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PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, pp. e37004 - e37004
Journal Article
PLoS Genetics, ISSN 1553-7390, 12/2010, Volume 6, Issue 12, pp. 1 - 21
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2008, Volume 105, Issue 5, pp. 1674 - 1679
Activating transcription factor 2 (ATF2) regulates transcription in response to stress and growth factor stimuli. Here, we use a mouse model in which ATF2 was... 
Genes | Skin cancers | Keratinocytes | Antibodies | Papilloma | Epidermis | Skin | Carcinogenesis | Cyclins | Tumors | Presenilin | β-catenin | Keratinocyte | Cyclin D1 | beta-catenin | CELLS | INDUCED TRANSFORMATION | MULTIDISCIPLINARY SCIENCES | TUMOR PROMOTION | RETINOIC ACID | GENE | keratinocyte | PATHWAY | presenilin | papilloma | IN-VIVO | cyclin D1 | MICE | C-JUN | MOUSE SKIN | 9,10-Dimethyl-1,2-benzanthracene - toxicity | Cyclin D1 - metabolism | Cell Proliferation | Activating Transcription Factor 2 - physiology | Tissue Array Analysis | Activating Transcription Factor 2 - genetics | Carcinogens - toxicity | Tumor Suppressor Proteins - genetics | DNA - biosynthesis | Papilloma - pathology | Papilloma - chemically induced | Skin Neoplasms - pathology | Epidermis - metabolism | Papilloma - genetics | Epidermis - pathology | Epidermis - drug effects | Skin Neoplasms - chemically induced | Receptor, Notch1 - metabolism | beta Catenin - metabolism | Proto-Oncogene Proteins c-myb - metabolism | Mice, Knockout | Tumor Suppressor Proteins - physiology | Keratinocytes - pathology | Animals | Tetradecanoylphorbol Acetate - toxicity | Keratinocytes - drug effects | Keratinocytes - metabolism | Presenilin-1 - metabolism | Skin Neoplasms - genetics | Activating Transcription Factor 2 - analysis | Mice | Apoptosis | Tumor Suppressor Proteins - analysis | Influence | Genetic aspects | Research | DNA binding proteins | Properties | Skin cancer | Rodents | Models | Mutation | Comparative analysis | Cells | Index Medicus | Biological Sciences
Journal Article
Pigment Cell & Melanoma Research, ISSN 1755-1471, 04/2008, Volume 21, Issue 2, pp. 111 - 112
Journal Article
Stem Cell Reports, ISSN 2213-6711, 04/2019, Volume 12, Issue 4, pp. 787 - 800
In type 1 diabetes, a renewable source of human pancreatic β cells, in particular from human induced pluripotent stem cell (hiPSC) origin, would greatly... 
encapsulation | differentiation | stem cell | therapy | iPSC | β cell | diabetes mellitus | human | ISLET TRANSPLANTATION | IN-VITRO | EMBRYONIC STEM-CELLS | BETA-CELLS | INSULIN-PRODUCING CELLS | DEVICE | MATURE | GENERATION | SECRETING CELLS | EFFICIENT DIFFERENTIATION | CELL & TISSUE ENGINEERING | CELL BIOLOGY
Journal Article
Cell, ISSN 0092-8674, 2004, Volume 117, Issue 7, pp. 941 - 952
Hypoxia-inducible factor-1α (HIF1α) is a central regulator of the cellular response to hypoxia. Prolyl-hydroxylation of HIF1α by PHD enzymes is prerequisite... 
MICE PARTIALLY DEFICIENT | MAMMALIAN HOMOLOGS | OXYGEN-TENSION | INDUCIBLE FACTOR 1-ALPHA | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR-SUPPRESSOR | GROWTH-FACTOR | HIF-1-ALPHA | O-2 TENSION | HIF-ALPHA | UBIQUITIN-PROTEASOME PATHWAY | CELL BIOLOGY | Ubiquitin | Hypoxia | Research | Gene expression
Journal Article
STEM CELLS Translational Medicine, ISSN 2157-6564, 10/2015, Volume 4, Issue 10, pp. 1214 - 1222
Journal Article
Molecular Cell, ISSN 1097-2765, 2005, Volume 18, Issue 5, pp. 577 - 587
Activating transcription factor 2 (ATF2) is regulated by JNK/p38 in response to stress. Here, we demonstrate that the protein kinase ATM phosphorylates ATF2 on... 
COMPLEX | REPAIR | GENE | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOUBLE-STRAND BREAKS | NUCLEAR FOCI | CHECKPOINT | C-JUN | IDENTIFICATION | ACTIVATING TRANSCRIPTION FACTOR-2 | CELL BIOLOGY | Protein Kinases - metabolism | Phosphorylation | Fibroblasts - physiology | Humans | Molecular Sequence Data | DNA-Binding Proteins - metabolism | MRE11 Homologue Protein | RNA Interference | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Transcription, Genetic | Protein-Serine-Threonine Kinases - metabolism | Radiation, Ionizing | Antibodies, Phospho-Specific - metabolism | Amino Acid Sequence | Cell Line | Tumor Suppressor Proteins - metabolism | Cell Cycle Proteins - metabolism | Protein-Serine-Threonine Kinases - genetics | Nuclear Proteins - metabolism | Ataxia Telangiectasia Mutated Proteins | Transcription Factors - genetics | DNA Repair Enzymes | DNA-Binding Proteins - genetics | Serine - metabolism | Transcription Factors - metabolism | Cyclic AMP Response Element-Binding Protein - genetics | Sequence Alignment | Animals | Activating Transcription Factor 2 | DNA Repair | Cyclic AMP Response Element-Binding Protein - metabolism | Checkpoint Kinase 2 | Checkpoint Kinase 1 | Fibroblasts - cytology | Mice | DNA Damage | Histones - metabolism | Automated teller machines | Research | DNA | Oncology, Experimental | Radiation | Cancer | Index Medicus
Journal Article
Molecular Cell, ISSN 1097-2765, 2005, Volume 19, Issue 3, pp. 309 - 320
Activation of the Jun-N-terminal kinase (JNK) signaling cascade by phorbol esters (TPA) or protein kinase C (PKC) is well documented, although the underlying... 
SIGNAL-TRANSDUCTION | APOPTOSIS | TYROSINE PHOSPHORYLATION | SRC | BIOCHEMISTRY & MOLECULAR BIOLOGY | PKC-ALPHA | INVOLVEMENT | MYELOID-LEUKEMIA CELLS | TUMORIGENICITY | MKK7 | BETA | CELL BIOLOGY | Phorbol esters | Protein kinases | Incendiary weapons | Melanoma
Journal Article