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Journal Article
Journal Article
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 2005, Volume 337, Issue 1, pp. 184 - 190
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 03/2005, Volume 168, Issue 7, p. 1087
  Using fluorescent variants of Fas and FasL, we show that membrane FasL and Fas form supramolecular clusters that are of flexible shape, but nevertheless... 
Signal transduction | Membranes | Cellular biology | Fluorescence
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 08/2005, Volume 25, Issue 15, pp. 6363 - 6379
FasL and gamma interferon (IFN- gamma ) are produced by activated T cells and NK cells and synergistically induce apoptosis. Although both cytokines can also... 
Journal Article
Univ., Diss., 2007--Würzburg. 
Dissertation
Blood, ISSN 0006-4971, 04/2014, Volume 123, Issue 16, pp. 2562 - 2572
Loss of inhibitor of apoptosis proteins (IAPs), particularly cIAP1, can promote production of tumor necrosis factor (TNF) and sensitize cancer cell lines to... 
RIP1 KINASE | ACTIVATION | NECROSIS | NECROPTOSIS | ALPHA-DEPENDENT APOPTOSIS | CASPASE-8 | TNF-ALPHA | ANTAGONISTS | HEMATOLOGY | NF-KAPPA-B | CELL-DEATH
Journal Article
Journal of Biomolecular Screening, ISSN 1087-0571, 8/2012, Volume 17, Issue 7, pp. 933 - 945
Journal Article
Blood, ISSN 0006-4971, 07/2015, Volume 126, Issue 4, pp. 437 - 444
Inhibition of the tumor necrosis factor (TNF)-like weak inducer of apoptosis (TWEAK)/fibroblast growth factor-inducible 14 (Fn14) system reduces intestinal... 
WEAK INDUCER | EPITHELIAL-CELLS | BONE-MARROW-TRANSPLANTATION | IN-VIVO | TNF-ALPHA | TWEAK/FN14 INTERACTION | HEMATOLOGY | VERSUS-HOST-DISEASE | ANTITUMOR ACTIVITIES | GAMMA RECEPTOR ENGAGEMENT | TUMOR ACTIVITY | Humans | Male | Intestines - metabolism | Graft vs Host Disease - metabolism | Luminescent Measurements | TWEAK Receptor | Intestines - immunology | Receptors, IgG - metabolism | Immunoglobulin G - immunology | Receptors, Tumor Necrosis Factor - antagonists & inhibitors | Hematopoietic Stem Cell Transplantation - adverse effects | Female | Graft vs Host Disease - etiology | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Receptors, Tumor Necrosis Factor - genetics | Receptors, Tumor Necrosis Factor - metabolism | Intestines - pathology | Antibody-Dependent Cell Cytotoxicity | Mice, Inbred C57BL | RNA, Messenger - genetics | Cells, Cultured | Tumor Necrosis Factors - metabolism | Rituximab | Receptors, IgG - immunology | Reverse Transcriptase Polymerase Chain Reaction | Tumor Necrosis Factors - immunology | Immunoglobulin G - administration & dosage | Blotting, Western | Graft vs Host Disease - pathology | Tumor Necrosis Factor-alpha - pharmacology | Cytokine TWEAK | Animals | Fluorescent Antibody Technique | Graft vs Host Disease - prevention & control | Mice | Mice, Inbred BALB C | Receptors, Tumor Necrosis Factor - immunology | Antibodies, Monoclonal, Murine-Derived - therapeutic use | Apoptosis | Tumor Necrosis Factors - antagonists & inhibitors | Plenary Paper
Journal Article
Journal of Molecular Medicine, ISSN 0946-2716, 7/2010, Volume 88, Issue 7, pp. 729 - 740
Journal Article
CELL DEATH & DISEASE, ISSN 2041-4889, 08/2019, Volume 10, Issue 8, pp. 611 - 15
The NEDD8-activating enzyme (NAE) inhibitor MLN4924 inhibits cullin-RING ubiquitin ligase complexes including the SKP1-cullin-F-box E3 ligase beta TrCP.... 
ACTIVATION | CYTOKINES | PROTEIN | PEVONEDISTAT TAK-924/MLN4924 | C-IAP2 | NECROSIS-FACTOR-ALPHA | PROLIFERATION | NF-KAPPA-B | PHASE-I | CANCER | CELL BIOLOGY | Tumor necrosis factor receptors | Ubiquitin | Enzymes | Multiple myeloma | Tumor cell lines | Gene expression | Cell surface | Cullin | Cell death | Tumor necrosis factor | Cell lines | Ubiquitin-protein ligase | Apoptosis
Journal Article
Cellular and Molecular Life Sciences, ISSN 1420-682X, 1/2014, Volume 71, Issue 2, pp. 331 - 348
Programmed necrosis is important in many (patho)physiological settings. For specific therapeutic intervention, however, a better knowledge is required whether... 
Life Sciences | Biochemistry, general | Life Sciences, general | Necroptosis | TNF | PARP-1 | Biomedicine general | Programmed necrosis | Cell Biology | APOPTOSIS | OXIDATIVE STRESS | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | PARP | CELL BIOLOGY | INHIBITION | RECEPTOR-INTERACTING PROTEIN | POLYMERASE-1 | CASPASES | EXPRESSION | Free Radical Scavengers - pharmacology | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Calpain - metabolism | Apoptosis - drug effects | Humans | Antineoplastic Agents, Alkylating - pharmacology | Guanidines - pharmacology | JNK Mitogen-Activated Protein Kinases - metabolism | Necrosis | MCF-7 Cells | RNA Interference | HEK293 Cells | Benzamides - pharmacology | Cell Line | Ceramides - metabolism | Jurkat Cells | Nuclear Pore Complex Proteins - metabolism | Piperazines - pharmacology | Poly(ADP-ribose) Polymerase Inhibitors | HT29 Cells | Phthalazines - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Poly(ADP-ribose) Polymerases - metabolism | Animals | Poly(ADP-ribose) Polymerases - genetics | Cathepsins - metabolism | Mice | HeLa Cells | Methyl Methanesulfonate - pharmacology | RNA-Binding Proteins - metabolism | RNA, Small Interfering - metabolism | Proteases | Tumor necrosis factor | Cell death | Biochemistry | Phosphotransferases | Sugars | Monosaccharides | Proteins | Gangrene | Signal transduction | Apoptosis
Journal Article