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Cell, ISSN 0092-8674, 03/2018, Volume 173, Issue 1, pp. 104 - 116.e12
Human diseases are often caused by loss of somatic cells that are incapable of re-entering the cell cycle for regenerative repair. Here, we report a... 
heart failure | cyclin | proliferation | cell cycle | regeneration | cytokinesis | cardiomyocyte | cell division | CDK | heart | HYPERTROPHY | INFARCTION | EXPRESSION ANALYSIS | AMPLIFICATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MICE | ZEBRAFISH HEART REGENERATION | FAILURE | CANCER | FIBROBLASTS | PROGRAM | CELL BIOLOGY | Cyclin D1 - metabolism | Cell Proliferation | Protein-Tyrosine Kinases - metabolism | Cyclin-Dependent Kinase 4 - genetics | Heart - physiology | Humans | Myosin Heavy Chains - genetics | Cyclin B1 - metabolism | CDC2 Protein Kinase - metabolism | Cell Cycle Proteins - antagonists & inhibitors | Myocardial Infarction - pathology | Transforming Growth Factor beta - antagonists & inhibitors | Myocardial Infarction - veterinary | Induced Pluripotent Stem Cells - cytology | Induced Pluripotent Stem Cells - metabolism | CDC2 Protein Kinase - genetics | Myocytes, Cardiac - cytology | Mice, Inbred C57BL | Cell Cycle Proteins - metabolism | Cyclin B1 - genetics | Rats | Mice, Transgenic | Nuclear Proteins - metabolism | Myocardial Infarction - metabolism | Cyclin-Dependent Kinase 4 - metabolism | Cytokinesis | Regeneration | Animals | Cyclin D1 - genetics | Nuclear Proteins - antagonists & inhibitors | Myocytes, Cardiac - metabolism | Mice | Transforming Growth Factor beta - metabolism | Protein-Tyrosine Kinases - antagonists & inhibitors | Heart | Bone morphogenetic proteins | Transforming growth factors | Cell cycle | Stem cells | Cell Cycle | Cardiomyocyte | Cell Division | Cyclin
Journal Article
Circulation, ISSN 0009-7322, 03/2017, Volume 135, Issue 10, pp. 978 - 995
BACKGROUND:Reprogramming of cardiac fibroblasts into induced cardiomyocyte-like cells in situ represents a promising strategy for cardiac regeneration. A... 
Heart | Regeneration | Transcription factors | Cell differentiation | MOUSE FIBROBLASTS | cell differentiation | DEXAMETHASONE | CARDIAC & CARDIOVASCULAR SYSTEMS | EXPRESSION ANALYSIS | regeneration | transcription factors | HEART-FAILURE | CATENIN | heart | PLURIPOTENT STEM-CELLS | GROWTH-FACTOR-BETA | INHIBITION | AMPLIFICATION | FUNCTIONAL CARDIOMYOCYTES | PERIPHERAL VASCULAR DISEASE | Dioxoles - therapeutic use | MEF2 Transcription Factors - genetics | Heterocyclic Compounds, 3-Ring - pharmacology | Humans | Wnt Proteins - metabolism | Benzamides - therapeutic use | Dioxoles - pharmacology | Transforming Growth Factor beta - antagonists & inhibitors | Benzamides - pharmacology | Fibroblasts - metabolism | GATA4 Transcription Factor - metabolism | Myocytes, Cardiac - cytology | Cells, Cultured | GATA4 Transcription Factor - genetics | Myocardium - pathology | Transcription Factors - genetics | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Heart - diagnostic imaging | Cellular Reprogramming - drug effects | Transcription Factors - metabolism | Magnetic Resonance Imaging | Animals | Myocardial Infarction - drug therapy | Fibroblasts - drug effects | Heterocyclic Compounds, 3-Ring - therapeutic use | MEF2 Transcription Factors - metabolism | Myocytes, Cardiac - metabolism | Fibroblasts - cytology | Mice | Wnt Proteins - antagonists & inhibitors | Transforming Growth Factor beta - metabolism | Care and treatment | Heart cells | Dosage and administration | Research | Transforming growth factors | Heart diseases | reprogramming | cardiac differentiation | calcium | heart regeneration
Journal Article
Nature, ISSN 0028-0836, 06/2010, Volume 465, Issue 7299, pp. 808 - 812
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2016, Volume 22, Issue 10, pp. 1131 - 1139
Epigenetic reprogramming is a critical process of pathological gene induction during cardiac hypertrophy and remodeling, but the underlying regulatory... 
MEDICINE, RESEARCH & EXPERIMENTAL | CARDIOMYOCYTE HYPERTROPHY | PRESSURE-OVERLOAD | PATHOLOGICAL HYPERTROPHY | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | HISTONE METHYLATION | MTOR | CELL BIOLOGY | RAPTOR | GENE-EXPRESSION | BINDING | EZH2 | TOR Serine-Threonine Kinases - metabolism | Humans | Immunoblotting | Gene Expression Profiling | Gene Knockdown Techniques | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - metabolism | Chromatin Immunoprecipitation | Computer Simulation | Myocardium - metabolism | Real-Time Polymerase Chain Reaction | Echocardiography | Rats | In Situ Hybridization, Fluorescence | RNA, Long Noncoding - genetics | Reverse Transcriptase Polymerase Chain Reaction | Heart - diagnostic imaging | Mice, Knockout | Blotting, Northern | Animals | Epigenesis, Genetic - genetics | Myocytes, Cardiac - metabolism | Mice | Cardiomegaly - genetics | In Vitro Techniques | Methylation | Polycomb Repressive Complex 2 - metabolism | Induced Pluripotent Stem Cells | Histone Code - genetics | Cardiomegaly - metabolism | Genetic aspects | Research | RNA | Heart enlargement | Heart | Epigenetic inheritance | Antisense RNA | Genes | TOR protein | Chromatin | Rapamycin | Gene expression | Ribonucleic acid--RNA | Polycomb group proteins | Lysine | Regulatory mechanisms (biology) | DNA methylation | Epigenetics | Catalysis | Inhibition | Histone H3 | Hypertrophy
Journal Article
Cell, ISSN 0092-8674, 12/2016, Volume 167, Issue 7, pp. 1734 - 1749.e22
Journal Article
Trends in Pharmacological Sciences, ISSN 0165-6147, 2011, Volume 32, Issue 7, pp. 394 - 401
Journal Article