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Journal Article
Journal Article
Cancer Cell, ISSN 1535-6108, 07/2008, Volume 14, Issue 1, pp. 79 - 89
Twist1 and Twist2 are major regulators of embryogenesis. Twist1 has been shown to favor the metastatic dissemination of cancer cells through its ability to... 
CELLBIO | HUMDISEASE | SIGNALING | BREAST-CANCER | TRANSFORMATION | DORSOVENTRAL PATTERN | ONCOGENIC RAS | ONCOLOGY | IN-VIVO | ZYGOTIC GENE | SUPPRESSION | EXPRESSION | TUMORIGENESIS | CELL IMMORTALIZATION | Neoplasms - metabolism | Up-Regulation | Fibroblasts - enzymology | Epithelial Cells - metabolism | Humans | Gene Expression Regulation, Neoplastic | ras Proteins - metabolism | Transplantation, Heterologous | Cell Transdifferentiation - genetics | Mammary Glands, Human - metabolism | Transfection | Neoplasms - genetics | RNA Interference | Cell Transformation, Neoplastic - genetics | Mice, Inbred DBA | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Fibroblasts - metabolism | Cell Line | Cellular Senescence - genetics | Neoplasm Invasiveness | Mice, Inbred C57BL | Retinoblastoma Protein - metabolism | Tumor Suppressor Protein p53 - metabolism | Neoplasms - enzymology | Repressor Proteins - genetics | Epithelial Cells - pathology | Mice, Transgenic | Nuclear Proteins - metabolism | Fibroblasts - pathology | Cell Transformation, Neoplastic - metabolism | Animals | Mice, Nude | Dogs | Epithelial Cells - enzymology | Twist-Related Protein 1 - genetics | Mice | Enzyme Activation | Cell Transformation, Neoplastic - pathology | Neoplasms - pathology | Twist-Related Protein 1 - metabolism | Tumor proteins | Stem cells | Tumors | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e92254 - e92254
Journal Article
Nature Immunology, ISSN 1529-2908, 07/2009, Volume 10, Issue 7, pp. 778 - 785
Studies have suggested involvement of interleukin 17 (IL-17) in autoimmune diseases, although its effect on B cell biology has not been clearly established.... 
SURVIVAL | RECEPTOR STIMULATION | TOLERANCE CHECKPOINTS | INDUCED APOPTOSIS | GENE-EXPRESSION | LYMPHOCYTE STIMULATOR | IMMUNOLOGY | NF-KAPPA-B | UP-REGULATION | T-HELPER-CELLS | CUTTING EDGE | Apoptosis - drug effects | Minor Histocompatibility Antigens | Humans | Immunoblotting | Male | NF-kappa B - metabolism | Lupus Erythematosus, Systemic - blood | Interleukin-17 - pharmacology | Dose-Response Relationship, Drug | Immunoglobulins - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Time Factors | Female | Nuclear Proteins - genetics | B-Lymphocytes - metabolism | Repressor Proteins - metabolism | Interleukin-17 - blood | Cell Survival - drug effects | B-Lymphocytes - cytology | Cells, Cultured | Interleukin-17 - genetics | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Drug Synergism | B-Lymphocytes - drug effects | Cell Differentiation - drug effects | Twist-Related Protein 1 - genetics | Cell Line, Tumor | Antigens, CD19 - metabolism | B-Cell Activating Factor - pharmacology | Cell Proliferation - drug effects | Twist-Related Protein 1 - metabolism | Lupus Erythematosus, Systemic - pathology | Proto-Oncogene Proteins c-bcl-2 - genetics | Systemic lupus erythematosus | Interleukins | Physiological aspects | Genetic aspects | B cells | Research | DNA binding proteins | Health aspects | Risk factors | Index Medicus | Life Sciences | Other
Journal Article
Molecular Cancer, ISSN 1476-4598, 09/2014, Volume 13, Issue 1, pp. 213 - 213
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, pp. e29954 - e29954
Metastasis is a multistep process and the main cause of mortality in lung cancer patients. We previously showed that EGFR mutations were associated with a copy... 
CANCER-CELLS | OVEREXPRESSION | METASTASIS | TUMOR PROGRESSION | MODELS | ONCOGENIC COOPERATION | GROWTH | BIOLOGY | GENE-EXPRESSION | MUTATIONS | UP-REGULATION | Immunohistochemistry | Receptor, Epidermal Growth Factor - genetics | Adenocarcinoma - pathology | Cadherins - metabolism | Humans | Lung Neoplasms - metabolism | Middle Aged | Gene Expression Regulation, Neoplastic | Epithelial-Mesenchymal Transition - drug effects | Lung Neoplasms - pathology | Male | Epithelial-Mesenchymal Transition - genetics | Antigens, CD - genetics | Cell Movement - genetics | Antigens, CD - metabolism | Receptor, Epidermal Growth Factor - metabolism | Adenocarcinoma - metabolism | RNA Interference | Aged, 80 and over | Female | Adenocarcinoma - genetics | Cadherins - genetics | Nuclear Proteins - genetics | Lung Neoplasms - genetics | Kaplan-Meier Estimate | Nuclear Proteins - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Twist-Related Protein 1 - genetics | Cell Line, Tumor | Aged | Epidermal Growth Factor - pharmacology | Mutation | Twist-Related Protein 1 - metabolism | Adenocarcinoma | Epidermal growth factor | RNA | Lung cancer | Mortality | Stem cells | Development and progression | Genetic aspects | Health aspects | Thoracic surgery | Senescence | Motility | Mesenchyme | Copy number | Activation | Biology | Metastasis | Kinases | E-cadherin | Metastases | Embryogenesis | Cell growth | Cooperation | Surgery | Epidermal growth factor receptors | Chromosome 7 | Tumor cell lines | Gene expression | Ribonucleic acid--RNA | Patients | Embryonic growth stage | Pathology | Cell lines | Hypoxia | Head & neck cancer | Smoking | Cancer | Tumors | Index Medicus | Lung | Ribonucleic acid
Journal Article
Journal Article
PLoS Genetics, ISSN 1553-7390, 05/2012, Volume 8, Issue 5, pp. e1002723 - e1002723
The epithelial-mesenchymal transition (EMT) is an embryonic transdifferentiation process consisting of conversion of polarized epithelial cells to motile... 
MIDDLE-T | TO-MESENCHYMAL TRANSITION | GENE | GROWTH | GENETICS & HEREDITY | PROTEIN PHOSPHATASE 2A | IDENTIFICATION | EXPRESSION | CANCER STEM-CELLS | TWIST | SNAIL | Epithelial Cells - metabolism | Homeodomain Proteins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Epithelial-Mesenchymal Transition - genetics | Mammary Glands, Human - metabolism | Breast Neoplasms - metabolism | Claudins - metabolism | Cell Transformation, Neoplastic - genetics | Female | Telomerase - metabolism | Cell Differentiation | Epithelial Cells - cytology | Cell Line | Claudins - genetics | Mammary Glands, Human - cytology | Protein Phosphatase 2 - antagonists & inhibitors | Retinoblastoma Protein - metabolism | Tumor Suppressor Protein p53 - metabolism | Mice, Transgenic | Transcription Factors - genetics | Cell Transformation, Neoplastic - metabolism | Homeodomain Proteins - genetics | Mammary Glands, Animal - cytology | Transcription Factors - metabolism | Animals | Breast Neoplasms - genetics | Mammary Glands, Animal - metabolism | Protein Phosphatase 2 - metabolism | Twist-Related Protein 1 - genetics | Mice | Genes, ras | Twist-Related Protein 1 - metabolism | Zinc Finger E-box-Binding Homeobox 1 | Transcription factors | Physiological aspects | Development and progression | Breast cancer | Genetic aspects | Research | Cell differentiation | Risk factors | Proteins | Rodents | Colonies & territories | Gene expression | Experiments | Tumors | Apoptosis | Index Medicus | Life Sciences | Cancer
Journal Article