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Nature, ISSN 0028-0836, 01/2010, Volume 463, Issue 7279, pp. 318 - 325
The inference of transcriptional networks that regulate transitions into physiological or pathological cellular states remains a central challenge in systems... 
SURVIVAL | GROWTH-FACTOR RECEPTOR | NEURAL STEM-CELLS | GLIOBLASTOMA | MULTIDISCIPLINARY SCIENCES | DISEASE | C-MYC | MECHANISMS | DIFFERENTIATION | EXPRESSION | BINDING | Neurons - pathology | Prognosis | Glioma - diagnosis | Humans | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Gene Regulatory Networks | Glioma - genetics | Cell Differentiation - genetics | Cell Transformation, Neoplastic - genetics | Neoplasm Invasiveness - pathology | Glioma - pathology | Transcription, Genetic | Neurons - metabolism | Cellular Reprogramming - genetics | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Reproducibility of Results | Brain Neoplasms - diagnosis | CCAAT-Enhancer-Binding Protein-beta - genetics | Computational Biology | Brain Neoplasms - genetics | Mesenchymal Stromal Cells - metabolism | Mice, SCID | Cell Transformation, Neoplastic - metabolism | CCAAT-Enhancer-Binding Protein-beta - metabolism | Animals | Cell Line, Tumor | Mice, Inbred NOD | Mesoderm - metabolism | Mice | Mesenchymal Stromal Cells - pathology | Cell Transformation, Neoplastic - pathology | Neoplasm Invasiveness - genetics | Mesoderm - pathology | Transcription factors | Gliomas | Physiological aspects | Genetic aspects | Research | Genetic transcription | Risk factors | Proteins | Genotype & phenotype | Brain | Reverse engineering | Brain cancer | Regression analysis | Gene expression | Index Medicus
Journal Article
Nature Genetics, ISSN 1061-4036, 07/2016, Volume 48, Issue 7, pp. 768 - 776
Glioblastoma (GBM) is the most common and aggressive primary brain tumor. To better understand how GBM evolves, we analyzed longitudinal genomic and... 
GENE FUSIONS | TGF-BETA | CANCER GENOMICS | LANDSCAPE | GLIOMA | GENETICS & HEREDITY | GENOMIC ALTERATIONS | MSH6 MUTATIONS | SEQUENCING DATA | BRAIN-TUMORS | TEMOZOLOMIDE | Cell Proliferation | Dacarbazine - therapeutic use | Genomics | Humans | Brain Neoplasms - pathology | DNA Repair Enzymes - genetics | Gene Expression Regulation, Neoplastic | Transcriptome | Neoplasm Recurrence, Local - drug therapy | Clonal Evolution - genetics | Neoplasm Recurrence, Local - pathology | Neoplasm Grading | Glioblastoma - genetics | Tumor Suppressor Proteins - genetics | Dacarbazine - analogs & derivatives | Brain Neoplasms - genetics | Isocitrate Dehydrogenase - genetics | Survival Rate | Brain Neoplasms - drug therapy | Mutation - genetics | Antineoplastic Agents, Alkylating - therapeutic use | Latent TGF-beta Binding Proteins - genetics | DNA Modification Methylases - genetics | Transforming Growth Factor beta - genetics | Glioblastoma - pathology | Neoplasm Recurrence, Local - genetics | Biomarkers, Tumor - genetics | Glioblastoma - drug therapy | Longitudinal Studies | Molecular targeted therapy | Care and treatment | Gene mutations | Development and progression | Genetic aspects | Genetic transcription | Glioblastoma multiforme | Health aspects | Methods | Brain cancer | Genomes | Gene expression | Cancer therapies | Studies | Archives & records | DNA methylation | Mathematical models | Mutation | Deoxyribonucleic acid--DNA | Tumors | Cancer | Index Medicus
Journal Article
Science, ISSN 0036-8075, 9/2012, Volume 337, Issue 6099, pp. 1231 - 1235
The brain tumor glioblastoma multiforme (GBM) is among the most lethal forms of human cancer. Here, we report that a small subset of GBMs (3.1%; 3 of 97 tumors... 
Exons | Neurons | Genes | REPORTS | Stem cells | Aneuploidy | Chromosomes | Cells | Tumors | Daughter cells | Cancer | ANEUPLOIDY | SELECTIVE INHIBITOR | POTENT | MULTIDISCIPLINARY SCIENCES | CANCER | RECEPTOR TYROSINE KINASE | DISCOVERY | CHROMOSOMAL INSTABILITY | FAMILY | Microtubule-Associated Proteins - chemistry | Neoplasm Transplantation | Translocation, Genetic | Oncogene Proteins, Fusion - metabolism | Receptor, Fibroblast Growth Factor, Type 3 - chemistry | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Mitosis | Humans | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Receptor, Fibroblast Growth Factor, Type 3 - antagonists & inhibitors | Fetal Proteins - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Receptor, Fibroblast Growth Factor, Type 3 - metabolism | Brain Neoplasms - metabolism | Oncogene Proteins, Fusion - chemistry | Spindle Apparatus - metabolism | Glioblastoma - genetics | Oncogene Fusion | Glioblastoma - metabolism | Antineoplastic Agents - pharmacology | Benzamides - pharmacology | Nuclear Proteins - genetics | Chromosomal Instability | Pyrazoles - pharmacology | Protein Structure, Tertiary | Receptor, Fibroblast Growth Factor, Type 3 - genetics | Enzyme Inhibitors - pharmacology | Brain Neoplasms - genetics | Nuclear Proteins - metabolism | Pyrimidines - pharmacology | Nuclear Proteins - chemistry | Piperazines - pharmacology | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Xenograft Model Antitumor Assays | Animals | Cell Transformation, Neoplastic | Oncogene Proteins, Fusion - genetics | Fetal Proteins - genetics | Mice | Receptor, Fibroblast Growth Factor, Type 1 - chemistry | Fetal Proteins - chemistry | Physiological aspects | Development and progression | Fibroblast growth factors | Genetic aspects | Research | Health aspects | Glioblastoma multiforme | Proteins | Kinases | Brain cancer | Genomics | Pharmaceutical sciences | Index Medicus
Journal Article
Journal Article
Immunity, ISSN 1074-7613, 04/2015, Volume 42, Issue 4, pp. 731 - 743
Microbiota-mediated effects on the host immune response facilitate colonization resistance against pathogens. However, it is unclear whether and how the host... 
ROR-GAMMA-T | ARYL-HYDROCARBON RECEPTOR | HUMAN GUT MICROBIOTA | TRANSCRIPTION | NOTCH | MUCOSAL INFECTION | INTERLEUKIN-22 | IMMUNOLOGY | CITROBACTER-RODENTIUM | PROTEINS | TISSUE INDUCER CELLS | Germ-Free Life - immunology | Enterobacteriaceae Infections - immunology | Interleukins - genetics | Lymphocytes - immunology | Inhibitor of Differentiation Protein 2 - deficiency | Enterobacteriaceae Infections - genetics | Inhibitor of Differentiation Protein 2 - genetics | Interleukins - immunology | Cell Differentiation | Citrobacter rodentium - immunology | Microbiota - immunology | Receptors, Interleukin - immunology | Signal Transduction | Enterobacteriaceae Infections - microbiology | Mice, Inbred C57BL | Gene Expression Regulation | Receptors, Aryl Hydrocarbon - genetics | Homeostasis - immunology | Inhibitor of Differentiation Protein 2 - immunology | Enterobacteriaceae Infections - pathology | Lymphocytes - microbiology | Immunity, Innate | Receptors, Interleukin - genetics | Mice, Knockout | Lymphocytes - pathology | Animals | Mice | Receptors, Aryl Hydrocarbon - immunology | Drug resistance in microorganisms | Pathogenic microorganisms | Microbiota (Symbiotic organisms) | Immunotherapy | Proteins | Studies | Flow cytometry | Transcription factors | E coli | Rodents | Gram-positive bacteria | Homeostasis | Infections | Deoxyribonucleic acid--DNA | Index Medicus
Journal Article
Journal Article
Neuro-Oncology, ISSN 1522-8517, 11/2015, Volume 17, Issue suppl 5, pp. v26.2 - v26
Journal Article
EMBO reports, ISSN 1469-221X, 04/2014, Volume 15, Issue 4, pp. 324 - 325
A study in this issue reveals an unexpected neurogenic function of MYC in the intact‐polarized chick neural tube. It shows that M yc represses N otch signaling... 
N-MYC | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | Neural Stem Cells - physiology | Avian Proteins - physiology | Proto-Oncogene Proteins c-myc - physiology | Animals | Oncogene Protein p55(v-myc) - physiology | Index Medicus | Hot off the Press
Journal Article
Nature, ISSN 0028-0836, 01/2016, Volume 529, Issue 7585, pp. 172 - 177
Mechanisms that maintain cancer stem cells are crucial to tumour progression. The ID2 protein supports cancer hallmarks including the cancer stem cell state.... 
PROGENITOR CELLS | LOOP-HELIX PROTEINS | NEURAL STEM-CELLS | TRANSCRIPTIONAL ACTIVITY | MULTIDISCIPLINARY SCIENCES | DOWN-SYNDROME | NEURONAL DIFFERENTIATION | PROTEIN-KINASE DYRK1A | INHIBITS PROLIFERATION | UBIQUITIN LIGASE HUWE1 | EXPRESSION | Phosphorylation | Protein-Tyrosine Kinases - metabolism | Humans | Male | Oxygen - metabolism | Cell Hypoxia | Ubiquitination | Basic Helix-Loop-Helix Transcription Factors - metabolism | Neoplastic Stem Cells - metabolism | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Cullin Proteins - metabolism | Neoplastic Stem Cells - pathology | Glioblastoma - metabolism | Von Hippel-Lindau Tumor Suppressor Protein - antagonists & inhibitors | Von Hippel-Lindau Tumor Suppressor Protein - metabolism | Protein-Serine-Threonine Kinases - metabolism | Inhibitor of Differentiation Protein 2 - metabolism | Phosphothreonine - metabolism | Xenograft Model Antitumor Assays | Animals | Hypoxia-Inducible Factor-Proline Dioxygenases - metabolism | Glioblastoma - pathology | Cell Line, Tumor | Protein Binding | Mice | Protein-Tyrosine Kinases - antagonists & inhibitors | Complications and side effects | Stem cell research | Care and treatment | Gliomas | Development and progression | Influence | Research | Phosphotransferases | Proteins | Genes | Stem cells | Hypoxia | Tumorigenesis | Kinases | Tumors | Cancer | Index Medicus
Journal Article