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Oncogene, ISSN 0950-9232, 01/2012, Volume 31, Issue 1, pp. 68 - 79
Ovarian cancers migrate and metastasize over the surface of the peritoneal cavity. Consequently, dysregulation of mechanisms that limit cell migration may be... 
migration suppression | Stat3 | RhoA GTPase | cytoskeleton | tumor-suppressor gene ARHI | LOCALIZATION | ACTIVATION | TYROSINE PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | G-PROTEIN | BREAST | FIBROBLASTS | CELL BIOLOGY | FAK | ONCOLOGY | EPIDERMAL-GROWTH-FACTOR | GENETICS & HEREDITY | FOCAL ADHESION KINASE | RHO-GTPASES | rho GTP-Binding Proteins - genetics | Humans | Ovarian Neoplasms - pathology | Focal Adhesions | rhoA GTP-Binding Protein - antagonists & inhibitors | rhoA GTP-Binding Protein - physiology | Focal Adhesion Protein-Tyrosine Kinases - antagonists & inhibitors | Focal Adhesion Protein-Tyrosine Kinases - physiology | Ovarian Neoplasms - genetics | STAT3 Transcription Factor - physiology | Genes, Tumor Suppressor - physiology | Cell Line, Tumor | Female | Signal Transduction - physiology | Janus Kinase 2 - physiology | STAT3 Transcription Factor - antagonists & inhibitors | Cell Movement | Cancer cells | Physiological aspects | Tumor suppressor genes | Cellular signal transduction | Genetic aspects | Research | Ovarian cancer | Genes | Tumors | Cell adhesion & migration | Phosphorylation | Stat3 protein | Homology | RhoA protein | Ras protein | siRNA | Chemotaxis | Nuclear transport | Fibers | Interleukin 6 | Signal transduction | Epidermal growth factor | Actin | Janus kinase 2 | Focal adhesion kinase | Cell migration | Cytoplasm | Guanosinetriphosphatase | Peritoneum | Tumor suppressor gene ARHI
Journal Article
Journal Article
Oncogene, ISSN 0950-9232, 11/2006, Volume 25, Issue 52, pp. 6986 - 6996
We determined the impact of HER2 signaling on two proangiogenic factors, vascular endothelial growth factor ( VEGF) and interleukin-8(IL-8), and on an... 
Angiogenesis | TSP-1 | p38 | PI3K | VEGF | HER2 | IL-8 | Trastuzumab | angiogenesis | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE INHIBITOR P27(KIP1) | CELL-CYCLE ARREST | P38 MAP KINASE | CELL BIOLOGY | trastuzumab | IN-VITRO | RECEPTOR TYROSINE KINASES | ONCOLOGY | METASTATIC BREAST-CANCER | GENETICS & HEREDITY | ANTI-HER2 ANTIBODY | TUMOR-GROWTH | UP-REGULATION | ENDOTHELIAL GROWTH-FACTOR | Immunohistochemistry | Humans | Receptor, ErbB-2 - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Vascular Endothelial Growth Factor A - metabolism | Mammary Neoplasms, Experimental - metabolism | Antibodies, Monoclonal, Humanized | Transfection | Female | Mammary Neoplasms, Experimental - blood supply | Antineoplastic Agents - pharmacology | Receptor, ErbB-2 - antagonists & inhibitors | Receptor, ErbB-2 - immunology | p38 Mitogen-Activated Protein Kinases - metabolism | Interleukin-8 - metabolism | Oncogene Protein v-akt - metabolism | Mammary Neoplasms, Experimental - drug therapy | Antibodies, Monoclonal - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | p38 Mitogen-Activated Protein Kinases - drug effects | Animals | Signal Transduction - drug effects | Mice, Nude | Signal Transduction - physiology | Mice | Neovascularization, Pathologic - metabolism | Thrombospondin 1 - metabolism | Cell growth | Chemotherapy | Breast cancer | Gene expression | Tumors
Journal Article
Journal Article
INTERNATIONAL JOURNAL OF GYNECOLOGICAL CANCER, ISSN 1048-891X, 11/2005, Volume 15, pp. 274 - 281
Journal Article
Cell Death and Disease, ISSN 2041-4889, 08/2015, Volume 6, Issue 8, pp. e1836 - e1836
Autophagy can sustain or kill tumor cells depending upon the context. The mechanism of autophagy-associated cell death has not been well elucidated and... 
DIRAS3 | APOPTOSIS | NECROPTOSIS | GROWTH | KINASE | TUMOR-SUPPRESSOR GENE | DOWN-REGULATION | RESISTANCE | INDUCTION | INHIBITOR | CELL BIOLOGY | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | RNA-Binding Proteins - genetics | Reactive Oxygen Species - metabolism | Receptor, ErbB-2 - genetics | Humans | Caspase 3 - metabolism | Gene Expression Regulation, Neoplastic | Ovarian Neoplasms - pathology | Receptor, ErbB-2 - metabolism | Autophagy - drug effects | Nuclear Pore Complex Proteins - genetics | Ovarian Neoplasms - genetics | Chloroquine - pharmacology | Proto-Oncogene Proteins c-bcl-2 - metabolism | Proteolysis | Mitogen-Activated Protein Kinase 1 - genetics | Caspase 3 - genetics | Female | Antineoplastic Agents - pharmacology | Ovarian Neoplasms - metabolism | Autophagy - genetics | Ovarian Neoplasms - drug therapy | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Nuclear Pore Complex Proteins - metabolism | rho GTP-Binding Proteins - genetics | Cisplatin - pharmacology | X-Linked Inhibitor of Apoptosis Protein - genetics | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Poly(ADP-ribose) Polymerases - metabolism | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Poly(ADP-ribose) Polymerases - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | rho GTP-Binding Proteins - metabolism | X-Linked Inhibitor of Apoptosis Protein - metabolism | Cell Line, Tumor | G1 Phase Cell Cycle Checkpoints - drug effects | Proto-Oncogene Proteins c-bcl-2 - genetics | RNA-Binding Proteins - metabolism | Drug Resistance, Neoplasm - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | Original
Journal Article
BJOG: An International Journal of Obstetrics & Gynaecology, ISSN 1470-0328, 01/2012, Volume 119, Issue 2, pp. 134 - 136
Journal Article
Cancer Research, ISSN 0008-5472, 08/2001, Volume 61, Issue 15, pp. 5895 - 5904
Journal Article