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by Garassino, Marina Chiara and Cho, Byoung-Chul and Kim, Dong-Wan and Kim, Joo-Hang and Kim, Sang-We and Kim, Young-Chul and Mazières, Julien and Vansteenkiste, Johan and Lena, Hervé and Corral Jaime, Jesus and Gray, Jhanelle E and Powderly, John and Chouaid, Christos and Bidoli, Paolo and Wheatley-Price, Paul and Park, Keunchil and Soo, Ross A and Huang, Yifan and Wadsworth, Catherine and Dennis, Phillip A and Rizvi, Naiyer A and Paz-Ares Rodriguez, Luis and Novello, Silvia and Hiret, Sandrine and Schmid, Peter and Laack, Eckart and Califano, Raffaele and Maemondo, Makoto and Chaft, Jamie and Vicente Baz, David and Berghmans, Thierry and Surmont, Veerle and Reck, Martin and Han, Ji-Youn and Holgado Martin, Esther and Belda Iniesta, Cristobal and Oe, Yuichiro and Chella, Antonio and Chopra, Akhil and Robinet, Gilles and Soto Parra, Hector and Thomas, Michael and Cheema, Parneet and Katakami, Nobuyuki and Su, Wu-Chou and Wolf, Juergen and Lee, Jong-Seok and Saka, Hideo and Milella, Michele and Ramos Garcia, Inmaculada and Sibille, Anne and Yokoi, Takashi and Kang, Eun Joo and Atagi, Shinji and Spaeth-Schwalbe, Ernst and Nishio, Makoto and Imamura, Fumio and Gabrail, Nashat and Veillon, Remi and Derijcke, Sofie and Maeda, Tadashi and Zylla, Dylan and Kubiak, Kendra and Santoro, Armando and Uy, Ma. Noemi and Lucien Geater, Sarayut and Italiano, Antoine and Kowalski, Dariusz and Barlesi, Fabrice and Chen, Yuh-Min and Spigel, David and Chewaskulyong, Busyamas and Garcia Gomez, Ramon and Alvarez Alvarez, Rosa and Yang, Chih-Hsin and Hsia, Te-Chun and Denis, Fabrice and Sakai, Hiroshi and Vincent, Mark and Goto, Koichi and Bosch-Barrera, Joaquim and Weiss, Glen and Canon, Jean-Luc and Scholz, Christian and Aglietta, Massimo and Kemmotsu, Hirotsugu and Azuma, Koichi and Bradbury, Penelope and Feld, Ronald and Chachoua, Abraham and Jassem, Jacek and Juergens, Rosalyn and Palmero Sanchez, Ramon and Malcolm, Albert and Vrindavanam, Nandagopal and Kubota, Kaoru and Waller, Cornelius and Waterhouse, David and Coudert, Bruno and Mark, Zsuzsanna and ... and ATLANTIC Investigators
The Lancet Oncology, ISSN 1470-2045, 04/2018, Volume 19, Issue 4, pp. 521 - 536
Immune checkpoint inhibitors are a new standard of care for patients with advanced non-small-cell lung cancer (NSCLC) without tyrosine kinase or anaplastic... 
MULTICENTER | METAANALYSIS | NSCLC | ONCOLOGY | RANDOMIZED CONTROLLED-TRIAL | ATEZOLIZUMAB | IMMUNE CHECKPOINT | NIVOLUMAB | MEDI4736 | CLINICAL ACTIVITY | DOCETAXEL | Lung cancer, Non-small cell | Care and treatment
Journal Article
PLoS ONE, ISSN 1932-6203, 2009, Volume 4, Issue 12, p. e8199
Journal Article
Frontiers in Physiology, ISSN 1664-042X, 06/2018, Volume 9, p. 651
Extracellular vesicles (EVs) - including exosomes, microvesicles and apoptotic bodies - have received much scientific attention last decade as mediators of a... 
Extracellular vesicles | Extracellular matrix | Cell function | Microenvironment | Exosomes | cell function | PROGENITOR CELLS | PHYSIOLOGY | extracellular vesicles | INTERCELLULAR TRANSFER | ANGIOGENESIS | TUMOR-CELLS | MESENCHYMAL STEM-CELLS | microenvironment | IN-VITRO | MEMBRANE-VESICLES | exosomes | ENDOTHELIAL-CELLS | extracellular matrix | T-CELLS | Neovascularization | Research | Analysis
Journal Article
Cancer Treatment Reviews, ISSN 0305-7372, 2004, Volume 30, Issue 2, pp. 193 - 204
Phosphatidylinositol-3 kinases, PI3Ks, constitute a lipid kinase family characterized by their ability to phosphorylate inositol ring 3′-OH group in inositol... 
Resistance | Chemotherapy | New cancer drugs | Akt | Proliferation | P13K | Signalling | Apoptosis
Journal Article
Journal of Neuro-Oncology, ISSN 0167-594X, 1/2017, Volume 131, Issue 1, pp. 105 - 115
Journal Article
Cancer Medicine, ISSN 2045-7634, 10/2014, Volume 3, Issue 5, pp. 1099 - 1111
Lung cancer is the top cause of cancer‐related deceases. One of the reasons is the development of resistance to the chemotherapy treatment. In particular,... 
cancer‐initiating cells | lung cancer | NSCLC | Cancer stem cells | cisplatin resistance | Cisplatin resistance | Cancer-initiating cells | Lung cancer | ACTIVATION | METASTASIS | ANGIOGENESIS | MECHANISM | ADENOCARCINOMA CELLS | ADRENOMEDULLIN | TUMOR-INITIATING CELLS | cancer-initiating cells | ONCOLOGY | ARYL-HYDROCARBON RECEPTOR | GENES | PROGRESSION | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Tumor Stem Cell Assay | Carcinoma, Non-Small-Cell Lung - genetics | Neoplasm Invasiveness | Neoplastic Stem Cells - drug effects | Humans | Neovascularization, Pathologic | Drug Resistance, Neoplasm | Lung Neoplasms - pathology | Gene Expression Profiling | Cisplatin - pharmacology | Animals | Heterografts | Neoplastic Stem Cells - metabolism | Cell Transformation, Neoplastic | Cell Line, Tumor | Female | Antineoplastic Agents - pharmacology | Mice | Disease Models, Animal | Antimitotic agents | Chemotherapy | Analysis | Stem cells | Development and progression | Lung cancer, Non-small cell | Antineoplastic agents | Gene expression | Cancer | Adherent cells | Cell culture | Media (culture) | EGR-1 protein | Immunodeficiency | Stem cell transplantation | Metastasis | Tumor cell lines | Cell differentiation | Drug resistance | Cancer therapies | Cisplatin | Metastases | Cell adhesion & migration | Latency | Angiogenesis | Cell lines | Cyclooxygenase-2 | Growth factors | Tumors | Cancer Biology
Journal Article
Oncogene, ISSN 0950-9232, 03/2013, Volume 32, Issue 10, pp. 1274 - 1283
Although many cancers initially respond to cisplatin (CDDP)-based chemotherapy, resistance frequently develops. Insulin-like growth factor-binding protein-3... 
IGFBP-3 | NSCLC | cisplatin resistance | hypermethylation | IGFIR | OXIDATIVE STRESS | GEFITINIB | BIOCHEMISTRY & MOLECULAR BIOLOGY | DRUG-RESISTANCE | CHEMOTHERAPY | CELL BIOLOGY | GROWTH-FACTOR RECEPTOR | INDUCED DNA HYPERMETHYLATION | GENE | ONCOLOGY | MUTATION | GENETICS & HEREDITY | TUMOR-GROWTH | EXPRESSION | Insulin-Like Growth Factor Binding Protein 3 - genetics | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Receptor, IGF Type 1 - metabolism | Phosphorylation | Humans | Lung Neoplasms - metabolism | Drug Resistance, Neoplasm | Lung Neoplasms - pathology | Insulin-Like Growth Factor Binding Protein 3 - deficiency | Proto-Oncogene Proteins c-akt - genetics | DNA Methylation | Receptor, Epidermal Growth Factor - metabolism | Transfection | Insulin-Like Growth Factor Binding Protein 3 - metabolism | Antineoplastic Agents - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Promoter Regions, Genetic | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Cisplatin - pharmacology | Receptor, IGF Type 1 - genetics | Insulin-Like Growth Factor Binding Protein 3 - biosynthesis | Cell Line, Tumor | Carcinoma, Non-Small-Cell Lung - drug therapy | Physiological aspects | Dosage and administration | Research | Growth factor receptors | Lung cancer, Non-small cell | Drug therapy | Drug resistance | Binding proteins | Health aspects | Cisplatin | Proteins | Signal transduction | Oncology | Epidermal growth factor | Lung cancer | Translation | Epidermal growth factor receptors | Tumor cells | biomarkers | Non-small cell lung carcinoma | AKT protein | Tumor cell lines | 1-Phosphatidylinositol 3-kinase | Chemotherapy | Derepression | Insulin-like growth factor I receptors | DNA methylation | Insulin-like growth factor-binding protein 3
Journal Article
Cancer Research, ISSN 0008-5472, 08/2015, Volume 75, Issue 15 Supplement, pp. 2060 - 2060
Journal Article
The European respiratory journal, ISSN 0903-1936, 06/2017, Volume 49, Issue 6, pp. 1602456 - 1602456
Journal Article
Journal Article
Oncogene, ISSN 0950-9232, 02/2011, Volume 30, Issue 6, pp. 668 - 678
Journal Article