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1. Full Text Mn- and Fe-porphyrin-based SOD mimics are powerful suppressors of itch in histamine-dependent and -independent mouse models
by Batinic-Haberle, Ines
Free Radical Biology and Medicine, ISSN 0891-5849, 11/2018, Volume 128, p. S100
Histamine | Pruritus | Analysis | Porphyrins
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2. Full Text 231 - Mn- and Fe-porphyrin-based SOD mimics are powerful suppressors of itch in histamine-dependent and -independent mouse models
by Batinic-Haberle, Ines
Free Radical Biology and Medicine, ISSN 0891-5849, 11/2018, Volume 128, pp. S100 - S100
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3. Full Text Superoxide dismutase mimics: Chemistry, pharmacology, and therapeutic potential
by Batinić-Haberle, Ines and Rebouças, Júlio S and Spasojević, Ivan
Antioxidants and Redox Signaling, ISSN 1523-0864, 09/2010, Volume 13, Issue 6, pp. 877 - 918
Oxidative stress has become widely viewed as an underlying condition in a number of diseases, such as ischemia-reperfusion disorders, central nervous system... 
ISCHEMIA-REPERFUSION INJURY | 4-BENZOIC ACID PORPHYRIN | PROTEIN-TYROSINE NITRATION | WATER-SOLUBLE PORPHYRINS | CARRAGEENAN-INDUCED PLEURISY | BIOCHEMISTRY & MOLECULAR BIOLOGY | DEFICIENT ESCHERICHIA-COLI | ENDOCRINOLOGY & METABOLISM | PEROXYNITRITE DECOMPOSITION CATALYST | AMYOTROPHIC-LATERAL-SCLEROSIS | NF-KAPPA-B | DIABETIC VASCULAR COMPLICATIONS | Polyamines - chemistry | Humans | Porphyrins - chemistry | Porphyrins - pharmacology | Superoxide Dismutase - chemistry | Biomimetic Materials - pharmacology | Polyamines - pharmacology | Animals | Manganese - metabolism | Organometallic Compounds - chemistry | Biomimetic Materials - chemistry | Organometallic Compounds - pharmacology | Superoxide Dismutase - metabolism | Oxidative stress | Care and treatment | Physiological aspects | Superoxide dismutase | Genetic aspects | Genetic transcription | Research | Health aspects | Cancer | Comprehensive Invited Reviews
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4. Redox-active therapeutics
by Batinic-Haberle, Ines and Rebouças, Júlio S and Spasojević, Ivan
2016, Oxidative stress in applied basic research and clinical practice, ISBN 9783319307053
Oxidative stress | Oxidation-reduction reaction | Therapeutic use
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5. Full Text Oxidants, antioxidants and the ischemic brain
by Warner, David S and Sheng, Huaxin and Batinić-Haberle, Ines
Journal of Experimental Biology, ISSN 0022-0949, 08/2004, Volume 207, Issue 18, pp. 3221 - 3231
Despite numerous defenses, the brain is vulnerable to oxidative stress resulting from ischemia/reperfusion. Excitotoxic stimulation of superoxide and nitric... 
Brain | Oxidative stress | Ischemia | Antioxidant | MITOCHONDRIAL CYTOCHROME-C | POLY(ADP-RIBOSE) POLYMERASE | antioxidant | REDUCES INFARCT SIZE | NITRIC-OXIDE SYNTHASE | FOCAL CEREBRAL-ISCHEMIA | INTRACELLULAR FLUORESCENCE DETECTION | NEURONAL CELL-DEATH | brain | ischemia | EXTRACELLULAR-SUPEROXIDE-DISMUTASE | TERT-BUTYL-NITRONE | BIOLOGY | HYDROGEN-PEROXIDE | oxidative stress | Glutathione Peroxidase - metabolism | Xanthine Oxidase - metabolism | Antioxidants - metabolism | Humans | Oxidative Stress - physiology | Oxidants - metabolism | Brain Ischemia - metabolism | Brain Ischemia - physiopathology | Lipid Peroxidation - physiology | Catalase - metabolism | Mitochondrial Membrane Transport Proteins | Poly(ADP-ribose) Polymerases - metabolism | DNA Damage - physiology | Ion Channels - metabolism | Spin Trapping | Nitric Oxide Synthase - metabolism | Apoptosis - physiology | Chelating Agents - metabolism | Superoxide Dismutase - metabolism | Physiological aspects | Cerebral ischemia | Research
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6. Full Text Redox-Active Therapeutics
by Batinić-Haberle, Ines and Reboucas, Julio S and Spasojević, Ivan
10/2016, 1st ed. 2016, Oxidative Stress in Applied Basic Research and Clinical Practice, ISBN 9783319307039, 709
This essential volume comprehensively discusses redox-active therapeutics, focusing particularly on their molecular design, mechanistic, pharmacological and... 
Drug development | Biology, life sciences | Oncology | Life Sciences | Cancer Research | Oxidative Stress | Enzymology
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7. Full Text Catalysis of Sulfite Oxidation Adds to the Array of Biologically Relevant Reactions of Cationic Mn(III) Porphyrins, Widely Known as SOD-Mimics
by Velayutham, Murugesan and Batinic-Haberle, Ines and Tovmasyan, Artak
Free Radical Biology and Medicine, ISSN 0891-5849, 11/2016, Volume 100, p. S112
Sulfite is generated in vivo as a result of metabolism of sulfur containing amino acids methionine and cysteine. Sulfite is consumed as... 
Oxidases | Medical colleges | Sulfur compounds | Hydrogen peroxide | Thiols | Amino acids | Superoxide | Molybdenum compounds | Analysis | Nitric oxide | Physiological aspects | Oxidation-reduction reaction | Catalysis | Porphyrins
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8. Full Text 248 - Catalysis of Sulfite Oxidation Adds to the Array of Biologically Relevant Reactions of Cationic Mn(III) Porphyrins, Widely Known as SOD-Mimics
by Velayutham, Murugesan and Batinic-Haberle, Ines and Tovmasyan, Artak
Free Radical Biology and Medicine, ISSN 0891-5849, 11/2016, Volume 100, pp. S112 - S112
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9. Full Text Mn Porphyrin-Based Redox-Active Drugs: Differential Effects as Cancer Therapeutics and Protectors of Normal Tissue Against Oxidative Injury
by Batinic-Haberle, Ines and Tovmasyan, Artak and Spasojevic, Ivan
Antioxidants & Redox Signaling, ISSN 1523-0864, 12/2018, Volume 29, Issue 16, pp. 1691 - 1724
Significance: After approximatelty three decades of research, two Mn(III) porphyrins (MnPs), MnTE-2-PyP 5+ (BMX-010, AEOL10113) and MnTnBuOE-2-PyP 5+... 
Comprehensive Invited Review | SOD mimics | M40403/GC4403 | Mn porphyrins | radioprotection | cancer | BMX-001 | protein cysteine oxidation | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PHARMACOLOGICAL ASCORBATE | INDUCED LUNG INJURY | DEPENDENT GENE-EXPRESSION | S-TRANSFERASE-PI | CATALYTIC METALLOPORPHYRIN ANTIOXIDANT | REACTIVE OXYGEN | MANGANESE-SUPEROXIDE-DISMUTASE | ENDOCRINOLOGY & METABOLISM | NF-KAPPA-B | RADIATION-INDUCED DAMAGE | Drugs | Medical research | NF-κB protein | Head | Protectors | Transcription | Therapeutic applications | Tumor cells | Metals | Clinical trials | MAP kinase | Superoxide dismutase | Kinases | Tissues | Radioprotection | Proteins | Cellular communication | Head and neck cancer | Catalysis | Coordination compounds | Cancer | Porphyrins | Tumors
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10. Redox-Active Therapeutics
by Batinić-Haberle, Ines and Rebouças, Júlio S and Spasojević, Ivan and SpringerLink (Online service)
2016, Oxidative Stress in Applied Basic Research and Clinical Practice, ISBN 9783319307053
This essential volume comprehensively discusses redox-active therapeutics, focusing particularly on their molecular design, mechanistic, pharmacological and... 
Oxidative stress | Cancer research | Life sciences | Enzymology
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11. Full Text IDH1 and IDH2 Mutations in Gliomas
by Yan, Hai and Parsons, D. Williams and Jin, Genglin and McLendon, Roger and Rasheed, B. Ahmed and Yuan, Weishi and Kos, Ivan and Batinic-Haberle, Ines and Jones, Siân and Riggins, Gregory J and Friedman, Henry and Friedman, Allan and Reardon, David and Herndon, James and Kinzler, Kenneth W and Velculescu, Victor E and Vogelstein, Bert and Bigner, Darell D
The New England Journal of Medicine, ISSN 0028-4793, 02/2009, Volume 360, Issue 8, pp. 765 - 773
Isocitrate dehydrogenases, encoded by the IDH1 and IDH2 genes, catalyze the reduction of NADP + to NADPH in the brain. One or the other of these two genes was... 
PATHWAYS | MAJORITY | MEDICINE, GENERAL & INTERNAL | GLIOBLASTOMA | AMPLIFICATION | MECHANISM | MALIGNANT GLIOMAS | COMPARATIVE GENOMIC HYBRIDIZATION | EXPRESSION | FACTOR RECEPTOR GENE | DEPENDENT ISOCITRATE DEHYDROGENASE | Astrocytoma - genetics | Brain Neoplasms - enzymology | Glioma - enzymology | Oligodendroglioma - genetics | Humans | Kaplan-Meier Estimate | Brain Neoplasms - genetics | Isocitrate Dehydrogenase - genetics | Disease Progression | Glioma - genetics | Young Adult | Glioblastoma - genetics | Neoplasms - genetics | DNA Mutational Analysis | Adult | Isocitrate Dehydrogenase - metabolism | Mutation | Tumor Cells, Cultured | Gene mutations | Gliomas | Causes of | Development and progression | Genetic aspects | Research | Isocitrate dehydrogenase | Leukemia | Brain cancer | Tumors
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12. Full Text An educational overview of the chemistry, biochemistry and therapeutic aspects of Mn porphyrins – From superoxide dismutation to H2O2-driven pathways
by Batinic-Haberle, Ines and Tovmasyan, Artak and Spasojevic, Ivan
Redox Biology, ISSN 2213-2317, 08/2015, Volume 5, pp. 43 - 65
Most of the SOD mimics thus far developed belong to the classes of Mn-(MnPs) and Fe porphyrins(FePs), Mn(III) salens, Mn(II) cyclic polyamines and metal salts.... 
Therapeutic effects | Mn-porphyrin-based non-SOD mimics | Reactivities of Mn porphyrins in aqueous solutions, cells and animals | Fe porphyrin-based SOD mimics | Mechanism(s) of action(s) of Mn porphyrins | Mn-porphyrin-based SOD mimics | Reactivities of Mn porphyrins in aqueous | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | ESCHERICHIA-COLI | PANCREATIC-CANCER | solutions, cells and animals | SACCHAROMYCES-CEREVISIAE | CELL-DEATH | SOD MIMICS | REDOX MODULATORS | NITRIC-OXIDE | MN(III) N-ALKYLPYRIDYLPORPHYRINS | MANGANESE-PORPHYRIN | Humans | Iron - chemistry | Porphyrins - chemistry | Superoxides - chemistry | Coordination Complexes - metabolism | Structure-Activity Relationship | Manganese - chemistry | Coordination Complexes - chemistry | Hydrogen Peroxide - metabolism | Brain - metabolism | Hydrogen Peroxide - chemistry | Superoxide Dismutase - chemistry | Superoxides - metabolism | Superoxide Dismutase - metabolism
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13. Full Text Manganese superoxide dismutase regulates mTORC2 mediated UVB-induced mitophagy
by Dhar, Sanjit and Batinic-Haberle, Ines and Clair, Daret St
Free Radical Biology and Medicine, ISSN 0891-5849, 11/2018, Volume 128, pp. S82 - S83
Medical colleges | Nitration | Glucose metabolism | Superoxide
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14. Full Text 181 - Manganese superoxide dismutase regulates mTORC2 mediated UVB-induced mitophagy
by Dhar, Sanjit and Batinic-Haberle, Ines and Clair, Daret St
Free Radical Biology and Medicine, ISSN 0891-5849, 11/2018, Volume 128, pp. S82 - S83
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15. Full Text Differences in Reperfusion-Induced Mitochondrial Oxidative Stress and Cell Death Between Hippocampal CA1 and CA3 Subfields Are Due to the Mitochondrial Thioredoxin System
by Yin, Bocheng and Barrionuevo, Germán and Batinic-Haberle, Ines and Sandberg, Mats and Weber, Stephen G and Sahlgrenska akademin and Göteborgs universitet and Gothenburg University and Institutionen för biomedicin, avdelningen för medicinsk kemi och cellbiologi and Institute of Biomedicine, Department of Medical Biochemistry and Cell Biology and Sahlgrenska Academy
Antioxidants & Redox Signaling, ISSN 1523-0864, 09/2017, Volume 27, Issue 9, pp. 534 - 549
Aims: The susceptibility of CA1 over CA3 to damage from cerebral ischemia may be related to the differences in reactive oxygen species (ROS) production/removal... 
Original Research Communications | OGD-RP | hippocampal pyramidal cell | thioredoxin | glutathione | mitochondria | ROS | SELECTIVE NEURONAL VULNERABILITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | OXYGEN SPECIES GENERATION | GLUTAREDOXIN SYSTEMS | SLICE CULTURES | REDOX SYSTEMS | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | CENTRAL-NERVOUS-SYSTEM | HYDROGEN-PEROXIDE | GAMMA OSCILLATIONS | CEREBRAL-ISCHEMIA | Reactive Oxygen Species - metabolism | Oxidative Stress | Brain Ischemia - metabolism | Rats | Male | Mitochondria - metabolism | Brain Ischemia - etiology | Hippocampus - cytology | Hippocampus - metabolism | Animals | Thioredoxin-Disulfide Reductase - metabolism | Glutathione - analysis | Hydrogen Peroxide - analysis | Cell Death | Reperfusion Injury - complications | Reperfusion Injury - metabolism | Organ Culture Techniques | Oxidative stress | Research | Thioredoxin | Reperfusion injury | Immunohistochemistry | Reactive oxygen species | Innovations | Glucose | Western blotting | Mitochondria | Reperfusion | Ischemia | Peroxide | Oxidation | Inhibition | Probes | Glutathione | Deprivation | Oxygen | Mortality | Cultures | Superoxide | Real time | Cell death | Pyramidal cells | Hippocampus | Apoptosis | Reductase | Neurosciences | Neurovetenskaper
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16. Full Text Thiol regulation by Mn porphyrins, commonly known as SOD mimics
by Batinic-Haberle, Ines and Tome, Margaret E
Redox biology, ISSN 2213-2317, 02/2019, p. 101139
Superoxide dismutases play an important role in human health and disease. Three decades of effort have gone into synthesizing SOD mimics for clinical use. The... 
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17. Full Text Manganese Porphyrin, MnTE-2-PyP5+, Acts as a Pro-Oxidant to Potentiate Glucocorticoid-Induced Apoptosis in Lymphoma Cells
by Jaramillo, Melba C and Briehl, Margaret M and Crapo, James D and Batinic-Haberle, Ines and Tome, Margaret E
Free Radical Biology and Medicine, ISSN 0891-5849, 04/2012, Volume 52, Issue 8, pp. 1272 - 1284
Using current chemotherapy protocols, over 55% of lymphoma patients fail treatment. Novel agents are needed to improve lymphoma survival. The manganese... 
NF-κB | Dexamethasone | Hydrogen peroxide | Glutathionylation | MnTE-2-PyP5 | Lymphoma | Glutathione | MULTIPLE-MYELOMA | OXIDATIVE STRESS | SUPEROXIDE | DEXAMETHASONE-INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | P50 SUBUNIT | DNA-BINDING ACTIVITY | IN-VITRO | NF-kappa B | INHIBITION | PROTEIN GLUTATHIONYLATION | ENDOCRINOLOGY & METABOLISM | NF-KAPPA-B | Reactive Oxygen Species - metabolism | Oxidation-Reduction | Apoptosis - drug effects | Glutathione - metabolism | Reactive Oxygen Species - pharmacology | Metalloporphyrins - pharmacology | Hydrogen Peroxide - metabolism | Thymus Neoplasms - metabolism | Drug Synergism | Lymphoma - metabolism | Animals | Dexamethasone - pharmacology | Thymus Neoplasms - pathology | Cell Line, Tumor | Lymphoma - pathology | Mice | dexamethasone | glutathione | glutathionylation | lymphoma | hydrogen peroxide
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18. Full Text Complex Chemistry and Biology of Redox-Active Compounds, Commonly Known as SOD Mimics, Affect Their Therapeutic Effects
by Batinic-Haberle, Ines and Spasojevic, Ivan
Antioxidants & Redox Signaling, ISSN 1523-0864, 05/2014, Volume 20, Issue 15, pp. 2323 - 2325
This Editorial has the intention to stress the complex chemistry and biology of redox-active compounds, regarded as SOD mimics. It further aims to caution the... 
Forum Editorial | CELLS | ANTIOXIDANTS | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOCRINOLOGY & METABOLISM | Molecular Mimicry | Animals | Superoxide Dismutase - pharmacology | Signal Transduction - drug effects | Humans | Oxidation-Reduction - drug effects | Superoxide Dismutase - metabolism | Superoxide Dismutase - therapeutic use | Therapeutics | Oxidation-reduction reaction | Research | Properties | Chemical compounds | Homeopathy | Materia medica and therapeutics
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19. Full Text SOD Therapeutics: Latest Insights into Their Structure-Activity Relationships and Impact on the Cellular Redox-Based Signaling Pathways
by Batinic-Haberle, Ines and Tovmasyan, Artak and Roberts, Emily R. H and Vujaskovic, Zeljko and Leong, Kam W and Spasojevic, Ivan
Antioxidants & Redox Signaling, ISSN 1523-0864, 05/2014, Volume 20, Issue 15, pp. 2372 - 2415
Significance: Superoxide dismutase (SOD) enzymes are indispensable and ubiquitous antioxidant defenses maintaining the steady-state levels of O 2 · − ; no... 
Forum Review Articles | SPINAL CORD INJURY | AIDED-DESIGN CAD | OXIDATIVE STRESS | MANGANESE SUPEROXIDE-DISMUTASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | DEFICIENT ESCHERICHIA-COLI | ENDOCRINOLOGY & METABOLISM | GLUCOCORTICOID-INDUCED APOPTOSIS | PEROXYNITRITE DECOMPOSITION CATALYST | IN-VIVO RADIOPROTECTION | ASCORBATE-INDUCED CYTOTOXICITY | NF-KAPPA-B | Superoxide Dismutase - pharmacology | Metalloporphyrins - metabolism | Humans | Structure-Activity Relationship | Metalloporphyrins - pharmacology | Superoxide Dismutase - therapeutic use | Superoxide Dismutase - chemistry | Molecular Mimicry | Animals | Signal Transduction - drug effects | Drug Design | Metalloporphyrins - chemistry | Oxidation-Reduction - drug effects | Superoxide Dismutase - metabolism | Oxidative stress | Usage | Therapeutics | Superoxide dismutase | Research | Homeopathy | Materia medica and therapeutics | Forum Review
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