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Pest Management Science, ISSN 1526-498X, 08/2018, Volume 74, Issue 8, pp. 1837 - 1844
Journal Article
PLoS ONE, ISSN 1932-6203, 2012, Volume 7, Issue 11, pp. e49940 - e49940
Introduction: Lymphedema is a chronic disorder that occurs commonly after lymph node removal for cancer treatment and is characterized by swelling, fibrosis,... 
BREAST-CANCER | IMMUNE PROTEINS | PERIPHERAL LYMPH | REGENERATION | MULTIDISCIPLINARY SCIENCES | INSUFFICIENCY | SKIN | FILARIAL LYMPHEDEMA | EXPRESSION | WOUND REPAIR | Adaptive Immunity | CD8-Positive T-Lymphocytes - cytology | Inflammation - pathology | Interleukin-2 Receptor alpha Subunit | Lymph Nodes - pathology | Lymphedema - immunology | Lymphedema - pathology | Lymph Nodes - surgery | Humans | CD4-Positive T-Lymphocytes - metabolism | Tail - immunology | Inflammation - immunology | Tail - pathology | CD4-Positive T-Lymphocytes - immunology | Animals | Flow Cytometry | Lymphangiogenesis - immunology | T-Lymphocytes, Helper-Inducer - immunology | Fibrosis - immunology | T-Lymphocytes, Helper-Inducer - cytology | Mice | Fibrosis - pathology | CD8-Positive T-Lymphocytes - immunology | Single-Cell Analysis | Viral antibodies | Medical research | Care and treatment | Dendritic cells | Analysis | Fibrosis | Physiological aspects | Medicine, Experimental | Antibodies | Inflammation | Cancer | Flow cytometry | Animal models | CD8 antigen | Helper cells | Inflammatory response | Infections | Arthritis | Macrophages | Cancer therapies | Tails | Lymph nodes | Lymphedema | Rodents | CD25 antigen | Dissection | Deposition | Lymphatic system | Wound healing | Immune response | Plastic surgery | Leukocytes (neutrophilic) | Radiation therapy | CD4 antigen | Studies | Cell suspensions | Pathology | Cytometry | Depletion | Biopsy | Infiltration | Lymph | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 11/2008, Volume 295, Issue 5, pp. 2113 - 2127
Although clinical studies have identified scarring/fibrosis as significant risk factors for lymphedema, the mechanisms by which lymphatic repair is impaired... 
Lymphedema | Myofibroblast | Scar | Transforming growth factor-β | Vascular endothelial growth factor C | Excisional wound healing | Fibrosis | Lymphangiogenesis
Journal Article
Aesthetic Plastic Surgery, ISSN 0364-216X, 4/2018, Volume 42, Issue 2, pp. 410 - 411
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1007/s00266-017-1035-4 
Medicine & Public Health | Plastic Surgery | Otorhinolaryngology | SURGERY | Plastic surgery | Index Medicus
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 09/2010, Volume 299, Issue 3, pp. 589 - 605
Although radiation therapy is a major risk factor for the development of lymphedema following lymphadenectomy, the mechanisms responsible for this effect... 
Lymphedema | Transforming growth factor-β1 | Endothelium | endothelium | transforming growth factor-β1 | Vascular Biology | lymphedema
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 01/2012, Volume 302, Issue 2, pp. C392 - C404
Zampell JC, Avraham T, Yoder N, Fort N, Yan A, Weitman ES, Mehrara BJ. Lymphatic function is regulated by a coordinated expression of lymphangiogenic and... 
Vascular endothelial growth factor-C | Lymphedema | Transforming growth factor-β1 | Lymphangiogenesis | Interferon-γ | BREAST-CARCINOMA | PHYSIOLOGY | lymphedema | INHIBITS LYMPHANGIOGENESIS | vascular endothelial growth factor-C | VEGF-C | INTERSTITIAL FLOW | MOUSE TAIL | transforming growth factor-beta 1 | CELL BIOLOGY | THERAPEUTIC LYMPHANGIOGENESIS | NODE METASTASIS | lymphangiogenesis | GENE-THERAPY | interferon-gamma | AMELIORATES SECONDARY LYMPHEDEMA | WOUND REPAIR | Inflammation - pathology | Lymphatic System - pathology | Tail - physiopathology | Vascular Endothelial Growth Factor A - metabolism | Interferon-gamma - metabolism | Tail - pathology | Inflammation - metabolism | Lymph - metabolism | Interferon-gamma - antagonists & inhibitors | Female | Lymphatic System - physiopathology | Lymphatic System - physiology | Tail - anatomy & histology | Lymphedema - pathology | Cytokines - metabolism | Mice, Inbred C57BL | Lymphangiogenesis - physiology | Regeneration - physiology | Hepatocyte Growth Factor - metabolism | Animals | Mice, Nude | Vascular Endothelial Growth Factor C - metabolism | T-Lymphocytes - immunology | Mice | Tail - metabolism | Lymphedema - metabolism | Physiological aspects | Research | Cytokines | Gene expression | Health aspects | Cell growth | Interferon | Rodents | Index Medicus | transforming growth factor-β1 | interferon-γ | Vascular Biology
Journal Article
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2010, Volume 177, Issue 6, pp. 3202 - 3214
Lymphedema is a complication of cancer treatment occurring in approximately 50% of patients who undergo lymph node resection. Despite its prevalence, the... 
Pathology | Regular
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 2, pp. e17201 - e17201
Journal Article
JAMA Surgery, ISSN 2168-6254, 04/2018, Volume 153, Issue 4, pp. 351 - 352
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 02/2012, Volume 302, Issue 4, pp. C709 - C719
Zampell JC, Elhadad S, Avraham T, Weitman E, Aschen S, Yan A, Mehrara BJ. Toll-like receptor deficiency worsens inflammation and lymphedema after lymphatic... 
Endogenous danger signals | ISCHEMIA/REPERFUSION INJURY | ACTIVATION | PHYSIOLOGY | endogenous danger signals | MACROPHAGES | HMGB1 | GROUP BOX-1 PROTEIN | CELL BIOLOGY | SYSTEMIC INFLAMMATION | BREAST-CANCER | DANGER SIGNALS | EXPRESSION | WOUND REPAIR | Inflammation - pathology | Lymphedema - immunology | Toll-Like Receptor 2 - genetics | Transforming Growth Factor beta1 - metabolism | Glycoproteins - metabolism | Toll-Like Receptor 9 - genetics | Fibrosis - metabolism | Leukemic Infiltration - complications | Inflammation - complications | Signal Transduction - immunology | Toll-Like Receptor 2 - deficiency | Transforming Growth Factor beta1 - immunology | Inflammation - metabolism | Lymphatic Vessels - metabolism | Lymphangiogenesis - immunology | Toll-Like Receptor 4 - deficiency | Gene Deletion | Fibrosis - immunology | Gene Expression - immunology | Lymphatic Vessels - injuries | Female | Macrophages - immunology | Leukemic Infiltration - metabolism | Lymphedema - complications | Lymphedema - pathology | Collagen - immunology | Fibrosis - complications | Toll-Like Receptor 9 - deficiency | Lymphatic Vessels - immunology | Toll-Like Receptor 4 - genetics | Inflammation - immunology | Macrophages - cytology | Vascular Endothelial Growth Factor C - immunology | Immunity, Innate | Mice, Knockout | Collagen - metabolism | Glycoproteins - immunology | Macrophages - metabolism | Animals | Vascular Endothelial Growth Factor C - metabolism | Leukemic Infiltration - pathology | Mice | Fibrosis - pathology | Lymphangiogenesis - genetics | Lymphedema - metabolism | Care and treatment | Lymphedema | Patient outcomes | Toll-like receptors | Inflammation | Lymphatics | Research | Health aspects | Signal transduction | Tissue | T cell receptors | Gene expression | Pathogenesis | Rodents | Index Medicus | Vascular Biology
Journal Article
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 05/2011, Volume 300, Issue 5, pp. C1107 - C1121
Journal Article