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Cell Cycle, ISSN 1538-4101, 10/2007, Volume 6, Issue 20, pp. 2429 - 2433
The mitogen-activated protein kinase (MAPK) p38! is involved in numerous biological processes and is a drug target for inflammation-associated diseases.... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Development | JNK/c-Jun | MAPK | p38α | development | ACTIVATED PROTEIN-KINASE | SKIN TUMOR-DEVELOPMENT | JNK2 | DEFICIENCY | P38 MAP KINASE | CELL BIOLOGY | PATHWAY | cancer | SENESCENCE | C-JUN | p38 alpha | SIGNAL-REGULATED KINASE
Journal Article
Journal Article
Methods in Molecular Biology, ISSN 1064-3745, 2015, Volume 1267, pp. 297 - 305
Osteosarcoma (OS) is the most common primary tumor of bone with a high incidence in children. Treatment options for OS are limited, and once metastasized, the... 
Mineralization | Osteosarcoma | C-fos | Imaging | Isolation | Tumor | Bone | X-ray | c-fos | Anthraquinones - metabolism | Animals | Cell Separation | Cell Transformation, Neoplastic | Staining and Labeling | Alkaline Phosphatase - metabolism | Cell Line, Tumor | Mice | Bone Neoplasms - pathology | Carcinogenesis | Osteosarcoma - pathology | Disease Models, Animal
Journal Article
Nature, ISSN 0028-0836, 07/2008, Volume 454, Issue 7201, pp. 221 - 225
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 05/2017, Volume 214, Issue 5, pp. 1387 - 1409
Journal Article
Nature, ISSN 0028-0836, 02/2018, Volume 554, Issue 7693, pp. 533 - 537
Chronic inflammation increases the risk of developing one of several types of cancer. Inflammatory responses are currently thought to be controlled by... 
GENE-EXPRESSION | GLUCOCORTICOID SYNTHESIS | LRH-1 | C-JUN | AP-1 | MULTIDISCIPLINARY SCIENCES | Chromatin - metabolism | Epithelial Cells - metabolism | Receptors, Cytoplasmic and Nuclear - deficiency | Genes, jun - genetics | Humans | Transcriptome | Promoter Regions, Genetic - genetics | Transcription Factor AP-1 - metabolism | Cell Differentiation - genetics | Organ Specificity - genetics | Pancreatitis - genetics | Acinar Cells - metabolism | Gene Regulatory Networks - genetics | Gene Expression Regulation | Pancreas - pathology | Epithelial Cells - pathology | Receptors, Cytoplasmic and Nuclear - genetics | Pancreas - metabolism | Acinar Cells - pathology | Animals | Inflammation - genetics | Heterozygote | Mice | Chromatin - genetics | Receptors, Cytoplasmic and Nuclear - metabolism | Genetic aspects | Genetic transcription | Pancreas | Observations | Health aspects | Adenocarcinoma | Networks | Transcription factors | Glucocorticoids | Liver | Gene regulation | Switching theory | Genomes | Single-nucleotide polymorphism | Gene deletion | Kinases | Glucose metabolism | Clonal deletion | Intestine | Rodents | Deletion | Lipid metabolism | Phenotypes | Activator protein 1 | Pancreatitis | Inflammation | Metabolism | Gene expression | Cholesterol | Heterozygosity | Regeneration | Pancreatic cancer | Differentiation | Endoplasmic reticulum | Cancer | Tumors | Index Medicus
Journal Article