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BMC Cancer, ISSN 1471-2407, 02/2018, Volume 18, Issue 1, pp. 157 - 8
Background: Diethylnitrosamine is a well known carcinogen that induces cancers of various organs in mice and rats. Using FVB/N mouse strain, here we show that... 
Mouse model | Tumorigenesis | NSCLC | Diethylnitrosamine | Lung cancer | N-NITROSODIETHYLAMINE | INITIATION | INDUCTION | CELL-CYCLE PROGRESSION | CANCER | TUMORS | EGFR | KRAS MUTATIONS | ONCOLOGY | NEVER SMOKERS | MICE | Adenocarcinoma | Rattus | Rats | Research | Nitrosoamines | Health aspects | Risk factors
Journal Article
Journal of Histochemistry & Cytochemistry, ISSN 0022-1554, 4/2012, Volume 60, Issue 4, pp. 262 - 268
Journal Article
The FEBS Journal, ISSN 1742-464X, 05/2013, Volume 280, Issue 10, pp. 2150 - 2164
Decorin, a secreted small leucine‐rich proteoglycan, acts as a tumor repressor in a variety of cancers, mainly by blocking the action of several receptor... 
platelet‐derived growth factor receptor alpha | liver cancer | carcinogenesis | decorin | proteoglycan | platelet-derived growth factor receptor alpha | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOWN-REGULATION | MONOCLONAL-ANTIBODY | CYCLIN-DEPENDENT KINASES | BREAST-CANCER | LEUCINE-RICH PROTEOGLYCANS | THERAPEUTIC TARGET | IN-VIVO | GENE-EXPRESSION | EXTRACELLULAR-MATRIX | HUMAN HEPATOCELLULAR-CARCINOMA | Liver - pathology | Phosphorylation | Humans | Liver Neoplasms, Experimental - chemically induced | Male | Thioacetamide - adverse effects | Hepatocytes - metabolism | Liver Neoplasms, Experimental - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Liver - drug effects | Tumor Suppressor Proteins - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Female | Liver Neoplasms, Experimental - pathology | Decorin - genetics | Hepatocytes - drug effects | Recombinant Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Protein Interaction Mapping | Platelet-Derived Growth Factor - metabolism | Animals | Receptor, Platelet-Derived Growth Factor alpha - genetics | Fluorescent Antibody Technique | Protein Binding | Ligands | Mice | Decorin - metabolism | Liver cancer | Platelet-derived growth factor | Phosphotransferases | Analysis | Proteins | Signal transduction | Pathogenesis | PDGFRα
Journal Article
Cellular Signalling, ISSN 0898-6568, 10/2019, Volume 62, p. 109354
In spite of therapeutic approaches, liver cancer is still one of the deadliest type of tumor in which tumor microenvironment may play an active role in the... 
Tumor-specific inhibition | Decorin | Liver carcinoma | Proliferation inhibition | Hepatoma cell line | APOPTOSIS | GROWTH-INHIBITION | PROTEOGLYCANS | RECEPTOR | CANCER | CELL BIOLOGY | HEPATOCELLULAR-CARCINOMA | RECOMBINANT HUMAN DECORIN | SUPPRESSES | UP-REGULATION | EXPRESSION
Journal Article
Laboratory Investigation, ISSN 0023-6837, 03/2011, Volume 91, Issue 3, pp. 439 - 451
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2015, Volume 10, Issue 5, p. e0128059
Background Diabetes mellitus is linked to pancreatic cancer. We hypothesized a role for pancreatic stellate cells (PSC) in the hyperglycemia induced... 
DIABETES-MELLITUS | FIBROSIS | ACTIVATION | SP1 | ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | GROWTH | CXCR4 | PROLIFERATION | IDENTIFICATION | GENE-TRANSCRIPTION | Cell Line | Up-Regulation | Oligonucleotide Array Sequence Analysis - methods | Humans | Pancreatic Neoplasms - pathology | Culture Media, Conditioned - pharmacology | Gene Expression Profiling - methods | Pancreatic Neoplasms - genetics | Hyperglycemia - genetics | MAP Kinase Signaling System | Chemokine CXCL12 - genetics | Pancreatic Stellate Cells - cytology | Receptors, CXCR4 - genetics | Cell Differentiation | Cell Proliferation - drug effects | Pancreatic Stellate Cells - pathology | Pancreatic Stellate Cells - metabolism | Hyperglycemia | RNA | Cancer cells | Bone morphogenetic proteins | Glucose | Transforming growth factors | Dextrose | Cell proliferation | Phosphorylation | CXCL12 protein | Insulin-like growth factor-binding protein 2 | Kinases | Cancer therapies | Proteins | Rodents | Fibroblasts | Extracellular matrix | Physiology | Conditioning | Growth factors | Communication | Medical research | Stellate cells | Internal medicine | Diabetes mellitus | Extracellular signal-regulated kinase | MAP kinase | Exposure | Sp1 protein | Gene expression | CXCR4 protein | Medicine | Pathology | Signaling | Pancreatic cancer | Medical prognosis | Cell lines | Stem cells | Ligands | Diabetes | Cell migration | Cancer
Journal Article
FRONTIERS IN GENETICS, ISSN 1664-8021, 06/2019, Volume 10, p. 544
Coincidences of more than one pathogenic mutation in high and/or moderate risk-associated cancer genes have been rarely reported, and the implication for... 
multiple endocrine neoplasia type 2 | RET mutation | BACA1 mutation | cancer genetics | MANAGEMENT | GUIDELINES | GENETICS & HEREDITY | medullary thyroid cancer | CANCER | P53 | Development and progression | Genetic aspects | Codon | Gene mutations | Genetic screening | Cancer | BRCA1 mutation
Journal Article
BMC Cancer, ISSN 1471-2407, 04/2015, Volume 15, Issue 1, p. 256
Journal Article
Pathology oncology research : POR, ISSN 1219-4956, 03/2019, pp. 1 - 7
Liver diseases such as liver cirrhosis, primary and metastatic liver cancers are still a major medical challenge. Syndecan-1 is one of the most important... 
Proteoglycans | Liver diseases | Liver | Viruses | Hepatocellular carcinoma | Diseases | Metastases | Cirrhosis | Hepatitis | Hepatocytes | Correlation analysis | Hepatitis C virus | Hepatitis C | Syndecan
Journal Article
Matrix Biology, ISSN 0945-053X, 04/2014, Volume 35, pp. 194 - 205
Hepatocellular carcinoma represents one of the most-rapidly spreading cancers in the world. In the majority of cases, an inflammation-driven fibrosis or... 
Small leucine-rich proteoglycan | Hepatocarcinogenesis | c-Myc | MSPR/RON | Receptor tyrosine kinase | β-Catenin | Cell signaling | C-Myc | GROWTH-FACTOR-RECEPTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | MECHANICAL-PROPERTIES | CELL BIOLOGY | CYCLIN-DEPENDENT KINASES | BREAST-CANCER | LEUCINE-RICH PROTEOGLYCANS | THERAPEUTIC TARGET | COLLAGEN FIBRILLOGENESIS | beta-Catenin | EXTRACELLULAR-MATRIX | HUMAN HEPATOCELLULAR-CARCINOMA | MAP Kinase Signaling System - physiology | Gene Expression Regulation, Neoplastic | Glycogen Synthase Kinase 3 beta | Carcinogenesis - metabolism | Receptor, Epidermal Growth Factor - metabolism | Carcinoma, Hepatocellular - genetics | Diethylnitrosamine | Statistics, Nonparametric | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Liver Neoplasms - pathology | Real-Time Polymerase Chain Reaction | Decorin - genetics | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Liver Neoplasms - genetics | Carcinogenesis - genetics | Decorin - deficiency | Glycogen Synthase Kinase 3 - metabolism | Receptor Protein-Tyrosine Kinases - metabolism | Blotting, Western | Thioacetamide | Animals | Models, Biological | Receptors, Somatomedin - metabolism | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Mice | Carcinoma, Hepatocellular - metabolism | Proteins | Development and progression | Carcinogenesis | RON | β-catenin | cell signaling | receptor tyrosine kinase | hepatocarcinogenesis | MSPR
Journal Article