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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2011, Volume 108, Issue 36, pp. 14944 - 14949
The bacterial pathogen Helicobacter pylori chronically infects the human gastric mucosa and is the leading risk factor for the development of gastric cancer.... 
Helicobacter pylori | Epithelial cells | DNA damage | DNA | Cell nucleus | Cell lines | Bacteria | Cultured cells | Infections | Mice | Chromosome breaks | DNA damage signaling | Gastric tumorigenesis | Genomic instability | CYTOLETHAL DISTENDING TOXIN | MULTIDISCIPLINARY SCIENCES | RISK | MISMATCH REPAIR | COLONIZATION | GENETIC INSTABILITY | EPITHELIAL-CELLS | chromosome breaks | GASTRIC-CANCER | INFLAMMATION | INFECTION | MICE | genomic instability | gastric tumorigenesis | Phosphorylation | Epithelial Cells - metabolism | Humans | Stomach Neoplasms - metabolism | Helicobacter Infections - complications | Stomach Neoplasms - pathology | Intracellular Signaling Peptides and Proteins - metabolism | Antigens, Bacterial - genetics | DNA Breaks, Double-Stranded | Helicobacter Infections - pathology | DNA-Binding Proteins - metabolism | Tumor Suppressor Proteins - genetics | Trans-Activators - genetics | Cell Cycle Proteins - genetics | Helicobacter Infections - metabolism | Nuclear Proteins - genetics | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Stomach Neoplasms - genetics | Tumor Suppressor Proteins - metabolism | Chromosomal Proteins, Non-Histone - metabolism | Bacterial Proteins - genetics | Cell Cycle Proteins - metabolism | Protein-Serine-Threonine Kinases - genetics | Genomic Islands | Epithelial Cells - pathology | Nuclear Proteins - metabolism | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | Chromosomal Proteins, Non-Histone - genetics | Bacterial Adhesion | Stomach Neoplasms - microbiology | Animals | Histones - genetics | Helicobacter pylori - metabolism | Epithelial Cells - microbiology | Chromosome Aberrations | Cell Line, Tumor | Bacterial Proteins - metabolism | Trans-Activators - metabolism | Histones - metabolism | Tumor Suppressor p53-Binding Protein 1 | Antigens, Bacterial - metabolism | Carcinogens | Cocarcinogens | Physiological aspects | Genetic aspects | Research | Health aspects | Cells | Pathogens | Gram-negative bacteria | Rodents | Index Medicus | Adhesins | Histone H2A | Mesenchyme | Gastric mucosa | Carcinogenesis | Risk factors | Mammalian cells | Metaphase | genomics | Blood groups | Chromosomes | Gastric cancer | Gel electrophoresis | virulence factors | Infection | Cytotoxins | gamma -Glutamyltransferase | Molecular modelling | Microscopy | pathogenicity islands | Ataxia telangiectasia mutated protein | Chromosome aberrations | Biological Sciences
Journal Article
Immunology, ISSN 0019-2805, 04/2005, Volume 114, Issue 4, pp. 531 - 541
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, pp. e73160 - e73160
Helicobacter bilis (H. bilis) infection is associated with cases of inflammatory bowel Disease, thyphlocolitis, hepatitis and cholecystitis. However, little is... 
TRANSCRIPTION FACTORS | MULTIDISCIPLINARY SCIENCES | ULCERATIVE-COLITIS | ENTEROHEPATIC HELICOBACTER | NECROSIS-FACTOR-ALPHA | GLUTATHIONE LEVELS | NF-KAPPA-B | LUNG INFLAMMATION | GUT IN-VITRO | BOWEL-DISEASE | GENE-TRANSCRIPTION | Colonic Neoplasms - genetics | Helicobacter - enzymology | Intestinal Mucosa - metabolism | Reactive Oxygen Species - metabolism | Oxidative Stress | Signal Transduction | Interleukin-8 - biosynthesis | Humans | Transcriptional Activation | Glutamine - metabolism | NF-kappa B - metabolism | gamma-Glutamyltransferase - metabolism | Intestinal Mucosa - microbiology | Helicobacter Infections - genetics | Transcription Factor AP-1 - metabolism | Colon - metabolism | Colonic Neoplasms - metabolism | Cyclic AMP Response Element-Binding Protein - metabolism | Cell Line, Tumor | Helicobacter Infections - metabolism | Colon - microbiology | Colonic Neoplasms - microbiology | Infection | Oxidative stress | Colon cancer | DNA binding proteins | Virulence (Microbiology) | Health aspects | Cholecystitis | Glutamine | Reactive oxygen species | Transcription factors | Inflammatory bowel diseases | Disease | Epithelial cells | Virulence | DNA damage | Colorectal cancer | Infections | Kinases | Inflammatory diseases | Hepatitis | Immunology | Intestine | Cascades | Bacteria | Colon | Interleukin 8 | Deoxyribonucleic acid--DNA | Glutathione | Biodegradation | Stresses | NF-κB protein | Deprivation | Cytokines | Activator protein 1 | Inflammation | Cyclic AMP response element-binding protein | Gene expression | Chronic fatigue syndrome | Stress | Inflammatory bowel disease | Signaling | Molecular modelling | Nitric oxide | Viability | Hygiene | Cancer | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2010, Volume 5, Issue 3, pp. e9500 - e9500
Amongst the most severe clinical outcomes of life-long infections with Helicobacter pylori is the development of peptic ulcers and gastric adenocarcinoma -... 
CYCLIC ADENOSINE-MONOPHOSPHATE | ACTIVATION | GAMMA-GLUTAMYL-TRANSPEPTIDASE | DENDRITIC CELLS | FOXP3 GENE-EXPRESSION | BIOLOGY | IV SECRETION | DEPENDENT PROTEIN-KINASE | VACUOLATING TOXIN | INFLAMMATORY RESPONSE | HUMAN VOLUNTEERS | T-Lymphocytes - microbiology | Jurkat Cells | Humans | MicroRNAs - biosynthesis | Animals | Helicobacter pylori - metabolism | T-Lymphocytes - metabolism | Forkhead Transcription Factors - metabolism | Models, Biological | Lipopolysaccharides - chemistry | Mice | MicroRNAs - genetics | Mutation | 3' Untranslated Regions | Gene Expression Regulation, Bacterial | Cyclic AMP - metabolism | Infection | MicroRNA | Helicobacter pylori | Peptic ulcer | Helicobacter infections | Cyclic adenylic acid | T cells | Health aspects | Mitogens | Stomach cancer | Antiulcer drugs | Adenocarcinoma | Protein kinase A | Regulators | Toxin A | Peptides | g-Glutamyltransferase | Laboratories | Lymphocytes T | Infections | Kinases | Positive feedback | Lipopolysaccharides | Proteins | Adenylate cyclase | Carcinogens | Peptic ulcers | Lymphocytes | Gastroenterology | Bacteria | Foxp3 protein | Immune system | Antigens | Adenosine | Adenosine monophosphate | AMP | Immunoregulation | RNA-mediated interference | MiRNA | Cyclic AMP | Mutants | Ribonucleic acids | Biopsy | Protein synthesis | MicroRNAs | Ulcers | Lymphomas | Molecular biology | Viral infections | Cancer | Apoptosis | Index Medicus
Journal Article
CARCINOGENESIS, ISSN 0143-3334, 04/2019, Volume 40, Issue 4, pp. 551 - 559
Ring finger protein 43 (RNF43) is an E3 ubiquitin ligase that has been described to be frequently mutated in gastrointestinal cancers. RNF43 downregulation was... 
SUPPRESSOR RNF43 | GENE | ONCOLOGY | PATHWAY | INFLAMMATION | RECURRENT MUTATIONS | CANCER | TUMORIGENESIS
Journal Article
Journal Article
Journal Article
Science signaling, ISSN 1937-9145, 2015, Volume 8, Issue 393, pp. ra90 - ra90
Journal Article
Jundishapur Journal of Microbiology, ISSN 2008-3645, 11/2015, Volume 8, Issue 11, pp. e25425 - e25425
Background: Helicobacter pylori infection and related diseases outcome are mediated by a complex interplay between bacterial, host and environmental factors.... 
Peptic ulcer | Helicobacter pylori | cagA | vacA | ICEA GENOTYPES | MICROBIOLOGY | PREVALENCE | Peptic Ulcer | VACA GENOTYPES | PATHOGENESIS | PEPTIC-ULCERATION | DISEASES | INFECTION | ASSOCIATION | EPIDEMIOLOGY
Journal Article
Genesis, ISSN 1526-954X, 11/2013, Volume 51, Issue 11, pp. 793 - 802
The HMG-box transcription factor Sox17 is essential for endoderm formation, vascular development, and definitive hematopoiesis. To investigate the fate of... 
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2011, Volume 127, Issue 5, pp. 1187 - 1194.e7
Journal Article
Mediators of Inflammation, ISSN 0962-9351, 2014, Volume 2014, pp. 1 - 9
Journal Article
Experimental Dermatology, ISSN 0906-6705, 01/2011, Volume 20, Issue 1, pp. 48 - 52
Primary human keratinocytes and immortalized HaCaT cells were analysed for their capacity to bind purified staphylococcal protein A (SpA). Co‐incubation with... 
Keratinocytes | TNF RI | Protein A | Staphylococcus aureus | CONTRIBUTE | NECROSIS-FACTOR-ALPHA | EPIDERMAL-KERATINOCYTES | DERMATOLOGY | ATOPIC-DERMATITIS | SUPERANTIGENS | DISEASE | SKIN | UP-REGULATION | EXPRESSION | AUREUS COLONIZATION | Interleukin-8 - genetics | Dermatitis, Atopic - genetics | Staphylococcal Protein A - biosynthesis | Humans | Dermatitis, Atopic - microbiology | Receptors, Tumor Necrosis Factor, Type I - metabolism | RNA, Messenger - metabolism | Transcription Factor AP-1 - metabolism | Cyclooxygenase 2 - genetics | Base Sequence | Inflammation Mediators - metabolism | Transcription, Genetic | Active Transport, Cell Nucleus | Keratinocytes - microbiology | Cell Line | Staphylococcus aureus - isolation & purification | RNA, Messenger - genetics | Cells, Cultured | Receptors, Tumor Necrosis Factor, Type I - genetics | Staphylococcus aureus - pathogenicity | Keratinocytes - immunology | Transcription Factor RelA - metabolism | Keratinocytes - metabolism | Protein Binding | Staphylococcus aureus - immunology | In Vitro Techniques | Staphylococcal Protein A - metabolism | Dermatitis, Atopic - metabolism | Allergy | Messenger RNA | Tumor necrosis factor | Genetic research | Genetic aspects | Chemical properties | Allergic reaction | Phosphotransferases | Protein binding | Index Medicus | Indexing in process
Journal Article