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European Journal of Immunology, ISSN 0014-2980, 05/2015, Volume 45, Issue 5, pp. 1377 - 1389
Journal Article
Immunity, ISSN 1074-7613, 06/2016, Volume 44, Issue 6, pp. 1350 - 1364
T follicular helper (Tfh) cells promote affinity maturation of B cells in germinal centers (GCs), whereas T follicular regulatory (Tfr) cells limit the GC... 
T follicular helper (Tfh) cells | calcium | STIM1 | STIM2 | T follicular regulatory (Tfr) cells | SOCE | CRAC channel | NFAT | Calcium | HELPER-CELL | ACTIVATION | TRANSCRIPTION FACTOR IRF4 | ION CHANNELS | DIFFERENTIATION | IMMUNOLOGY | CALCIUM SENSORS STIM1 | EXPRESSION | DEFICIENCY | IMMUNODEFICIENCY | Immunoglobulins | Transcription factors | Lymphocytes | Cloning | Data processing | T cell receptors | Mutation | Localization | Patients
Journal Article
European Journal of Immunology, ISSN 0014-2980, 02/2015, Volume 45, Issue 2, pp. 480 - 491
Anergic T cells can survive for long time periods passively in a hyporesponsive state without obvious active functions. Thus, the immunological reason for... 
CD28 | CTLA‐4 | Dendritic cell | Treg cell | T‐cell anergy | CTLA-4 | T-cell anergy | ENTEROTOXIN-B | BONE-MARROW | CLONAL ANERGY | MONOCLONAL-ANTIBODY | INDUCTION | IMMUNOLOGY | IN-VIVO | NUCLEAR FACTOR | TRANSCRIPTION FACTOR | TRANSGENIC MICE | SELF-TOLERANCE | Forkhead Transcription Factors - immunology | T-Lymphocytes, Regulatory - metabolism | Coculture Techniques | Dendritic Cells - immunology | Early Growth Response Protein 2 - genetics | Clonal Anergy | T-Lymphocytes, Regulatory - immunology | Interleukin-2 - immunology | Interleukin-2 Receptor alpha Subunit - genetics | NFATC Transcription Factors - immunology | CD28 Antigens - genetics | T-Lymphocytes, Regulatory - cytology | Dendritic Cells - drug effects | Cell Differentiation | Early Growth Response Protein 2 - immunology | Dendritic Cells - metabolism | Interleukin-2 Receptor alpha Subunit - immunology | Signal Transduction | Gene Expression Regulation - immunology | Lymphocyte Activation | Mice, Inbred C57BL | Cell Communication | CTLA-4 Antigen - genetics | CD28 Antigens - immunology | CTLA-4 Antigen - immunology | Forkhead Transcription Factors - genetics | Interleukin-2 - genetics | Transforming Growth Factor beta - pharmacology | T-Lymphocytes, Regulatory - drug effects | Animals | Interleukin-10 - genetics | Dendritic Cells - cytology | Mice | Mice, Inbred BALB C | Interleukin-10 - immunology | NFATC Transcription Factors - genetics | Bone morphogenetic proteins | Peptides | Dendritic cells | T cells | Transforming growth factors
Journal Article
Journal Article
European Journal of Immunology, ISSN 0014-2980, 05/2011, Volume 41, Issue 5, pp. 1420 - 1434
Tolerance to self‐antigens expressed in peripheral organs is maintained by CD4+ CD25+ Foxp3+ Treg cells, which are generated as a result of thymic selection or... 
Tolerance | Treg cells | Immune regulation | Cell trafficking | DCs | TGF-BETA | LYMPHOID ORGAN DEVELOPMENT | RETINOIC-ACID | MULTIORGAN INFLAMMATION | DEFICIENT MICE | IMMUNOLOGY | LANGERHANS CELLS | TARGETED DISRUPTION | IN-VIVO | SKIN | T-Lymphocytes, Regulatory - metabolism | NF-kappa B - immunology | Transcription Factor RelB - analysis | T-Lymphocytes, Regulatory - immunology | Antigens, CD - analysis | NF-kappa B - deficiency | Forkhead Transcription Factors - metabolism | Langerhans Cells - immunology | Lectins, C-Type - analysis | Mannose-Binding Lectins - analysis | Cell Differentiation | Skin - immunology | Major Histocompatibility Complex | Mice, Inbred C57BL | Self Tolerance | Integrin alpha Chains - analysis | Lymph Nodes - metabolism | Immunophenotyping | Mice, Transgenic | Lymph Nodes - immunology | Forkhead Transcription Factors - genetics | Microscopy, Confocal | Animals | CD4 Antigens - analysis | Fluorescent Antibody Technique | Interleukin-2 Receptor alpha Subunit - analysis | Mice | Langerhans Cells - metabolism | Receptors, CCR7 - analysis | Transforming Growth Factor beta - metabolism | Antigens, Surface - analysis | Cell Movement | Langerhans Cells | Transcription Factor RelB | Life Sciences | NF-kappa B | Immunology | T-Lymphocytes, Regulatory | Transforming Growth Factor beta | Interleukin-2 Receptor alpha Subunit | Mannose-Binding Lectins | Antigens, CD4 | Lectins, C-Type | Receptors, CCR7 | Lymph Nodes | Skin | Antigens, Surface | Integrin alpha Chains | Forkhead Transcription Factors | Antigens, CD
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2012, Volume 109, Issue 40, pp. 16258 - 16263
Journal Article
European Journal of Immunology, ISSN 0014-2980, 05/2015, Volume 45, Issue 5, pp. 1377 - 1389
Journal Article
Blood, ISSN 0006-4971, 12/2016, Volume 128, Issue 22, pp. 809 - 809
Abstract Allogenic hematopoietic cell transplantation (allo-HCT) is an established therapy for the treatment of malignant diseases such as leukemia or... 
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2012, Volume 7, Issue 12, p. e52208
Natalizumab is a recombinant monoclonal antibody raised against integrin alpha-4 (CD49d). It is approved for the treatment of patients with multiple sclerosis... 
ANTIBODIES | ALPHA-4-BETA-1 INTEGRIN | RESPONSES | ACTIVATION | PROGRESSIVE MULTIFOCAL LEUKOENCEPHALOPATHY | MULTIDISCIPLINARY SCIENCES | REGULATORY T-CELLS | UPDATE | FOCAL ADHESION KINASE | EXPRESSION | LYMPHOCYTES | Interleukin-2 - metabolism | Leukoencephalopathy, Progressive Multifocal - immunology | Antibodies, Monoclonal, Humanized - adverse effects | Antibodies, Monoclonal, Humanized - therapeutic use | Natalizumab | Humans | Cells, Cultured | Interferon-gamma - metabolism | Integrin alpha4 - immunology | CD4-Positive T-Lymphocytes - immunology | Interleukin-17 - metabolism | Leukoencephalopathy, Progressive Multifocal - chemically induced | Signal Transduction - drug effects | T-Lymphocytes - drug effects | Multiple Sclerosis - immunology | T-Lymphocytes - immunology | Phosphorylation - drug effects | CD4-Positive T-Lymphocytes - drug effects | Mitogen-Activated Protein Kinases - metabolism | Multiple Sclerosis - drug therapy | Multiple Sclerosis - metabolism | Medical research | Multiple sclerosis | Medicine, Experimental | Genetic aspects | T cells | Health aspects | Integrins | Phosphorylation | Central nervous system | Blocking antibodies | Effector cells | Intracellular signalling | Lymphocytes T | Kinases | Leukoencephalopathy | Blood | Progressive multifocal leukoencephalopathy | Proteins | Infusion | Interleukin 2 | Blood-brain barrier | Lymphocytes | Etiology | Peripheral blood | Recombinant | Binding | Antigens | Receiving | CD49d antigen | Cytokines | Extracellular signal-regulated kinase | MAP kinase | T cell receptors | Patients | Adhesion | Interleukin 17 | CD4 antigen | Mode of action | Pathology | Internalization | γ-Interferon | Monoclonal antibodies | Ligands | Interferon
Journal Article