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American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 07/2009, Volume 297, Issue 1, pp. 277 - 282
Recombinant human activated protein C (rhAPC) is one of the treatment panels for improving vascular dysfunction in septic patients. In a previous study, we... 
Endothelial dysfunction | Endothelial nitric oxide synthase | Endotoxemia | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | NITRIC-OXIDE SYNTHASE | endotoxemia | RECEPTOR | FACTOR-KAPPA-B | SEPSIS | endothelial dysfunction | SEPTIC SHOCK PATIENTS | THERAPY | PATHWAY | INFLAMMATION | LIPOPOLYSACCHARIDE | endothelial nitric oxide synthase | PERIPHERAL VASCULAR DISEASE | EXPRESSION | Recombinant Proteins - therapeutic use | Mesenteric Arteries - physiology | Nitric Oxide Synthase Type II - biosynthesis | Humans | Endotoxemia - chemically induced | Endothelium, Vascular - drug effects | Male | Endotoxemia - pathology | Protein C - pharmacology | Vascular Resistance - physiology | Lipopolysaccharides | Nitric Oxide Synthase Type III - metabolism | NG-Nitroarginine Methyl Ester - pharmacology | Vasodilator Agents - pharmacology | Acetylcholine - pharmacology | Aorta - drug effects | Endotoxemia - drug therapy | Vasodilator Agents - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Arteries - drug effects | Recombinant Proteins - pharmacology | Enzyme Activation - drug effects | Blotting, Western | Cyclooxygenase Inhibitors - pharmacology | Nitric Oxide Synthase Type III - antagonists & inhibitors | Animals | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | NF-kappa B - biosynthesis | Mice | Acetylcholine - antagonists & inhibitors | Protein C - therapeutic use | Proteins | Rodents | Medical treatment | Coronary vessels | Nitric oxide | Physiology | Veins & arteries | Index Medicus
Journal Article
BRAIN, ISSN 0006-8950, 06/2013, Volume 136, pp. 1830 - 1845
Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy, or CADASIL, one of the most common inherited small vessel diseases... 
SUBCORTICAL INFARCTS | LIGAND-BINDING | cerebrovasculature | extracellular matrix proteins | NEUROSCIENCES | CLINICAL NEUROLOGY | SORSBYS FUNDUS DYSTROPHY | CADASIL | MUTANT NOTCH3 | SIGNALING PATHWAY | protein aggregation | SMALL-VESSEL DISEASE | Notch3 | TISSUE INHIBITOR | AUTOSOMAL-DOMINANT ARTERIOPATHY | SMOOTH-MUSCLE-CELLS | METALLOPROTEINASES-3 TIMP3
Journal Article
ANNALS OF NEUROLOGY, ISSN 0364-5134, 08/2018, Volume 84, Issue 2, pp. 246 - 259
ObjectiveCADASIL (cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy), caused by dominant mutations in the NOTCH3... 
PATHOGENESIS | LEUKOENCEPHALOPATHY | IMMUNIZATION | LOSS-OF-FUNCTION | SUBCORTICAL INFARCTS | ALZHEIMERS-DISEASE | MOUSE MODEL | TARGETING AMYLOID-BETA | AUTOSOMAL-DOMINANT ARTERIOPATHY | MONOCLONAL-ANTIBODY | NEUROSCIENCES | CLINICAL NEUROLOGY
Journal Article
Journal Article
The American Journal of Physiology, ISSN 0002-9513, 07/2009, Volume 297, Issue 1, p. H277
Recombinant human activated protein C (rhAPC) is one of the treatment panels for improving vascular dysfunction in septic patients. In a previous study, we... 
Blood circulation disorders | Diagnosis | Properties | Recombinant proteins | Health aspects | Arteries | Endothelium
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2013, Volume 33, Issue 1, pp. 76 - 86
OBJECTIVE—Notch3 is critically important for the structure and myogenic response of distal arteries, particularly of cerebral arteries. However, signaling... 
smooth muscle cell | cerebrovasculature | transcriptome | myogenic response | Notch3 | CELLS | MEMBRANE | KV1 CHANNELS | VASCULAR SMOOTH-MUSCLE | AMPA RECEPTORS | IN-VIVO | PERIPHERAL VASCULAR DISEASE | MICE | CEREBRAL-ARTERIES | HEMATOLOGY | EXPRESSION | RECEPTOR-BINDING PROTEIN | Muscle, Smooth, Vascular - metabolism | Receptors, Notch - metabolism | Nerve Tissue Proteins - deficiency | Male | Receptors, Notch - genetics | Gene Expression Profiling | Vasodilation | Alanine - analogs & derivatives | Kv1.5 Potassium Channel - genetics | Myocytes, Smooth Muscle - drug effects | Receptors, Notch - deficiency | Immunoglobulin J Recombination Signal Sequence-Binding Protein - genetics | Myocytes, Smooth Muscle - metabolism | Alanine - pharmacology | Muscle, Smooth, Vascular - drug effects | Vasoconstrictor Agents - pharmacology | Immunoglobulin J Recombination Signal Sequence-Binding Protein - metabolism | Vasodilator Agents - pharmacology | Signal Transduction | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Gene Expression Regulation | Reverse Transcriptase Polymerase Chain Reaction | Nerve Tissue Proteins - genetics | Vasoconstriction - drug effects | Mice, Knockout | Azepines - pharmacology | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Amyloid Precursor Protein Secretases - metabolism | Animals | Carrier Proteins - metabolism | Cerebral Arteries - metabolism | Kv1.5 Potassium Channel - metabolism | Receptor, Notch3 | Mice | Muscle, Smooth, Vascular - growth & development | Amyloid Precursor Protein Secretases - antagonists & inhibitors | Index Medicus
Journal Article
Journal Article
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2017, Volume 127, Issue 12, pp. 4516 - 4526
Journal Article
ACTA PHYSIOLOGICA, ISSN 1748-1708, 06/2016, Volume 217, pp. 20 - 20
Conference Proceeding
CEREBROVASCULAR DISEASES, ISSN 1015-9770, 2014, Volume 37, pp. 162 - 162
Conference Proceeding
Hypertension, ISSN 0194-911X, 01/2010, Volume 55, Issue 1, pp. 109 - 115
Angiotensin II is a potent growth factor involved in arterial wall homeostasis. In resistance arteries, chronic increases in blood flow induce a rise in... 
ERK1/2 | Angiotensin I-converting enzyme inhibitors | Resistance arteries | Shear stress | Angiotensin II type 1 receptor inhibitor | Angiotensin II | Blood flow | VASCULAR HYPERTROPHY | ACTIVATION | SPONTANEOUSLY HYPERTENSIVE-RATS | OBESE ZUCKER RATS | RECEPTOR | angiotensin II type 1 receptor inhibitor | resistance arteries | BLOOD-FLOW | INDUCED CONTRACTION | IN-VITRO | shear stress | TONE | angiotensin II | PERIPHERAL VASCULAR DISEASE | DYSFUNCTION | blood flow | angiotensin I-converting enzyme inhibitors | Mesenteric Arteries - physiopathology | Reactive Oxygen Species - metabolism | Tetrazoles - pharmacology | Nitriles - pharmacology | Rats, Wistar | Hydralazine - pharmacology | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Mesenteric Arteries - pathology | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Spin Labels | Angiotensin II Type 1 Receptor Blockers - pharmacology | Vascular Resistance - physiology | Perindopril - pharmacology | Cyclic N-Oxides - pharmacology | Vascular Resistance - drug effects | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Nitric Oxide Synthase Type III - metabolism | NG-Nitroarginine Methyl Ester - pharmacology | Butadienes - pharmacology | Vasodilator Agents - pharmacology | Angiotensin II - metabolism | Rats | Antioxidants - pharmacology | Mesenteric Arteries - metabolism | Nitric Oxide Synthase Type III - genetics | Blotting, Western | Mice, Knockout | Nitric Oxide Synthase Type III - antagonists & inhibitors | Microscopy, Confocal | Animals | Blood Flow Velocity | Benzimidazoles - pharmacology | Mice | Angiotensin II - physiology | Index Medicus
Journal Article