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Journal of the American Society of Nephrology, ISSN 1046-6673, 08/2012, Volume 23, Issue 8, pp. 1283 - 1290
Journal Article
Journal Article
Neuron, ISSN 0896-6273, 07/2017, Volume 95, Issue 1, pp. 33 - 50
Several neurodegenerative disorders, such as Alzheimer’s and Parkinson’s disease, are characterized by prominent loss of synapses and neurons associated with... 
protein-aggregation | prion-like propagation | protein diffusion | pathogenic proteins | Alzheimer | Parkinson | membrane interaction | ALPHA-SYNUCLEIN OLIGOMERS | ALZHEIMERS-DISEASE BRAIN | SELECTIVE NEURONAL VULNERABILITY | IN-VIVO | NICOTINIC ACETYLCHOLINE-RECEPTOR | A-BETA | ION-CHANNEL FORMATION | HEPARAN-SULFATE PROTEOGLYCANS | NEUROSCIENCES | CELLULAR PRION PROTEIN | AMYLOID-BETA OLIGOMERS | Prions - metabolism | Humans | Huntingtin Protein - metabolism | tau Proteins - metabolism | Neurodegenerative Diseases - metabolism | Huntington Disease - metabolism | Protein Folding | Protein Transport | Extracellular Space - metabolism | Animals | Superoxide Dismutase-1 - metabolism | Alzheimer Disease - metabolism | Amyloid beta-Peptides - metabolism | Amyotrophic Lateral Sclerosis - metabolism | Cell Membrane - metabolism | Parkinson Disease - metabolism | alpha-Synuclein - metabolism | Protein Aggregation, Pathological - metabolism | Nervous system diseases | Neurosciences | Cell membranes | Membrane proteins | Therapeutics | Alzheimer's disease | Homeopathy | Materia medica and therapeutics | Plasma | Peptides | Neurodegenerative diseases | Propagation | Neurons | Creutzfeldt-Jakob disease | Disorders | Amyotrophic lateral sclerosis | Agglomeration | Drug development | Clustering | Cell surface | Phase transitions | Crowding | Proteins | Pathology | Protein folding | Neurodegeneration | Assemblies | Diffusion | Synapses | Plasmas (physics) | Life Sciences | Cognitive Sciences | Neurons and Cognition | Psychology and behavior | Neurobiology
Journal Article
Journal of Pediatrics, The, ISSN 0022-3476, 2012, Volume 160, Issue 6, pp. 896 - 897
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 05/2019, Volume 316, Issue 5, pp. F1078 - F1089
It is generally believed that cells that are unable to downregulate glucose transport are particularly vulnerable to hyperglycemia. Yet, little is known about... 
Podocytes | Hyperglycemia | Sodium-dependent glucose transporter | Apoptosis | Proximal tubular cells | KIDNEY-DISEASE | PHYSIOLOGY | sodium-dependent glucose transporter | TRANSPORTER | DAPAGLIFLOZIN | MITOCHONDRIA | apoptosis | DIABETIC-NEPHROPATHY | MECHANISMS | hyperglycemia | podocytes | proximal tubular cells | UROLOGY & NEPHROLOGY | GLOMERULI | INHIBITORS | OUTCOMES | PODOCYTE
Journal Article
BMC Neuroscience, ISSN 1471-2202, 01/2011, Volume 12, Issue 1, pp. 16 - 16
Journal Article
Science, ISSN 0036-8075, 12/2009, Volume 326, Issue 5959, pp. 1554 - 1557
Journal Article
American Journal of Physiology: Cell Physiology, ISSN 0363-6143, 04/2016, Volume 310, Issue 7, pp. C491 - C491
The Na+-K+-ATPase (NKA) differs from most other ion transporters, not only in its capacity to maintain a steep electrochemical gradient across the plasma... 
Journal Article
American Journal of Physiology, ISSN 0363-6143, 04/2016, Volume 310, Issue 7, p. C491
  The Na+-K+-ATPase (NKA) differs from most other ion transporters, not only in its capacity to maintain a steep electrochemical gradient across the plasma... 
Plasma | Molecules | Signal transduction | Membranes | Mitochondria
Journal Article