation - physiology | Ovarian Neoplasms - pathology | Neoplasms, Glandular and Epithelial - metabolism | Random Allocation | Pyrroles - pharmacology | Animals | Heterografts | Signal Transduction - drug effects | Mice, Nude | Receptors, Somatomedin - metabolism | Neoplasms, Glandular and Epithelial - drug therapy | Carcinoma, Ovarian Epithelial | Transforming Growth Factor beta - antagonists & inhibitors | Female | Mice | Ovarian Neoplasms - metabolism | Ovarian Neoplasms - drug therapy | Transforming Growth Factor beta - metabolism | Pyrazoles - pharmacology | Factors de creixement | Cell proliferation | Càncer d'ovari | Proliferació cel·lular | Cèl·lules epitelials | Insulin receptors | Epithelial cells | Receptors d'insulina | Growth factors | Ovarian cancer
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2. Full Text ALK1 Loss Results in Vascular Hyperplasia in Mice and Humans Through PI3K Activation
by Alsina-Sanchís, Elisenda and García-Ibáñez, Yaiza and Figueiredo, Ana M and Riera-Domingo, Carla and Figueras, Agnès and Matias-Guiu, Xavier and Casanovas, Oriol and Botella, Luisa M and Pujana, Miquel A and Riera-Mestre, Antoni and Graupera, Mariona and Viñals, Francesc
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 05/2018, Volume 38, Issue 5, pp. 1216 - 1229
OBJECTIVE—ALK1 (activin-receptor like kinase 1) is an endothelial cell-restricted receptor with high affinity for BMP (bone morphogenetic protein) 9 TGF-β... 
endothelium | ARTERIOVENOUS-MALFORMATIONS | HEREDITARY HEMORRHAGIC TELANGIECTASIA | PROTEIN | telangiectasia | ANGIOGENESIS | ENDOTHELIAL-CELL MIGRATION | mice | hereditary hemorrhagic | rare diseases | RECEPTOR-LIKE KINASE-1 | GENE | retina | MOLECULAR REGULATION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | humans | EXPRESSION | TYPE-2 | Growth Differentiation Factor 2 - pharmacology | Telangiectasia, Hereditary Hemorrhagic - pathology | Phosphatidylinositol 3-Kinase - antagonists & inhibitors | Humans | Hyperplasia | Neovascularization, Pathologic | Telangiectasia, Hereditary Hemorrhagic - genetics | Retinal Telangiectasis - genetics | Case-Control Studies | Gene Deletion | Phosphatidylinositol 3-Kinase - metabolism | Vascular Endothelial Growth Factor A - pharmacology | Genetic Predisposition to Disease | Signal Transduction | Mice, Inbred C57BL | Activin Receptors, Type I - deficiency | Cells, Cultured | Angiogenesis Inhibitors - pharmacology | Retinal Telangiectasis - enzymology | Telangiectasia, Hereditary Hemorrhagic - drug therapy | Telangiectasia, Hereditary Hemorrhagic - enzymology | Activin Receptors, Type I - genetics | Retinal Telangiectasis - drug therapy | Mice, Knockout | Phenotype | Animals | Human Umbilical Vein Endothelial Cells - enzymology | Activin Receptors, Type II - genetics | Retinal Telangiectasis - pathology | Human Umbilical Vein Endothelial Cells - pathology | Protein Kinase Inhibitors - pharmacology | Enzyme Activation | Mutation | Endothelial Cells - pathology | Endothelial Cells - enzymology | Endothelial Cells - drug effects | Proteïnes quinases | Rats as laboratory animals | Retina | Hemorrhage | Hemorràgia | Endothelium | Vascular endothelial growth factors | Vascular diseases | Factor de creixement de l'endoteli vascular | Malalties vasculars | Endoteli | Rates (Animals de laboratori) | Protein kinases
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3. Full Text A Role for CXCR4 in Peritoneal and Hematogenous Ovarian Cancer Dissemination
by Figueras, A and Alsina-Sanchis, E and Lahiguera, A and Abreu, M and Muinelo-Romay, L and Moreno-Bueno, G and Casanovas, O and Graupera, M and Matias-Guiu, X and Vidal, A and Villanueva, A and Vinals, F
MOLECULAR CANCER THERAPEUTICS, ISSN 1535-7163, 02/2018, Volume 17, Issue 2, pp. 532 - 543
Epithelial ovarian cancer is characterized by a low recovery rate because the disease is typically diagnosed at an advanced stage, by which time most patients... 
SURVIVAL | PROGNOSTIC-FACTOR | METASTASIS | INHIBITION | CISPLATIN | ONCOLOGY | GROWTH | RESISTANCE | TUMOR-CELLS | CHEMOKINE CXCL12 | AXIS | Signal Transduction | Humans | Middle Aged | Cell Proliferation - physiology | Ovarian Neoplasms - pathology | Receptors, CXCR4 - metabolism | Ovarian Neoplasms - genetics | Animals | Mice, Nude | Cell Line, Tumor | Female | Receptors, CXCR4 - genetics | Aged | Mice | Ovarian Neoplasms - metabolism | Retrospective Studies | Animal models | Phenotypes | Ovarian carcinoma | Mesenchyme | Metastasis | Gene expression | Patients | CXCR4 protein | Ovarian cancer | Metastases | Angiogenesis | Inhibition | Tumors | Peritoneum | Cancer
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4. Full Text TGFβ controls ovarian cancer cell proliferation
by Alsina-Sanchís, Elisenda and Figueras, Agnès and Lahiguera, Alvaro and Gil-Martín, Marta and Pardo, Beatriz and Piulats, Josep M and Martí, Lola and Ponce, Jordi and Matias-Guiu, Xavier and Vidal, August and Villanueva, Alberto and Viñals, Francesc
International Journal of Molecular Sciences, ISSN 1661-6596, 08/2017, Volume 18, Issue 8, p. 1658
There have been no major improvements in the overall survival of ovarian cancer patients in recent decades. Even though more accurate surgery and more... 
TGFβ | Proliferation | IGF1R | Ovarian cancer | Ovarian Neoplasms - genetics | Animals | Cell Proliferation | Transforming Growth Factor beta - genetics | Signal Transduction | Humans | Ovarian Neoplasms - pathology | Female | Ovarian Neoplasms - metabolism | Neoplasm Proteins - genetics | Neoplasm Proteins - metabolism | Transforming Growth Factor beta - metabolism | Cell proliferation | Signal transduction | Cell growth | Cell survival | Transforming growth factor | Surgery | Homeostasis | Disruption | Tumors | Cancer | Factors de creixement | Càncer d'ovari | Proliferació cel·lular | Growth factors | proliferation | ovarian cancer
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5. Full Text The TGF[beta] pathway stimulates ovarian cancer cell proliferation by increasing IGF1R levels
by Alsina-Sanchis, Elisenda and Figueras, Agnes and Lahiguera, Alvaro and Vidal, August and Casanovas, Oriol and Graupera, Mariona and Villanueva, Alberto and Vinals, Francesc
International Journal of Cancer, ISSN 0020-7136, 10/2016, Volume 139, Issue 8, p. 1894
  In a search for new therapeutic targets for treating epithelial ovarian cancer, we analyzed the Transforming Growth Factor Beta (TGF[beta]) signaling pathway... 
Viral antibodies | Tyrosine | RNA | Analysis | Antibodies | Transforming growth factors | Ovarian cancer | Cell growth | Growth factors | Cancer therapies | Tumors
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6. Full Text ARole forCXCR4in peritonealandhematogenous ovarian cancer dissemination
by Figueras, Agnès and Alsina-Sanchís, Elisenda and Lahiguera, Álvaro and Abreu, Manuel and Muinelo-Romay, Laura and Moreno-Bueno, Gema and Casanovas, Oriol and Graupera, Mariona and Matias-Guiu, Xavier and Vidal, August and Villanueva, Alberto and Viñals, Francesc
Molecular Cancer Therapeutics, ISSN 1535-7163, 02/2018, Volume 17, Issue 2, pp. 532 - 543
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7. Full Text The TGFβ pathway stimulates ovarian cancer cell proliferation by increasing IGF1R levels
: TGFΒ inhibition as alternative to anti-IGF1R treatment
by Alsina-Sanchis, Elisenda and Figueras, Agnès and Lahiguera, Álvaro and Vidal, August and Casanovas, Oriol and Graupera, Mariona and Villanueva, Alberto and Viñals, Francesc
International Journal of Cancer, ISSN 0020-7136, 10/2016, Volume 139, Issue 8, pp. 1894 - 1903
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8. Full Text ALK1 loss results in vascular hyperplasia in mice and humans through PI3K activation
by Alsina Sanchís, Elisenda and García-Ibáñez, Yaiza and Figueiredo, Ana Raquel Martins and Riera-Domingo, Carla and Figueras i Amat, Agnès and Matias-Guiu, Xavier and Casanovas i Casanovas, Oriol and Botella, Luisa M and Pujana Genestar, M. Ángel and Riera Mestre, Antoni and Graupera i Garcia-Milà, Mariona and Viñals Canals, Francesc
ISSN 1079-5642, 05/2018
Objective: ALK1 (activin-receptor like kinase 1) is an endothelial cell-restricted receptor with high affinity for BMP (bone morphogenetic protein) 9... 
Proteïnes quinases | Rats as laboratory animals | Retina | Hemorrhage | Hemorràgia | Endothelium | Vascular endothelial growth factors | Vascular diseases | Factor de creixement de l'endoteli vascular | Malalties vasculars | Endoteli | Rates (Animals de laboratori) | Protein kinases
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9. Full Text ALK1 loss results in vascular hyperplasia in mice and humans through PI3K activation
by Alsina Sanchís, Elisenda and García-Ibáñez, Yaiza and Figueiredo, Ana Raquel Martins and Riera-Domingo, Carla and Figueras i Amat, Agnès and Matias-Guiu, Xavier and Casanovas i Casanovas, Oriol and Botella, Luisa M and Pujana Genestar, M. Ángel and Riera Mestre, Antoni and Graupera i Garcia-Milà, Mariona and Viñals Canals, Francesc
ISSN 1079-5642, 05/2018
Objective: ALK1 (activin-receptor like kinase 1) is an endothelial cell-restricted receptor with high affinity for BMP (bone morphogenetic protein) 9... 
Proteïnes quinases | Rats as laboratory animals | Retina | Hemorrhage | Hemorràgia | Endothelium | Vascular endothelial growth factors | Vascular diseases | Factor de creixement de l'endoteli vascular | Malalties vasculars | Endoteli | Rates (Animals de laboratori) | Protein kinases
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10. Full Text Systematic mutation analysis of seven dystonia genes in complex regional pain syndrome with fixed dystonia
by Gosso, M. Florencia and De Rooij, Annetje M and Alsina-Sanchis, Elisenda and Kamphorst, Jessica T and Marinus, Johan and Van Hilten, Jacobus J and Van Den Maagdenberg, Arn J. M
Journal of Neurology, ISSN 0340-5354, 05/2010, Volume 257, Issue 5, pp. 820 - 824
Complex regional pain syndrome type 1 (CRPS-1) is a chronic pain disorder that in some patients is associated with fixed dystonia. The pathogenesis of CRPS and... 
Fixed dystonia | Complex regional pain syndrome | DYT genes | TORSION DYSTONIA | PROTEIN | ONSET DYSTONIA | IDENTIFICATION | CLINICAL NEUROLOGY | THAP1 | PERIPHERAL-NERVE | GTP CYCLOHYDROLASE | INFLAMMATION | REFLEX SYMPATHETIC DYSTROPHY | PLASTICITY | Dystonia - genetics | Phenotype | Reflex Sympathetic Dystrophy - genetics | DNA Mutational Analysis | Humans | Age of Onset | Adult | Female | Male | Mutation | Complications and side effects | Gene mutations | Causes of | Genetic aspects | Research | Dystonia | Health aspects | Chronic pain | Risk factors
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