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BioEssays, ISSN 0265-9247, 07/2016, Volume 38, Issue 7, pp. S56 - S64
Journal Article
Science Translational Medicine, ISSN 1946-6234, 01/2019, Volume 11, Issue 477, p. eaaw5320
High somatic tumor mutational burden predicts response to immune checkpoint inhibitors across multiple cancers. 
Journal Article
Science Translational Medicine, ISSN 1946-6234, 11/2018, Volume 10, Issue 469, p. eaav9148
Obesity promotes PD-1–mediated T cell dysfunction but also improves tumor response to immune checkpoint blockade. 
Journal Article
Science Translational Medicine, ISSN 1946-6234, 10/2018, Volume 10, Issue 461, p. eaav3884
Hepatic microenvironment dictates lineage commitment of hepatocytes, which can form two completely distinct tumor types. 
Journal Article
Science Translational Medicine, ISSN 1946-6234, 08/2018, Volume 10, Issue 453, p. eaau7386
Journal Article
Science Translational Medicine, ISSN 1946-6234, 06/2018, Volume 10, Issue 445, p. eaau0469
Journal Article
Science Translational Medicine, ISSN 1946-6234, 04/2018, Volume 10, Issue 437, p. eaat4477
ROS1 inhibition in E-cadherin–deficient tumors leads to synthetic lethality, which could be exploited for the selective treatment of breast cancer patients. 
Journal Article
Science Translational Medicine, ISSN 1946-6234, 02/2018, Volume 10, Issue 429, p. eaas8964
Journal Article
BioEssays, ISSN 0265-9247, 07/2016, Volume 38, pp. S56 - S64
Journal Article
Gut, ISSN 0017-5749, 09/2019, Volume 68, Issue 9, pp. 1543 - 1544
Journal Article
Current Opinion in Genetics & Development, ISSN 0959-437X, 02/2019, Volume 54, pp. 17 - 24
Cellular senescence is implicated in numerous biological processes, and can play pleiotropic, sometimes opposing, roles in cancer. Several triggers, cell... 
CELLS | CLEARANCE | APOPTOSIS | ONCOGENE | TRANSCRIPTOME | GENETICS & HEREDITY | SURVEILLANCE | CGAS | IDENTIFICATION | CELLULAR SENESCENCE | P53 | CELL BIOLOGY
Journal Article
JOURNAL OF EXPERIMENTAL MEDICINE, ISSN 0022-1007, 04/2019, Volume 216, Issue 4, pp. 723 - 724
In this issue of , Muto et al. (https://doi.org/10.1084/jem.20180900) generate a novel mouse model of liver cancer induced by iron overload by deleting the... 
MEDICINE, RESEARCH & EXPERIMENTAL | IMMUNOLOGY | 314 | 307
Journal Article
Nature, ISSN 0028-0836, 2016, Volume 534, Issue 7609, pp. 647 - 651
Therapeutic targeting of KRAS-mutant lung adenocarcinoma represents a major goal of clinical oncology. KRAS itself has proved difficult to inhibit, and the... 
COLON-CANCER | SELECTIVE INHIBITOR | POTENT | FEEDBACK ACTIVATION | MEK INHIBITION | MODELS | RAS | MULTIDISCIPLINARY SCIENCES | ACQUIRED-RESISTANCE | MEDIATED REACTIVATION | RECEPTOR TYROSINE KINASE | Lung Neoplasms - drug therapy | Colonic Neoplasms - genetics | Adenocarcinoma - pathology | Adenocarcinoma of Lung | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Drug Resistance, Neoplasm | Lung Neoplasms - pathology | Pyridazines - pharmacology | Female | Pyrimidinones - pharmacology | Adenocarcinoma - genetics | Cell Death - drug effects | Imidazoles - therapeutic use | Phosphorylation - drug effects | Pyridazines - therapeutic use | Disease Models, Animal | Lung Neoplasms - genetics | Pancreatic Neoplasms - pathology | Pyrimidinones - therapeutic use | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Mutant Proteins - genetics | Antineoplastic Combined Chemotherapy Protocols | Pancreatic Neoplasms - genetics | Imidazoles - pharmacology | Enzyme Activation - drug effects | Adenocarcinoma - drug therapy | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Xenograft Model Antitumor Assays | Feedback, Physiological | Animals | MAP Kinase Signaling System - drug effects | Colonic Neoplasms - pathology | Cell Proliferation - drug effects | Mice | Pyridones - therapeutic use | Mutation | Pyridones - pharmacology | Drug Screening Assays, Antitumor | Molecular targeted therapy | Care and treatment | Cell death | Lung cancer | Methods | Cancer cells | Phosphorylation | Chemotherapy | Inhibitor drugs | Drug resistance | Tumors
Journal Article
Nature Cell Biology, ISSN 1465-7392, 09/2017, Volume 19, Issue 9, pp. 1008 - 1009
Cellular senescence, a cell-autonomous growth arrest program, also executes pleiotropic non-cell-autonomous activities through the senescence-associated... 
CGAS | SECRETION | CELLULAR SENESCENCE | CELL BIOLOGY | Cellular Senescence - genetics | Phenotype | Cell Proliferation | DNA Damage | DNA | Humans | Aging | Cellular signal transduction | Genetic aspects | Health aspects | DNA damage | Cells | Senescence | Deoxyribonucleic acid--DNA
Journal Article
Genes and Development, ISSN 0890-9369, 08/2014, Volume 28, Issue 16, pp. 1800 - 1814
Journal Article
Journal Article