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European Journal of Cancer, ISSN 0959-8049, 04/2017, Volume 75, p. 63
Neuroblastoma is predominantly characterised by chromosomal rearrangements. Next to V-Myc Avian Myelocytomatosis Viral Oncogene Neuroblastoma Derived Homolog... 
Cell survival | Differentiation (biology) | Chromosome 7 | Homology | Neuroblastoma | Myc protein | Chromosome rearrangements | Gene expression | Survival | Amplification | Polycomb group proteins | Gene silencing | Inhibitors | Lysine | Medical prognosis | Cell lines | Cell cycle | Histone methyltransferase | Catalysis | Inhibition | Chromosomes | Histone H3 | Apoptosis | Cancer
Journal Article
Cancer Research, ISSN 0008-5472, 03/2016, Volume 76, Issue 5 Supplement, pp. B09 - B09
Journal Article
Journal Article
Oncotarget, ISSN 1949-2553, 2016, Volume 7, Issue 19, pp. 27946 - 27958
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2015, Volume 10, Issue 12, p. e0145744
Tumor cells might resist therapy with ionizing radiation (IR) by non-homologous end-joining (NHEJ) of IR-induced double-strand breaks. One of the key players... 
CANCER-CELLS | RECOMBINATION | STRAND BREAK REPAIR | INHIBITION | MULTIDISCIPLINARY SCIENCES | IONIZING-RADIATION | IDENTIFICATION | CYTOTOXICITY | GEMCITABINE | DAMAGE | DNA Damage - drug effects | DNA End-Joining Repair - drug effects | Apoptosis - drug effects | DNA End-Joining Repair - genetics | Humans | Radiation-Sensitizing Agents - pharmacology | Morpholines - pharmacology | Radiation Tolerance - genetics | Apoptosis - genetics | Nuclear Proteins - metabolism | DNA-Binding Proteins - metabolism | DNA-Activated Protein Kinase - metabolism | Neuroblastoma - drug therapy | Neuroblastoma - radiotherapy | Cell Line, Tumor | DNA Damage - genetics | Neuroblastoma - metabolism | Chromones - pharmacology | Radiation, Ionizing | Molecular targeted therapy | Innovations | Development and progression | Genetic aspects | Neuroblastoma | Radiotherapy | Protein kinases | Health aspects | Methods | Cell culture | Protein kinase C | DNA damage | Homology | Kinases | Cancer therapies | DNA repair | Neuroblastoma cells | Proteins | Ionizing radiation | Radiosensitization | Penicillin | Cell cycle | Fibroblasts | Catalysis | Inhibition | Pretreatment | Deoxyribonucleic acid--DNA | Tumor cells | I.R. radiation | Breast cancer | Radiation therapy | Gene expression | DNA-dependent protein kinase | Cell lines | Non-homologous end joining | Combined treatment | Synergistic effect | Cervical cancer | Tumors | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article
PLoS ONE, 12/2015, Volume 10, Issue 12
Tumor cells might resist therapy with ionizing radiation (IR) by non-homologous end-joining (NHEJ) of IR-induced double-strand breaks. One of the key players... 
Journal Article
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