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Blood, ISSN 0006-4971, 04/2017, Volume 129, Issue 15, pp. 2048 - 2049
Journal Article
Thrombosis Research, ISSN 0049-3848, 04/2012, Volume 129, Issue 1, pp. S80 - S84
Cancer is frequently associated with activation of blood coagulation, which in turn has been suggested to promote tumor growth and metastasis. Indeed, low... 
Protein C | Metastasis | APC | Coagulation | Cancer | Glycosaminoglycans | Analysis | Development and progression | Anticoagulants (Medicine)
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 12/2017, Volume 377, Issue 23, pp. 2215 - 2227
Journal Article
Revista Brasileira de Hematologia e Hemoterapia, ISSN 1516-8484, 2013, Volume 35, Issue 4, pp. 233 - 234
Journal Article
Nature Medicine, ISSN 1078-8956, 03/2006, Volume 12, Issue 3, pp. 342 - 347
Journal Article
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 05/2008, Volume 358, Issue 21, pp. 2240 - 2248
Journal Article
Revista Brasileira de Hematologia e Hemoterapia, ISSN 1806-0870, 2013, Volume 35, Issue 4, pp. 233 - 234
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 10/2009, Volume 361, Issue 17, pp. 1671 - 1675
A 23-year-old man with deep-vein thrombosis was found to have a gain-of-function mutation in his factor IX gene. The plasma level of factor IX was normal, but... 
MEDICINE, GENERAL & INTERNAL | RECURRENT VENOUS THROMBOEMBOLISM | GENE | VARIANTS | DETERMINANTS | INCREASE | MUTATION | FACTOR-V | RISK | CARRIERS | Thrombophilia - genetics | Humans | Factor IX - genetics | Genotype | Male | Mutation, Missense | Young Adult | Point Mutation | DNA Mutational Analysis | Pedigree | Factor IX - metabolism | Female | Genetic Diseases, X-Linked - genetics | Index Medicus | Abridged Index Medicus
Journal Article
Molecular Therapy, ISSN 1525-0016, 08/2017, Volume 25, Issue 8, pp. 1815 - 1830
Hemophilia A (HA) is an X-linked bleeding disease caused by factor VIII (FVIII) deficiency. We previously demonstrated that FVIII is produced specifically in... 
hemophilia A | gene therapy | targeted FVIII expression | inhibitor titers reversion | Tregs | MEDICINE, RESEARCH & EXPERIMENTAL | EFFICACY | HUMAN-FACTOR VIII | VON-WILLEBRAND-FACTOR | LENTIVIRAL VECTORS | TRANSPLANTATION | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | ENDOTHELIAL-CELLS | GENETICS & HEREDITY | GENE-THERAPY | COAGULATION-FACTOR VIII | EXPRESSION | T-CELLS | Factor VIII - immunology | T-Lymphocytes, Regulatory - metabolism | Immunosuppression - methods | Male | Mice, 129 Strain | T-Lymphocytes, Regulatory - immunology | Isoantibodies - blood | Organ Specificity - genetics | CD11b Antigen - genetics | Lentivirus - genetics | Transgenes | Genes, Reporter | Disease Models, Animal | Promoter Regions, Genetic | Gene Expression | Immunization | Transduction, Genetic | Endothelial Cells - metabolism | Hemophilia A - immunology | Mice, Inbred C57BL | Factor VIII - genetics | Genetic Vectors - genetics | Animals | Immune Tolerance - genetics | Isoantibodies - immunology | Hemophilia A - genetics | Mice | Mice, Inbred BALB C | Factor VIII - metabolism | Whole Blood Coagulation Time | Myeloid cells | Immunoglobulins | Liver | Cloning | MiRNA | Lymphocytes T | Experiments | Immunological tolerance | Endothelial cells | Hemopoiesis | Coagulation factors | Hepatocytes | MicroRNAs | Hemophilia | Conflicts of interest | Software | Gene therapy | Index Medicus | Original
Journal Article
PLoS ONE, 03/2016, Volume 11, Issue 3
Severe hemophilia A (HA) is an inherited bleeding disorder characterized by <1% of residual factor VIII (FVIII) clotting activity. The disease affects several... 
Journal Article
Journal Article
Journal Article