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Biochemical Journal, ISSN 0264-6021, 09/2004, Volume 382, Issue 3, pp. 877 - 884
Metformin, a drug widely used in the treatment of Type 11 diabetes, has recently received attention owing to new findings regarding its mitochondrial and... 
Oxidative stress | Complex 1 | Mitochondria | Metformin | Cell death | Permeability transition pore (PTP) | complex 1 | DEPENDENT ANION CHANNEL | APOPTOSIS | ACTIVATED PROTEIN-KINASE | cell death | HEXOKINASE | mitochondria | BIOCHEMISTRY & MOLECULAR BIOLOGY | metformin | COMMON MECHANISM | INSULIN | THERAPY | RESPIRATORY-CHAIN COMPLEX | permeability transition pore (PTP) | PORE | TYPE-2 DIABETES-MELLITUS | oxidative stress | Electron Transport Complex I - antagonists & inhibitors | Cytochromes c - metabolism | Humans | KB Cells | Metformin - pharmacology | Ion Channels - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Permeability | Hypoglycemic Agents - pharmacology | Mitochondrial Membrane Transport Proteins | Ion Channels - metabolism | Oxygen Consumption - drug effects | Citrate (si)-Synthase - metabolism | Intracellular Membranes - drug effects | Cell Death - drug effects | Intracellular Membranes - metabolism | Electron Transport Complex I | Ion Channels | Biochemistry, Molecular Biology | Oxygen Consumption | Cytochromes c | Intracellular Membranes | Citrate (si)-Synthase | Life Sciences | Hypoglycemic Agents | Cell Death | PTP, permeability transition pore | CsA, cyclosporin A | AMPK, AMP-activated protein kinase | PLSD, protected least-significant difference | CCCP, carbonyl cyanide m-chlorophenylhydrazone | tBH, t-butyl hydroperoxide | TMPD, N,N,N′,N′-tetramethyl-1,4-phenylenediamine | VDAC, voltage-dependent anion channel | ROS, reactive oxygen species
Journal Article
Journal of Bioenergetics and Biomembranes, ISSN 0145-479X, 2/2006, Volume 38, Issue 1, pp. 33 - 42
Mitochondrial reactive oxygen species (ROS) production was investigated in mitochondria extracted from liver of rats treated with or without metformin, a mild... 
Biochemistry, general | Antimycin | Animal Biochemistry | Malonate | Rat liver mitochondria | Bioorganic Chemistry | Rotenone | Chemistry | Oxidative phosphorylation | ROS | Metformin | Membrane potential | Animal Anatomy / Morphology / Histology | Organic Chemistry | OXIDATIVE-PHOSPHORYLATION | rat liver mitochondria | SUPEROXIDE-PRODUCTION | UBIQUINONE OXIDOREDUCTASE | GENERAL PROPERTIES | rotenone | metformin | malonate | oxidative phosphorylation | CELL-DEATH | CELL BIOLOGY | CYTOCHROME-C | MITOCHONDRIAL PERMEABILITY TRANSITION | BIOPHYSICS | membrane potential | HYDROGEN-PEROXIDE | ENERGY-METABOLISM | antimycin | HYPERGLYCEMIC DAMAGE | Electron Transport | Phosphorylation | Electron Transport Complex I - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Glutamic Acid - pharmacology | Oxidation-Reduction | Rats, Wistar | Malates - pharmacology | Metformin - pharmacology | Rats | Malonates - pharmacology | Adenosine Triphosphate - biosynthesis | Hypoglycemic Agents - pharmacology | Mitochondria, Liver - physiology | Mitochondria, Liver - drug effects | Animals | Membrane Potentials | Succinic Acid - pharmacology | 2,4-Dinitrophenol - pharmacology | Rotenone - pharmacology | Electron Transport Complex I - physiology | In Vitro Techniques | Hypoglycemic agents | Oxygen | Life Sciences | Cellular Biology
Journal Article
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 05/2012, Volume 682, Issue 1-3, pp. 92 - 98
Metyrapone is a cytochrome P inhibitor that protects against ischemia- and excitotoxicity-induced brain damages in rodents. This study examines whether... 
Mitochondrion | Hyperglycemia | Lactate | Magnetic Resonance Spectroscopy | Glutamine | Hypothermia | GLUTAMATE REUPTAKE | SYNAPTIC FUNCTION | CORTICOSTERONE SYNTHESIS | ISCHEMIA | GLUCOSE-UTILIZATION | 11-BETA-HYDROXYSTEROID DEHYDROGENASE | INHIBITION | N-ACETYLASPARTATE | PHARMACOLOGY & PHARMACY | RAT-BRAIN | Transcription, Genetic - drug effects | AMP-Activated Protein Kinases - metabolism | Metyrapone - pharmacology | TOR Serine-Threonine Kinases - metabolism | Male | Hippocampus - drug effects | RNA, Messenger - metabolism | Oxygen - metabolism | Prosencephalon - drug effects | Brain - metabolism | Prosencephalon - metabolism | Brain - cytology | RNA, Messenger - genetics | Enzyme Inhibitors - pharmacology | Rats | HSP70 Heat-Shock Proteins - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Hippocampus - cytology | Rats, Sprague-Dawley | Brain - drug effects | Hippocampus - metabolism | Animals | Prosencephalon - cytology | Energy Metabolism - drug effects | Brain | RNA | Blood sugar | Corticosterone | Metyrapone | Heat shock proteins | Mitochondrial DNA | Glutamate | Lactates | Metabolites | Analysis | Physiological aspects | GABA | Brain damage | Aspartate | Nuclear magnetic resonance spectroscopy | Glucose metabolism | TOR protein | Neuroprotection | gamma -Aminobutyric acid | Energy metabolism | Glucocorticoids | mRNA | Blood | Western blotting | Blood levels | Polymerase chain reaction | Mitochondria | Adenosine kinase | Protein kinase | Temperature effects | Lactic acid | c-Fos protein | Hippocampus | Life Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 05/2018, Volume 115, Issue 19, pp. E4416 - E4425
Journal Article
The Journal of Physiology, ISSN 0022-3751, 12/2017, Volume 595, Issue 23, pp. 7049 - 7062
Journal Article
Journal Article
The Journal of experimental biology, ISSN 0022-0949, 05/2019, Volume 222, Issue 10, p. jeb194969
It is now well established that the intrauterine environment is of major importance for offspring health during later life. Endurance training during pregnancy... 
Offspring | Endurance training | Oxidative phosphorylation | Intrauterine environment | OXIDATIVE STRESS | FATTY-ACIDS | ADAPTATIONS | INSULIN SENSITIVITY | CHILDHOOD | BLOOD-PRESSURE | ENDURANCE EXERCISE | BIOLOGY | PHYSICAL-ACTIVITY | HEALTH | ELECTRON-TRANSPORT | Life Sciences
Journal Article
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