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The Journal of Dermatology, ISSN 0385-2407, 02/2018, Volume 45, Issue 2, pp. 128 - 138
Systemic sclerosis (SSc) is a multisystem autoimmune disease characterized by vasculopathy and tissue fibrosis of the skin and various internal organs. A... 
epithelial cells | vasculogenesis | fibrosis | autoimmunity | angiogenesis | SCLERODERMA FIBROBLASTS | INTERCELLULAR-ADHESION MOLECULE-1 | INCREASED EXPRESSION | DERMATOLOGY | GROWTH-FACTOR-BETA | TUMOR-NECROSIS-FACTOR | PROTEIN-C RECEPTOR | REGULATORY T-CELLS | DERMAL FIBROBLASTS | ENDOTHELIAL-CELL | FLI1 DEFICIENCY CONTRIBUTES | Genetic Predisposition to Disease | Skin - cytology | Vascular Remodeling - immunology | Humans | Autoimmune Diseases - immunology | Scleroderma, Systemic - genetics | Scleroderma, Systemic - pathology | Autoimmunity - genetics | Scleroderma, Systemic - immunology | Autoimmune Diseases - genetics | CD4-Positive T-Lymphocytes - immunology | Endothelial Cells - immunology | Skin - blood supply | Autoantibodies - immunology | Autoimmunity - immunology | Fibrosis | Fibroblasts - immunology | Autoimmune Diseases - pathology | Autoimmune Diseases - drug therapy | Macrophages - immunology | Scleroderma, Systemic - drug therapy | Skin - pathology | Immunologic Factors - therapeutic use | Skin - immunology | Autoimmunity | Systemic scleroderma | Scleroderma (Disease) | Physiological aspects | Disease susceptibility | T cells | Epidemiology | Animal models | Immune response | Autoantibodies | Disease | Cytokines | Secretion | Lymphocytes T | Inflammation | Endothelial cells | CD4 antigen | Vascular diseases | Systemic sclerosis | Fibroblasts | Extracellular matrix | Genetic factors | Autoimmune diseases | Growth factors
Journal Article
International Journal of Biochemistry and Cell Biology, ISSN 1357-2725, 10/2015, Volume 67, pp. 86 - 91
Systemic sclerosis (SSc) is a multisystem connective tissue disease featured by immune abnormalities, vasculopathy and tissue fibrosis with unknown etiology. A... 
Fibroblasts | Macrophages | Systemic sclerosis | Endothelial cells | Fli1 | DNA-BINDING ABILITY | SCLERODERMA FIBROBLASTS | COLLAGEN EXPRESSION | LEUKEMIA-VIRUS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOWN-REGULATION | TRANSCRIPTION FACTOR FLI1 | CELL BIOLOGY | GROWTH-FACTOR-BETA | DERMAL FIBROBLASTS | PROTEIN-ASSOCIATED FACTOR | T-CELLS | Bleomycin - antagonists & inhibitors | Epigenesis, Genetic | Humans | Scleroderma, Systemic - pathology | Th2 Cells - immunology | Th2 Cells - drug effects | Th17 Cells - drug effects | Proto-Oncogene Protein c-fli-1 - agonists | Macrophages - immunology | Th2 Cells - pathology | Disease Models, Animal | Th17 Cells - pathology | Genetic Predisposition to Disease | Macrophages - pathology | Proto-Oncogene Protein c-fli-1 - genetics | Scleroderma, Systemic - genetics | Mice, Transgenic | Scleroderma, Systemic - immunology | Fibroblasts - pathology | Sulfonamides - pharmacology | Endothelial Cells - immunology | Proto-Oncogene Protein c-fli-1 - antagonists & inhibitors | Animals | Fibroblasts - drug effects | Th17 Cells - immunology | Bleomycin - pharmacology | Fibroblasts - immunology | Macrophages - drug effects | Mice | Endothelial Cells - pathology | Proto-Oncogene Protein c-fli-1 - immunology | Scleroderma, Systemic - drug therapy | Endothelial Cells - drug effects | Autoimmunity | Epigenetic inheritance | Medical colleges | Endothelin | Systemic scleroderma | Leukemia | Scleroderma (Disease) | Development and progression | Health aspects | Risk factors
Journal Article
The Journal of Dermatology, ISSN 0385-2407, 01/2010, Volume 37, Issue 1, pp. 54 - 70
Systemic sclerosis (SSc) is an autoimmune disorder with clinical manifestations resulting from immune activation, fibrosis development and damage of small... 
immune dysfunction | fibrosis | vasculopathy | new treatments | scleroderma | Vasculopathy | Scleroderma | Immune dysfunction | Fibrosis | New treatments | PULMONARY ARTERIAL-HYPERTENSION | ACTIVATES LATENT TGF-BETA-1 | ANTIENDOTHELIAL CELL ANTIBODIES | BETA TYPE-I | TRANSCRIPTION FACTOR FLI1 | DERMATOLOGY | INTERSTITIAL LUNG-DISEASE | SCLERODERMA RENAL CRISIS | TRANSFORMING-GROWTH-FACTOR | PROTEIN-ASSOCIATED FACTOR | CONNECTIVE-TISSUE DISEASES | Endothelin-1 - antagonists & inhibitors | Autoimmune Diseases - physiopathology | Platelet-Derived Growth Factor - immunology | Receptors, Transforming Growth Factor beta - immunology | Connective Tissue Growth Factor - immunology | Humans | Phosphodiesterase 5 Inhibitors | Smad3 Protein - immunology | Scleroderma, Systemic - physiopathology | Transforming Growth Factor beta - antagonists & inhibitors | Autoimmune Diseases - drug therapy | Endothelin Receptor Antagonists | Transforming Growth Factor beta - immunology | Connective Tissue Growth Factor - antagonists & inhibitors | Autoimmune Diseases - immunology | Serotonin Uptake Inhibitors - therapeutic use | Piperazines - therapeutic use | Scleroderma, Systemic - immunology | Imatinib Mesylate | Smad3 Protein - antagonists & inhibitors | Randomized Controlled Trials as Topic | Endothelin-1 - immunology | Animals | B-Lymphocytes - immunology | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Pyrimidines - therapeutic use | Mice | Benzamides | Immunoglobulins - therapeutic use | Platelet-Derived Growth Factor - antagonists & inhibitors | Scleroderma, Systemic - drug therapy | Hypertension | Immunoglobulins | Platelet-derived growth factor | Peptides | Stem cells | Transplantation | Transforming growth factors
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2013, Volume 8, Issue 9, p. e74930
Fli1, a member of the Ets transcription factor family, is a key repressor of the human α2(I) collagen (COL1A2) gene. Although our previous studies have... 
Dermis - metabolism | Proto-Oncogene Protein c-fli-1 - genetics | Humans | Cells, Cultured | E1A-Associated p300 Protein - genetics | Male | Multiprotein Complexes - genetics | E1A-Associated p300 Protein - metabolism | Protein Processing, Post-Translational - physiology | Epigenesis, Genetic - physiology | Multiprotein Complexes - metabolism | Collagen Type I - biosynthesis | Transcription, Genetic - physiology | Collagen Type I - genetics | Transforming Growth Factor beta - genetics | Proto-Oncogene Protein c-fli-1 - metabolism | Female | Fibroblasts - cytology | Dermis - cytology | Response Elements - physiology | Histone Deacetylase 1 - genetics | Transforming Growth Factor beta - metabolism | Fibroblasts - metabolism | Histone Deacetylase 1 - metabolism | ETS protein | Chromatin | Collagen (type I) | Phosphorylation | Transcription factors | Ets-1 protein | Peptides | Laboratories | Pathogenesis | Protein purification | Gene regulation | Event-related potentials | Arthritis | Kinases | Remodeling | Recruitment | Proteins | Rodents | Transcription activation | Deoxyribonucleic acid | Post-translation | Fibroblasts | Extracellular matrix | Acetylation | Growth factors | Medical research | Immunoglobulins | Threonine | Mass spectroscopy | Pharmacology | Gene expression | Chromatin remodeling | Studies | Plasmids | Deacetylation | Collagen | DNA | Skin | Protein interaction | Mass spectrometry | Dismantling | Histone H3 | Binding sites
Journal Article
The Journal of Dermatology, ISSN 0385-2407, 08/2017, Volume 44, Issue 8, pp. 967 - 971
Journal Article
European Journal of Dermatology, ISSN 1167-1122, 7/2019, Volume 29, Issue 4, pp. 439 - 440
Journal Article