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Journal Article
Journal Article
Angiogenesis, ISSN 0969-6970, 5/2018, Volume 21, Issue 2, pp. 215 - 228
IL-11 has been detected in inflamed joints; however, its role in the pathogenesis of arthritis is not yet clear. Studies were conducted to characterize the... 
Biomedicine, general | RA synovial tissue | Biomedicine | Endothelial migration and tube formation | RA synovial fluid | RA synovial tissue fibroblasts | Cancer Research | Oncology | Ophthalmology | Cardiology | IL-11 | Cell Biology | RHEUMATOID-ARTHRITIS | HUMAN COLORECTAL ADENOCARCINOMA | FAMILY CYTOKINE | INTERLEUKIN-11 | CHEMOTACTIC CYTOKINE | RECEPTOR-ALPHA | DISEASE-ACTIVITY | SYNOVIOCYTES | PERIPHERAL VASCULAR DISEASE | GROWTH-FACTOR | EXPRESSION | Endothelial Cells - metabolism | Humans | Interleukin-11 - metabolism | Joints - pathology | Male | Vascular Endothelial Growth Factor A - metabolism | Fibroblasts - pathology | Interleukin-11 Receptor alpha Subunit - metabolism | Arthritis, Rheumatoid - pathology | Neovascularization, Pathologic - pathology | Arthritis, Rheumatoid - metabolism | Transendothelial and Transepithelial Migration | Female | Neovascularization, Pathologic - metabolism | Interleukin-8 - metabolism | Endothelial Cells - pathology | Joints - metabolism | Fibroblasts - metabolism | Fibroblasts | Neovascularization | Research | Osteoarthritis | Vascular endothelial growth factor | Analysis | Cell proliferation | Pathogenesis | Interleukin 11 | Inflammation | Arthritis | Macrophages | Tissues | Joint diseases | Endothelial cells | Vascularization | Synovial fluid | Angiogenesis | Biomedical materials | Rheumatoid arthritis | Lining (process) | Biocompatibility | Infiltration | Cell migration | Interleukin 8 | endothelial migration and tube formation
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 10/2015, Volume 74, Issue 10, pp. 1898 - 1906
ObjectiveThis study was conducted to determine the expression pattern, regulation and function of CCL28 and CCR10 in rheumatoid arthritis (RA)... 
RHEUMATOID-ARTHRITIS | B-CELLS | ANGIOGENESIS | CHEMOKINE CCL28 | LIGATION | TNF-ALPHA | ACCUMULATION | RHEUMATOLOGY | MYELOID CELLS | IDENTIFICATION | CUTTING EDGE | Joints - blood supply | Receptors, CCR10 - genetics | MAP Kinase Signaling System - physiology | Humans | Middle Aged | Synovial Membrane - immunology | Male | Monocytes - immunology | MAP Kinase Signaling System - immunology | Signal Transduction - immunology | Chemokines, CC - biosynthesis | Adult | Female | Chemokines, CC - immunology | Neovascularization, Pathologic - immunology | Chemotaxis - immunology | Macrophages - immunology | Endothelium, Vascular - immunology | Gene Expression Regulation - immunology | Osteoarthritis - immunology | RNA, Messenger - genetics | Cells, Cultured | Gene Silencing | Receptors, CCR10 - deficiency | Receptors, CCR10 - biosynthesis | Osteoarthritis - genetics | Receptors, CCR10 - immunology | Endothelial Cells - immunology | Arthritis, Rheumatoid - genetics | Chemokines, CC - genetics | Aged | Arthritis, Rheumatoid - immunology | Usage | Histology | Research | Joints | Chemotaxis | Biotechnology | Plasma | Immunoglobulins | Cytokines | Pathogenesis | Laboratories | Arthritis | Kinases | Gene expression | Ovarian cancer | Studies | Angiogenesis | Hypoxia | TNF inhibitors | Ligands | Tumor necrosis factor-TNF | Vascular endothelial growth factor | macrophages | endothelial cells | CCL28 | angiogenesis | CCR10
Journal Article
Drug Design, Development and Therapy, ISSN 1177-8881, 03/2014, Volume 8, pp. 349 - 364
Rheumatoid arthritis (RA) is a systemic inflammatory disease characterized by joint pain, swelling, stiffness, and progressive destruction of the small joints... 
Tocilizumab | Biologics | Rheumatoid arthritis | IL-6 | MODIFYING ANTIRHEUMATIC DRUGS | SYSTEMIC-LUPUS-ERYTHEMATOSUS | CHEMISTRY, MEDICINAL | ADALIMUMAB MONOTHERAPY | biologics | OPEN-LABEL | MONOCLONAL-ANTIBODY | RANDOMIZED CONTROLLED-TRIAL | rheumatoid arthritis | DISEASE-ACTIVITY | tocilizumab | DOUBLE-BLIND | PHARMACOLOGY & PHARMACY | INTERLEUKIN-6 RECEPTOR INHIBITION | REFRACTORY TAKAYASU ARTERITIS | Antibodies, Monoclonal, Humanized - therapeutic use | Antibodies, Monoclonal, Humanized - pharmacokinetics | Arthritis, Rheumatoid - drug therapy | Arthritis, Rheumatoid - etiology | Humans | Interleukin-6 - physiology | Receptors, Interleukin-6 - antagonists & inhibitors | Drug Therapy, Combination | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Drug therapy | Transplants & implants | Disease | Pathogenesis | Interleukin | Colorectal cancer | Clinical trials | Transplantation | Arthritis | Cancer therapies | Inflammatory diseases | Immunosuppressive agents | Interleukin 6 | Angiogenesis | Signal transduction | Pain | Lymphocytes | Interleukin 6 receptors | Fibroblasts | Tumor necrosis factor-TNF | Stiffness | Safety | Methotrexate | Vascular endothelial growth factor | Recombinant | Medical research | Signs and symptoms | Effectiveness | Cytokines | Medical treatment | Neutrophils | Rheumatology | Inflammation | Pharmacology | FDA approval | Tumor necrosis factor-α | Joint diseases | Patients | Mode of action | Perforations | Monoclonal antibodies | Cardiovascular diseases | Autoimmune diseases | Pharmacokinetics
Journal Article
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 01/2019, Volume 78, Issue 1, pp. 122 - 130
Journal Article
Journal of Immunology, ISSN 0022-1767, 10/2014, Volume 193, Issue 8, pp. 3902 - 3913
Journal Article
CELLULAR AND MOLECULAR LIFE SCIENCES, ISSN 1420-682X, 04/2020, Volume 77, Issue 7, pp. 1387 - 1399
In rheumatoid arthritis (RA), synovial tissue abundantly expresses CCL21, a chemokine strongly associated with RA susceptibility. In this study, we aimed to... 
CCL21 | BIOCHEMISTRY & MOLECULAR BIOLOGY | SUSCEPTIBILITY | DRIVE | CELL BIOLOGY | Osteoclastogenesis | LIGANDS | M1 macrophages | Rheumatoid arthritis | DISEASE | CCR7 | CHEMOKINE | CCL19 | DIFFERENTIATION | TLR5 | EXPRESSION | Th17 cells | Cell Polarity | Inflammation - pathology | Up-Regulation | Arthritis, Rheumatoid - blood | Humans | Middle Aged | Synovial Fluid - metabolism | Male | Chemokine CCL21 - metabolism | Interleukins - metabolism | Receptors, CCR7 - metabolism | Monocytes - pathology | Female | Cell Differentiation | Biomarkers - metabolism | Osteoclasts - pathology | Signal Transduction | Mice, Inbred C57BL | Joints - pathology | Receptors, CCR7 - blood | Chemotaxis | Arthritis, Rheumatoid - pathology | Macrophages - metabolism | Animals | Myeloid Cells - metabolism | Th17 Cells - immunology | Mice | Arthritis, Rheumatoid - immunology | Osteogenesis | CC chemokine receptors | Polarization | Transcription | CD86 antigen | Leukocyte migration | Pathogenesis | Crosstalk | Helper cells | Interleukin 23 | Lymphocytes T | Arthritis | Macrophages | CD14 antigen | Vascularization | Lipopolysaccharides | Interleukin 6 | Synovial fluid | Interleukin 4 | Biomedical materials | Lymphocytes | Joints (anatomy) | CCL21 protein | Biocompatibility | Cytokines | CCR7 protein | Cell differentiation | Interleukin 17 | Signaling | Monocytes | γ-Interferon | Disruption
Journal Article
Journal of immunology (Baltimore, Md. : 1950), ISSN 0022-1767, 10/2014, Volume 193, Issue 8, pp. 3902 - 3913
The aim was to examine the impact of TLR5 ligation in rheumatoid arthritis (RA) and experimental arthritis pathology. Studies were conducted to investigate the... 
osteoclast differentiation | TNF-α | TLR5 | collagen induced arthritis | monocyte migration
Journal Article
Journal of immunology (Baltimore, Md. : 1950), ISSN 0022-1767, 5/2013, Volume 190, Issue 10, pp. 5256 - 5266
Although role of IL-7 and IL-7R has been implicated in the pathogenesis of Rheumatoid arthritis (RA), the majority of the studies have focused on the impact of... 
collagen induced arthritis | IL-7R and TNF-α | monocyte migration | disease correlation | IL-7
Journal Article