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Journal of Immunology, ISSN 0022-1767, 04/2013, Volume 190, Issue 7, pp. 3289 - 3298
Journal Article
Science, ISSN 0036-8075, 2/2009, Volume 323, Issue 5915, pp. 793 - 797
Journal Article
Journal of immunology (Baltimore, Md. : 1950), ISSN 0022-1767, 6/2015, Volume 194, Issue 11, pp. 5120 - 5128
Interferon beta (IFNβ) is widely used in the treatment of multiple sclerosis, yet the mechanism facilitating its efficacy remains unclear. IL-2 production by... 
Journal Article
Journal of Immunology, ISSN 0022-1767, 01/2005, Volume 174, Issue 2, pp. 609 - 613
Journal Article
Nature Communications, ISSN 2041-1723, 09/2016, Volume 7, Issue 1, p. 12754
The ubiquitin-like protein ISG15 and its conjugation to proteins (ISGylation) are strongly induced by type I interferon. Influenza B virus encodes... 
A VIRUS | CONJUGATION | IMMUNITY | NS1 PROTEIN | ISOPEPTIDASE ACTIVITY | REPLICATION | RNA | STRUCTURAL BASIS | NUCLEOPROTEIN | MULTIDISCIPLINARY SCIENCES | DEFICIENCY
Journal Article
Immunity, ISSN 1074-7613, 04/2016, Volume 44, Issue 4, pp. 901 - 912
Sickness behavior and cognitive dysfunction occur frequently by unknown mechanisms in virus-infected individuals with malignancies treated with type I... 
influenza | endothelia | signal transduction | brain | neurons | virus infection | type I interferon | IFN | CXCL10 | MAVS | CXCR3 | IPS-1 | depression | behavior | epithelia | IFNAR1 | CELLS | DOUBLE-STRANDED-RNA | INFLAMMATION | IN-VIVO | RIG-I | CENTRAL-NERVOUS-SYSTEM | MICE | LONG-TERM POTENTIATION | MAJOR DEPRESSION | SYNAPTIC PLASTICITY | Brain | Virus infection | Neurons | Depression | Epithelia | Endothelia | Signal transduction | Type I interferon | Influenza | Behavior | IMMUNOLOGY | Cell Communication - immunology | Male | Interferon-Induced Helicase, IFIH1 | Epithelium - immunology | Signal Transduction - immunology | Interferon Type I - therapeutic use | Chemokine CXCL10 - immunology | Receptor, Interferon alpha-beta - genetics | DEAD-box RNA Helicases - metabolism | Cognition Disorders - psychology | DEAD Box Protein 58 | Brain - cytology | Virus Diseases - immunology | Cells, Cultured | Endothelium - immunology | Cognition Disorders - genetics | Endothelium - cytology | Endothelial Cells - immunology | Animals | Receptor, Interferon alpha-beta - immunology | Epithelial Cells - immunology | RNA, Double-Stranded - genetics | Receptors, CXCR3 - immunology | Mice | Adaptor Proteins, Signal Transducing - metabolism | Brain - immunology | Illness Behavior - physiology | Virus diseases | Medical colleges | RNA | Melanoma | Physiological aspects | Interferon | Biological response modifiers | Health aspects | Tretinoin | Neurophysiology | Endothelium | Multiple sclerosis | Cytokines | Cognitive ability | Viruses | Infections | Gene expression | Skin cancer | Brain research | Ligands | Software | Viral infections | Immune system
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2008, Volume 105, Issue 12, pp. 4808 - 4813
Journal Article
Journal Article
Journal of Molecular Biology, ISSN 0022-2836, 2005, Volume 353, Issue 2, pp. 271 - 281
Type I interferons activate cellular responses by forming a ternary complex with two receptor components, IFNAR1 and IFNAR2. While the binding of the IFNAR2... 
structure–function analysis | protein–protein interaction | type I interferons | IFNAR1 | Type I interferons | Protein-protein interaction | Structure-function analysis
Journal Article
Multiple Sclerosis Journal, ISSN 1352-4585, 11/2019, p. 135245851988861
Background: Alemtuzumab is given as two annual courses. Patients with continued disease activity may receive as-needed additional courses. Objective: To... 
Journal Article
Journal Article
Journal Article