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Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2012, Volume 122, Issue 6, pp. 2114 - 2129
Journal Article
Nature Communications, ISSN 2041-1723, 09/2016, Volume 7, Issue 1, p. 12756
Thymic selection shapes an appropriate T cell antigen receptor (TCR) repertoire during T cell development. Here, we show that a serine/threonine kinase,... 
SIGNAL | PHOSPHOPROTEOMICS | THRESHOLD | ANTIGEN RECEPTORS | MULTIDISCIPLINARY SCIENCES | IN-VIVO | TYROSINE PHOSPHATASES | DEFICIENT MICE | DIFFERENTIATION | T-CELL DEVELOPMENT | EXPRESSION
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 11/2016, Volume 480, Issue 1, p. 23
Cellular FLICE-inhibitory protein (cFLIP) is a catalytically inactive homolog of the initiator caspase, caspase 8 and blocks apoptosis through binding to... 
Medical colleges | Tumor necrosis factor | Analysis | Genetic engineering | Protein kinases
Journal Article
Cancer Research, ISSN 0008-5472, 05/2017, Volume 77, Issue 9, pp. 2279 - 2291
Journal Article
Cancer Research, ISSN 0008-5472, 08/2015, Volume 75, Issue 15 Supplement, pp. 4195 - 4195
Journal Article
Cell Reports, ISSN 2211-1247, 01/2018, Volume 22, Issue 2, pp. 482 - 496
A subset of mitochondrial tRNAs (mt-tRNAs) contains taurine-derived modifications at 34U of the anticodon. Loss of taurine modification has been linked to the... 
taurine | tRNA | Opa1 | modification | mitochondria | POINT MUTATIONS | WOBBLE MODIFICATION DEFICIENCY | MUTANT TRANSFER-RNAS | CHEMICAL CHAPERONES | STRESS-RESPONSE | LACTIC-ACIDOSIS | SIDEROBLASTIC ANEMIA | TRANSLATION | BILE-ACIDS | COMPLEX-I DEFICIENCY | CELL BIOLOGY
Journal Article
Oncogene, ISSN 0950-9232, 02/2019, Volume 38, Issue 8, pp. 1211 - 1224
The accumulation of prostaglandin E2 (PGE(2)) during chronic inflammation has been implicated in the progression of several cancers. Cyclooxygenase is the key... 
TARGET | SUPPRESSOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOWN-REGULATION | TRANS-RETINOIC ACID | CANCER STEM-CELLS | CELL BIOLOGY | ENGINEERED MOUSE MODELS | ONCOLOGY | ROLES | INFLAMMATION | GENETICS & HEREDITY | 15-HYDROXYPROSTAGLANDIN DEHYDROGENASE | Inflammation - pathology | Adenocarcinoma - pathology | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Retinoic Acid 4-Hydroxylase - genetics | Carcinoma, Pancreatic Ductal - genetics | Neoplastic Stem Cells - metabolism | Neoplastic Stem Cells - pathology | Adenocarcinoma - genetics | Gene Expression Regulation, Neoplastic - drug effects | Disease Models, Animal | Cell Proliferation - genetics | Tretinoin - administration & dosage | Dinoprostone - genetics | Isoenzymes - genetics | Pancreas - drug effects | Pancreas - metabolism | Retinal Dehydrogenase - genetics | Adenocarcinoma - drug therapy | Carcinoma, Pancreatic Ductal - pathology | Hydroxyprostaglandin Dehydrogenases - genetics | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Cell Line, Tumor | Inflammation - genetics | Mice | Prostaglandins | Care and treatment | Pancreatic tumors | Development and progression | Cellular signal transduction | Genetic aspects | Oxidoreductases | Gene expression | Health aspects | Adenocarcinoma | Enzymes | 15-Hydroxyprostaglandin dehydrogenase (NAD+) | Molecular modelling | Clonal deletion | Tumor cells | Stem cells | Prostaglandin E2 | Pancreas | Retinoic acid | Prostaglandin endoperoxide synthase
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2014, Volume 111, Issue 10, pp. 3805 - 3810
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 07/2019, Volume 139, Issue 7, pp. 1459 - 1469
Identification of the specific genetic variants responsible for the increased susceptibility to familial or sporadic cancers is important. Using a forward... 
Journal Article