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Annals of Neurology, ISSN 0364-5134, 08/2018, Volume 84, Issue 2, pp. 246 - 259
ObjectiveCADASIL (cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy), caused by dominant mutations in the NOTCH3... 
Immunohistochemistry | Brain | Phenotypes | Deposits | Pathogenesis | Vasodilators | Medical treatment | Blood vessels | Electron microscopy | Doppler effect | Substantia alba | Leukoencephalopathy | Blood | Arteries | Blood flow | Cerebral blood flow | Immunotherapy | Monoclonal antibodies | Affinity | Topical application | Mutation | Lesions | Deposition | Recombinant
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2017, Volume 127, Issue 12, pp. 4516 - 4526
Journal Article
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2013, Volume 33, Issue 1, pp. 76 - 86
OBJECTIVE—Notch3 is critically important for the structure and myogenic response of distal arteries, particularly of cerebral arteries. However, signaling... 
smooth muscle cell | cerebrovasculature | transcriptome | myogenic response | Notch3 | CELLS | MEMBRANE | KV1 CHANNELS | VASCULAR SMOOTH-MUSCLE | AMPA RECEPTORS | IN-VIVO | PERIPHERAL VASCULAR DISEASE | MICE | CEREBRAL-ARTERIES | HEMATOLOGY | EXPRESSION | RECEPTOR-BINDING PROTEIN | Muscle, Smooth, Vascular - metabolism | Receptors, Notch - metabolism | Nerve Tissue Proteins - deficiency | Male | Receptors, Notch - genetics | Gene Expression Profiling | Vasodilation | Alanine - analogs & derivatives | Kv1.5 Potassium Channel - genetics | Myocytes, Smooth Muscle - drug effects | Receptors, Notch - deficiency | Immunoglobulin J Recombination Signal Sequence-Binding Protein - genetics | Myocytes, Smooth Muscle - metabolism | Alanine - pharmacology | Muscle, Smooth, Vascular - drug effects | Vasoconstrictor Agents - pharmacology | Immunoglobulin J Recombination Signal Sequence-Binding Protein - metabolism | Vasodilator Agents - pharmacology | Signal Transduction | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Gene Expression Regulation | Reverse Transcriptase Polymerase Chain Reaction | Nerve Tissue Proteins - genetics | Vasoconstriction - drug effects | Mice, Knockout | Azepines - pharmacology | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Amyloid Precursor Protein Secretases - metabolism | Animals | Carrier Proteins - metabolism | Cerebral Arteries - metabolism | Kv1.5 Potassium Channel - metabolism | Receptor, Notch3 | Mice | Muscle, Smooth, Vascular - growth & development | Amyloid Precursor Protein Secretases - antagonists & inhibitors | Index Medicus
Journal Article
Brain, ISSN 0006-8950, 06/2013, Volume 136, Issue 6, pp. 1830 - 1845
Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy, or CADASIL, one of the most common inherited small vessel diseases... 
CADASIL | cerebrovasculature | extracellular matrix proteins | Original | protein aggregation | Notch3
Journal Article
Annals of Neurology, ISSN 0364-5134, 03/2016, Volume 79, Issue 3, pp. 387 - 403
Journal Article
Journal Article
Hypertension, ISSN 0194-911X, 01/2010, Volume 55, Issue 1, pp. 109 - 115
Angiotensin II is a potent growth factor involved in arterial wall homeostasis. In resistance arteries, chronic increases in blood flow induce a rise in... 
ERK1/2 | Angiotensin I-converting enzyme inhibitors | Resistance arteries | Shear stress | Angiotensin II type 1 receptor inhibitor | Angiotensin II | Blood flow | VASCULAR HYPERTROPHY | ACTIVATION | SPONTANEOUSLY HYPERTENSIVE-RATS | OBESE ZUCKER RATS | RECEPTOR | angiotensin II type 1 receptor inhibitor | resistance arteries | BLOOD-FLOW | INDUCED CONTRACTION | IN-VITRO | shear stress | TONE | angiotensin II | PERIPHERAL VASCULAR DISEASE | DYSFUNCTION | blood flow | angiotensin I-converting enzyme inhibitors | Mesenteric Arteries - physiopathology | Reactive Oxygen Species - metabolism | Tetrazoles - pharmacology | Nitriles - pharmacology | Rats, Wistar | Hydralazine - pharmacology | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Mesenteric Arteries - pathology | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Spin Labels | Angiotensin II Type 1 Receptor Blockers - pharmacology | Vascular Resistance - physiology | Perindopril - pharmacology | Cyclic N-Oxides - pharmacology | Vascular Resistance - drug effects | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Nitric Oxide Synthase Type III - metabolism | NG-Nitroarginine Methyl Ester - pharmacology | Butadienes - pharmacology | Vasodilator Agents - pharmacology | Angiotensin II - metabolism | Rats | Antioxidants - pharmacology | Mesenteric Arteries - metabolism | Nitric Oxide Synthase Type III - genetics | Blotting, Western | Mice, Knockout | Nitric Oxide Synthase Type III - antagonists & inhibitors | Microscopy, Confocal | Animals | Blood Flow Velocity | Benzimidazoles - pharmacology | Mice | Angiotensin II - physiology | Index Medicus
Journal Article
Journal Article
Journal Article
Annals of neurology, 08/2018, Volume 84, Issue 2, p. 246
CADASIL (cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy), caused by dominant mutations in the NOTCH3 receptor, is... 
Journal Article