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Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 09/2017, Volume 21, Issue 9, pp. 2199 - 2210
Immature myeloid cells including myeloid‐derived suppressor cells (MDSCs) and tumour‐associated macrophages (TAMs) promote tumour growth and metastasis by... 
tumour‐associated macrophages | B7‐H3 | HNSCC | myeloid‐derived suppressor cells | immunotherapy | B7-H3 | tumour-associated macrophages | myeloid-derived suppressor cells | SURVIVAL | MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | METASTASIS | CANCER | CELL BIOLOGY | REGULATORY T-CELLS | TUMOR-ASSOCIATED MACROPHAGES | SUPPRESSOR-CELLS | INHIBITOR | EXPRESSION | Prognosis | Macrophages - pathology | Carcinoma, Squamous Cell - pathology | Humans | Carcinogenesis - metabolism | Carcinoma, Squamous Cell - immunology | B7 Antigens - antagonists & inhibitors | B7 Antigens - metabolism | Head and Neck Neoplasms - pathology | Mice, Knockout | Carcinogenesis - pathology | Head and Neck Neoplasms - immunology | Animals | Receptors, Transforming Growth Factor beta - metabolism | Myeloid Cells - metabolism | Myeloid Cells - pathology | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Myeloid-Derived Suppressor Cells - pathology | Squamous cell carcinoma | Genetic engineering | T cells | Analysis | Immunotherapy | Flow cytometry | Transformation | Mucosa | Transgenic | Cytotoxicity | Lymphocytes T | Macrophages | Suppressor cells | B7 antigen | Metastases | Angiogenesis | Cell activation | Neck | Inhibition | Activation analysis | Myeloid cells | Head | Immune response | Immune systems | Cytometry | Immunosuppression | Immune checkpoint | Head and neck cancer | In vivo methods and tests | Tumors | Index Medicus | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, pp. e74888 - e74888
The molecular mechanism of human anal squamous cell carcinoma (ASCC) is unclear, and the accumulating evidence indicate association of ASCC with the activation... 
HEAD | ACTIVATION | TGF-BETA | PATHWAY | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | GROWTH | SQUAMOUS-CELL CARCINOMA | PTEN | NECK | CANCER | Immunohistochemistry | In Situ Nick-End Labeling | PTEN Phosphohydrolase - genetics | Protein-Serine-Threonine Kinases - deficiency | PTEN Phosphohydrolase - deficiency | Phosphorylation | Receptors, Transforming Growth Factor beta - genetics | Sirolimus - therapeutic use | TOR Serine-Threonine Kinases - metabolism | Anus Neoplasms - metabolism | Protein-Serine-Threonine Kinases - genetics | Male | Mice, Transgenic | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Mice, Knockout | Anti-Bacterial Agents - therapeutic use | Animals | TOR Serine-Threonine Kinases - genetics | Anus Neoplasms - genetics | Female | Mice | Receptors, Transforming Growth Factor beta - deficiency | Prevention | Squamous cell carcinoma | Genetic engineering | Carcinogenesis | Health aspects | Cancer | TOR protein | Genomics | AKT protein | Infections | Kinases | Carcinogens | Human papillomavirus | Clonal deletion | Transgenic animals | Education | Rodents | Deletion | Tumorigenesis | Bioindicators | Lesions | Growth factors | Developmental biology | Transgenic mice | Rapamycin | Molecular chains | Biomarkers | Mutation | Laboratory animals | PTEN protein | Cervical cancer | Tumors | Index Medicus
Journal Article
Journal of Navigation, ISSN 0373-4633, 11/2018, Volume 71, Issue 6, pp. 1511 - 1530
Journal Article
Neoplasia, ISSN 1476-5586, 2014, Volume 16, Issue 10, pp. 789 - 800
Abstract Cancer stem cells (CSC) and genes have been linked to cancer development and therapeutic resistance, but the signaling mechanisms regulating CSC genes... 
Oncology | DMAT, 2-Dimethylamino-4,5,6,7-tetrabromo-1H-benzimidazole | UM-SCC, University of Michigan Squamous Cell Carcinoma | mt, Mutant | HEKA, Human epidermal keratinocytes | HOK, Human oral keratinocytes | TP53, Transforming Protein p53 | wt, Wild-type | TAp73, Transactivating p73 | HNSCC, Head and neck squamous cell carcinoma | CK2, Casein Kinase 2 | SP, Side population | CSC, Cancer Stem Cells | APOPTOSIS | ONCOLOGY | PROTEIN-KINASE CK2 | KAPPA-B | DIFFERENTIATION | INDUCTION | IDENTIFICATION | CARCINOMA | P73 | ASSOCIATION | P53 | Phosphorylation | Neoplastic Stem Cells - drug effects | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Gene Expression Regulation, Neoplastic | Threonine - metabolism | Head and Neck Neoplasms - metabolism | Tumor Suppressor Protein p53 - genetics | DNA-Binding Proteins - metabolism | Octamer Transcription Factor-3 - genetics | Squamous Cell Carcinoma of Head and Neck | Neoplastic Stem Cells - metabolism | SOXB1 Transcription Factors - genetics | Tumor Suppressor Proteins - genetics | Neoplastic Stem Cells - pathology | Nuclear Proteins - genetics | Tumor Suppressor Proteins - metabolism | Nanog Homeobox Protein | RNA, Small Interfering - pharmacology | Casein Kinase II - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Nuclear Proteins - metabolism | DNA-Binding Proteins - genetics | Homeodomain Proteins - genetics | Head and Neck Neoplasms - pathology | Tumor Protein p73 | Cell Line, Tumor | Benzimidazoles - pharmacology | Head and Neck Neoplasms - genetics | Mutation | Casein Kinase II - metabolism | Index Medicus
Journal Article
Journal of Translational Medicine, ISSN 1479-5876, 05/2017, Volume 15, Issue 1, pp. 92 - 92
Background: Patients with hereditary diffuse gastric cancer (HDGC), a cancer predisposition syndrome associated with germline mutations of the CDH1... 
C.1380delA CDH1 gastric cancer cells | Hereditary diffuse gastric cancer (HDGC) | Therapeutic leads | Differential gene expression profiling | High throughput drug screening | EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | MEDICINE, RESEARCH & EXPERIMENTAL | RESPONSES | ADHESION | c. 1380delA CDH1 gastric cancer cells | CYTOSKELETAL ORGANIZATION | CADHERIN MISSENSE MUTATIONS | Stomach Neoplasms - genetics | Genetic Predisposition to Disease | Reproducibility of Results | Humans | Gene Expression Regulation, Neoplastic | Male | Stomach Neoplasms - pathology | Up-Regulation - genetics | Gene Expression Profiling | Extracellular Signal-Regulated MAP Kinases - metabolism | Stomach Neoplasms - drug therapy | Germ-Line Mutation - genetics | High-Throughput Screening Assays | Diglycerides - metabolism | Pedigree | Cell Line, Tumor | Adult | Female | Cadherins - genetics | Drug Evaluation, Preclinical | Inositol Phosphates - metabolism | Cadherins - deficiency | TOR protein | Protein kinase C | Diacylglycerol | Cytotoxicity | Intracellular signalling | Genomes | Metastasis | Kinases | Drug screening | Cancer therapies | Stomach cancer | E-cadherin | Metastases | Cell adhesion & migration | Histopathology | Drug dosages | Gastric cancer | Inositol 1,4,5-trisphosphate | DNA topoisomerase (ATP-hydrolysing) | Extracellular signal-regulated kinase | MAP kinase | Rapamycin | Breast cancer | Tumor cell lines | Gene expression | Src protein | Patients | 1-Phosphatidylinositol 3-kinase | Inositols | Inhibitors | Mutation | Focal adhesion kinase | Cancer | Apoptosis | Index Medicus | c.1380delA CDH1 gastric cancer cells
Journal Article
2003, Cancer treatment and research, ISBN 9781402073403, Volume 115, 354
Book
Journal Article
Oncogene, ISSN 0950-9232, 07/2012, Volume 31, Issue 28, pp. 3322 - 3332
The molecular mechanisms that contribute to the initiation and progression of head and neck squamous cell carcinoma (HNSCC) have not been completely... 
cancer mouse model | TGF-β | conditional knockout | head and neck squamous cell carcinoma | PI3K/Akt | PATHWAYS | APOPTOSIS | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MAMMARY-CARCINOMA | FACTOR-KAPPA-B | MODEL | CELL BIOLOGY | COLON-CANCER | TGF-beta | GROWTH-FACTOR-BETA | ONCOLOGY | GENETICS & HEREDITY | TUMOR-SUPPRESSOR | TUMORIGENESIS | Cell Proliferation | Epithelial Cells - metabolism | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Cell Count | Humans | Transforming Growth Factor beta1 - metabolism | Apoptosis - genetics | Phosphatidylinositol 3-Kinases - metabolism | Inflammation - complications | Neoplastic Stem Cells - metabolism | Neoplastic Stem Cells - pathology | Proto-Oncogene Proteins c-akt - metabolism | Cellular Senescence - genetics | PTEN Phosphohydrolase - genetics | PTEN Phosphohydrolase - deficiency | PTEN Phosphohydrolase - metabolism | Epithelial Cells - pathology | Penetrance | Signal Transduction - genetics | Transforming Growth Factor beta1 - genetics | Gene Knockout Techniques | Head and Neck Neoplasms - pathology | Animals | Carcinoma, Squamous Cell - complications | Cell Line, Tumor | Head and Neck Neoplasms - complications | Head and Neck Neoplasms - genetics | Mice | Myeloid Cells - pathology | Index Medicus | Cell proliferation | Animal models | Senescence | Transformation | biomarkers | AKT protein | Inflammation | Transforming growth factor- beta | Suppressor cells | Carcinogenesis | 1-Phosphatidylinositol 3-kinase | Metastases | Angiogenesis | Signal transduction | Cell activation | Molecular modelling | Stem cells | Head and neck | Head and neck cancer | Microenvironments | PTEN protein | Apoptosis | Tumors | squamous cell carcinoma | Head and Neck Squamous Cell Carcinoma (HNSCC) | Akt | PI3K | Conditional Knockout | Cancer Mouse Model
Journal Article
Cancer Research, ISSN 0008-5472, 10/2014, Volume 74, Issue 19 Supplement, pp. 1925 - 1925
Journal Article
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 07/2013, Volume 19, Issue 14, pp. 3808 - 3819
Journal Article