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Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2012, Volume 109, Issue 38, pp. 15354 - 15359
Journal Article
Cancer Cell, ISSN 1535-6108, 05/2017, Volume 31, Issue 5, pp. 607 - 608
In this issue of , Diamantopoulou et al. uncover dual mechanisms of inhibiting YAP/TAZ by TIAM1 that oppose invasiveness of colorectal cancer cells: TIAM1... 
YAP | ONCOLOGY | BETA-CATENIN | TAZ | TUMORIGENESIS | CELL BIOLOGY | Adaptor Proteins, Signal Transducing | Signal Transduction | Phosphoproteins | Humans | Colorectal Neoplasms | Transcription Factors | Cell Nucleus | Colorectal cancer | Medical colleges | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 08/2019, Volume 116, Issue 36, pp. 17848 - 17857
Autophagy, besides ensuring energy metabolism and organelle renewal, is crucial for the biology of adult normal and cancer stem cells. However, it remains... 
Energy metabolism | Fluctuations | Transcription | Tumor cells | Phenotypic plasticity | Flux | Biology | Metabolism | Autophagy | Yes-associated protein | Organoids | Rewiring | Stem cells | Cytoskeleton | Extracellular matrix | Mechanotransduction | Pancreas | Phagocytosis | Cancer | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 01/2016, Volume 18, Issue 2, pp. 168 - 180
Chronic inflammation is associated with a variety of pathological conditions in epithelial tissues, including cancer, metaplasia and aberrant wound healing. In... 
FIBROSIS | STEM-CELLS | ACTIVATION | YAP/TAZ | SQUAMOUS METAPLASIA | EXTRACELLULAR-MATRIX | T-CELL | DIFFERENTIATION | SUPPRESSION | CANCER | CELL BIOLOGY | Administration, Ophthalmic | Keratitis - pathology | Epithelial Cells - metabolism | Mechanotransduction, Cellular - drug effects | Epithelial Cells - drug effects | Humans | Corneal Injuries - genetics | Extracellular Matrix - metabolism | Epithelium, Corneal - pathology | Phosphoproteins - metabolism | Stem Cells - metabolism | Epithelium, Corneal - injuries | Corneal Injuries - metabolism | Receptor, Notch1 - deficiency | Transfection | Time Factors | Swine | HEK293 Cells | Inflammation Mediators - metabolism | Epithelium, Corneal - drug effects | Anti-Inflammatory Agents - administration & dosage | Wnt Signaling Pathway | Disease Models, Animal | Keratitis - genetics | Corneal Injuries - pathology | Keratitis - prevention & control | Mice, Inbred C57BL | Epithelial Cells - pathology | Phosphoproteins - genetics | Transcription Factors - genetics | beta Catenin - metabolism | beta Catenin - genetics | Mice, Knockout | Transcription Factors - metabolism | Regeneration - drug effects | Animals | Cell Differentiation - drug effects | Adaptor Proteins, Signal Transducing - genetics | Keratitis - metabolism | Epithelium, Corneal - metabolism | Stem Cells - drug effects | Stem Cells - pathology | Adaptor Proteins, Signal Transducing - metabolism | Receptor, Notch1 - genetics | Chronic Disease | Physiological aspects | Cellular signal transduction | Inflammation | Epithelium | Health aspects | Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2018, Volume 24, Issue 10, pp. 1599 - 1610
Journal Article
Nature Cell Biology, ISSN 1465-7392, 01/2018, Volume 20, Issue 1, pp. 28 - 35
Tumour-associated p53 missense mutants act as driver oncogenes affecting cancer progression, metastatic potential and drug resistance (gain-of-function)(1).... 
UBIQUITINATION | CELLS | GLUCOCORTICOID-RECEPTOR | INHIBITION | PATHWAY | ACETYLATION | MUTATION | YAP | HUMAN BREAST-CANCER | EXPRESSION | CELL BIOLOGY | Genetic aspects | Research | Gene mutations | Drug resistance | Cancer cells | Cancer | Cues | Ubiquitin | Hsp90 protein | p53 Protein | RhoA protein | Accumulation | Mutants | Metastases | Mevalonic acid | Actin | Proteolysis | Stiffness | Control stability | Tumors | Index Medicus
Journal Article