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Language in India, ISSN 1930-2940, 03/2015, Volume 15, Issue 3, pp. 85 - 95
The current study is conducted for presenting a critical discourse analysis of the mob's language that assembled to record protest against gas and electricity... 
Journal Article
Lancet, The, ISSN 0140-6736, 2009, Volume 373, Issue 9663, pp. 567 - 574
Journal Article
Journal Article
Journal Article
Neuro-Oncology, ISSN 1522-8517, 11/2019, Volume 21, Issue Supplement_6, pp. vi44 - vi44
Abstract Low-grade gliomas (LGGs) present a high incidence of epilepsy, infiltrative growth, resistance to therapy, and high risk of transforming into... 
Journal Article
Glia, ISSN 0894-1491, 01/2018, Volume 66, Issue 1, pp. 126 - 144
Stimulation of Na+/H+ exchanger isoform 1 (NHE1) in astrocytes causes ionic dysregulation under ischemic conditions. In this study, we created a Nhe1flox/flox... 
MMP | blood–brain barrier | gliosis | cerebral edema | astrocyte end‐feet | neurovascular unit | astrocyte end-feet | blood-brain barrier | NEUROSCIENCES | Glial Fibrillary Acidic Protein - genetics | Cerebrovascular Circulation - physiology | Male | Cerebrovascular Circulation - genetics | Glial Fibrillary Acidic Protein - metabolism | Blood-Brain Barrier - ultrastructure | Gliosis - pathology | Matrix Metalloproteinase 9 - metabolism | Reperfusion | Infarction, Middle Cerebral Artery - complications | Gliosis - etiology | Disease Models, Animal | Microscopy, Electron, Transmission | Gliosis - genetics | Brain Infarction - diagnosis | Gene Expression Regulation - genetics | Gliosis - metabolism | Mice, Inbred C57BL | Sodium-Hydrogen Exchanger 1 - genetics | Mice, Transgenic | Infarction, Middle Cerebral Artery - pathology | Astrocytes - ultrastructure | Blood-Brain Barrier - pathology | Brain Infarction - etiology | Motor Activity - genetics | Sodium-Hydrogen Exchanger 1 - deficiency | Animals | Neurologic Examination | Mice | Brain Infarction - genetics | Astrocytes - metabolism | Stroke (Disease) | Brain damage | Ischemia | Cytochemistry | Proteases | Analysis | Occlusion | Matrix metalloproteinases | Hydrogen | Recombinase | Gene deletion | Corn oil | Clonal deletion | Blood-brain barrier | Nhe1 gene | Cerebral blood flow | Rodents | Deletion | Cerebral infarction | Edema | Stroke | Tight junctions | Astrocytes | Na+/H+-exchanging ATPase | Inflammation | Tamoxifen | Ablation | Endothelial cells | Flox | Gelatinase B | Blood flow | Neurological diseases | Injury prevention | Gliosis | Infarction | Brain injury | Hypertrophy
Journal Article
Glia, ISSN 0894-1491, 11/2018, Volume 66, Issue 11, pp. 2279 - 2298
Na+/H+ exchanger (NHE1) activation is required for multiple microglial functions. We investigated effects of selective deletion of microglial Nhe1 in... 
phagocytosis | macrophages | inflammation | microglia | white matter tissue repair | SURVIVAL | BRAIN-INJURY | FOCAL CEREBRAL-ISCHEMIA | RECEPTOR | CNS | NEUROSCIENCES | NEURONAL DEATH | GROWTH | MICE | EXPRESSION | Microglia - metabolism | Somatosensory Disorders - etiology | Brain - diagnostic imaging | Recovery of Function - drug effects | Male | CX3C Chemokine Receptor 1 - metabolism | CX3C Chemokine Receptor 1 - genetics | Infarction, Middle Cerebral Artery - complications | Recovery of Function - physiology | Female | Demyelinating Diseases - etiology | Infarction, Middle Cerebral Artery - drug therapy | Microfilament Proteins - metabolism | Infarction, Middle Cerebral Artery - diagnostic imaging | Demyelinating Diseases - drug therapy | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Macrophages - pathology | Microglia - drug effects | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Sodium-Hydrogen Exchanger 1 - genetics | Mice, Transgenic | Nerve Tissue Proteins - genetics | White Matter - pathology | Brain - drug effects | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Macrophages - metabolism | White Matter - diagnostic imaging | Animals | Tamoxifen - pharmacology | Sodium-Hydrogen Exchanger 1 - metabolism | Mice | Stroke (Disease) | Neurons | Analysis | Brain damage | Bone morphogenetic proteins | Inflammation | Macrophages | Transforming growth factors | Brain | CD86 antigen | Hydrogen | CX3CR1 protein | Activation | Recovery | Proteins | Cell activation | Corn oil | Clonal deletion | Ischemia | Demyelination | Rodents | Animal tissues | Deletion | Lesions | Repair | Stroke | CD11b antigen | Cell survival | Therapeutic applications | Astrocytes | Na+/H+-exchanging ATPase | Glial fibrillary acidic protein | Tamoxifen | Substantia alba | Survival | Microglia | Myelination | Brain injury | Ca2+/calmodulin-dependent protein kinase II
Journal Article
Journal Article
JOURNAL OF EXPERIMENTAL & CLINICAL CANCER RESEARCH, ISSN 1756-9966, 10/2018, Volume 37, Issue 1, pp. 255 - 16
Journal Article