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index medicus (62) 62
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1. Full Text Aryl Hydrocarbon Receptor Nuclear Translocator-Like (BMAL1) Is Associated with Susceptibility to Hypertension and Type 2 Diabetes
by Peng Y. Woon and Pamela J. Kaisaki and José Bragança and Marie-Thérèse Bihoreau and Jonathan C. Levy and Martin Farrall and Dominique Gauguier
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2007, Volume 104, Issue 36, pp. 14412 - 14417
Many aspects of physiology and behavior follow a circadian rhythm. Brain and muscle Arnt-like protein-1 (BMAL1) is a key component of the mammalian molecular... 
Hypertension | Haplotypes | Plasmids | Genes | Genomics | Blood pressure | Gene expression regulation | Promoter regions | Type 2 diabetes mellitus | Human genetics | Comparative genomics | SNP | Sequence variation | Molecular clock | Genetic polymorphism | METABOLIC SYNDROME | MYOCARDIAL-INFARCTION | CIRCADIAN-RHYTHMS | MULTIDISCIPLINARY SCIENCES | MAMMALIAN CLOCK GENE | QUANTITATIVE TRAIT LOCUS | RAT-CHROMOSOME 1 | WISTAR-KYOTO | sequence variation | comparative genomics | BLOOD-PRESSURE | molecular clock | SUPRACHIASMATIC NUCLEUS | genetic polymorphism | Blood Pressure | Diabetes Mellitus, Type 2 - genetics | Humans | Diabetes Mellitus, Type 2 - metabolism | Promoter Regions, Genetic - genetics | Basic Helix-Loop-Helix Transcription Factors - metabolism | Base Sequence | Hypertension - genetics | Diabetes Mellitus, Type 2 - complications | Rats, Inbred SHR | ARNTL Transcription Factors | Genetic Predisposition to Disease | Basic Helix-Loop-Helix Transcription Factors - genetics | GATA4 Transcription Factor - metabolism | Risk Factors | Rats | GATA4 Transcription Factor - genetics | Genotype | Hypertension - physiopathology | Hypertension - metabolism | Animals | Hypertension - complications | Polymorphism, Single Nucleotide - genetics | Quality of Life | Diabetes Mellitus, Type 2 - pathology | Transcription, Genetic - genetics | Type 2 diabetes | Evaluation | Molecular clock (Genetics) | Physiological aspects | Genetic aspects | Single nucleotide polymorphisms | Muscle proteins | Risk factors | Dopamine receptors | Proteins | Rodents | Genetics | Circadian rhythm | Diabetes | Comparative analysis | Index Medicus | Biological Sciences
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2. Full Text ANKS3 Co-localises with ANKS6 in mouse renal cilia and is associated with vasopressin signaling and apoptosis in vivo in mice
by Delestré, Laure and Bakey, Zeineb and Prado, Cécilia and Hoffmann, Sigrid and Bihoreau, Marie-Thérèse and Lelongt, Brigitte and Gauguier, Dominique
PLoS ONE, ISSN 1932-6203, 09/2015, Volume 10, Issue 9, pp. e0136781 - e0136781
Mutations in Ankyrin repeat and sterile alpha motif domain containing 6 (ANKS6) play a causative role in renal cyst formation in the PKD/Mhm(cy/+) rat model of... 
STERILE ALPHA-MOTIF | PROTEIN | POLYCYSTIC KIDNEY-DISEASE | MULTIDISCIPLINARY SCIENCES | ANKYRIN REPEAT | BICAUDAL-C | SAM DOMAIN | PROGRESSION | BICC1 | CAMP | SYSTEMS BIOLOGY | Polycystic Kidney Diseases - genetics | Polycystic Kidney Diseases - metabolism | Cell Proliferation - genetics | Protein Binding - genetics | Mice, Inbred C57BL | Cell Proliferation - physiology | Vasopressins - genetics | Apoptosis - genetics | Vasopressins - metabolism | Signal Transduction - genetics | Mutation - genetics | Cilia - metabolism | Down-Regulation - genetics | Cilia - genetics | Ankyrin Repeat - genetics | Carrier Proteins - genetics | Kidney - metabolism | Animals | Carrier Proteins - metabolism | Signal Transduction - physiology | Mice | Apoptosis - physiology | Amino Acid Motifs - genetics | Protein Binding - physiology | Physiological aspects | Genetic aspects | Pituitary hormones | Research | Protein binding | Apoptosis | Cell proliferation | Yeast | Immunoprecipitation | Transcription | Laboratories | Syngeneic grafts | Genes | Oligonucleotides | Amino acids | Cardiovascular disease | Biology | Proteins | Genotype & phenotype | Rodents | Localization | Cilia | Nephronophthisis | Polycystic kidney | Antisense oligonucleotides | Permeability | Gene expression | Vasopressin | Signaling | Molecular modelling | Cysts | Acids | Plasmids | MicroRNAs | Hybrid systems | Ankyrin | Mutation | Kidney diseases | Structure-function relationships | Index Medicus | Life Sciences
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3. Full Text Molecular genetics of the transcription factor GLIS3 identifies its dual function in beta cells and neurons
by Calderari, Sophie and Ria, Massimiliano and Gérard, Christelle and Nogueira, Tatiane C and Villate, Olatz and Collins, Stephan C and Neil, Helen and Gervasi, Nicolas and Hue, Christophe and Suarez-Zamorano, Nicolas and Prado, Cécilia and Cnop, Miriam and Bihoreau, Marie-Thérèse and Kaisaki, Pamela J and Cazier, Jean-Baptiste and Julier, Cécile and Lathrop, Mark and Werner, Michel and Eizirik, Decio L and Gauguier, Dominique
Genomics, ISSN 0888-7543, 03/2018, Volume 110, Issue 2, pp. 98 - 111
The GLIS family zinc finger 3 isoform (GLIS3) is a risk gene for Type 1 and Type 2 diabetes, glaucoma and Alzheimer's disease endophenotype. We identified... 
Goto-Kakizaki rat | Quantitative trait locus | Single nucleotide polymorphism | Alzheimer's disease | ChIP sequencing | Diabetes mellitus | COMMON VARIANTS | NEONATAL DIABETES-MELLITUS | METAANALYSIS | ZINC-FINGER | LOCI | AUTOPHAGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | INSULIN-RESISTANCE | GENETICS & HEREDITY | NEUROGENIN 3 | TYPE-1 | GENOME-WIDE ASSOCIATION | Index Medicus
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4. Full Text SNP and haplotype mapping for genetic analysis in the rat
by Saar, K and Beck, A and Bihoreau, M and Birney, E and Brocklebank, D and Chen, Y and Cuppen, E and Demonchy, S and Dopazo, J and Flicek, P and Foglio, M and Fujiyama, A and Gut, I.G and Gauguier, D and Guigo, R and Guryev, V and Heinig, M and Hummel, O and Jahn, N and Klages, S and Kren, V and Kube, M and Kuhl, H and Kuramoto, T and Kuroki, Y and Lechner, D and Lee, Y.A and Lopez-Bigas, N and Lathrop, G.M and Mashimo, T and Medina, I and Mott, R and Patone, G and Perrier-Cornet, J.A and Platzer, M and Pravenec, M and Reinhardt, R and Sakaki, Y and Schilhabel, M and Schulz, H and Serikawa, T and Shikhagaie, M and Tatsumoto, S and Taudien, S and Toyoda, A and Voigt, B and Zelenika, D and Zimdahl, H and Hubner, N and STAR Consortium and The STAR Consortium
Nature Genetics, ISSN 1061-4036, 2008, Volume 40, Issue 5, pp. 560 - 566
The laboratory rat is one of the most extensively studied model organisms. Inbred laboratory rat strains originated from limited Rattus norvegicus founder... 
INBRED MOUSE STRAINS | LABORATORY MOUSE | WISTAR-KYOTO RATS | EVOLUTIONARY | HUMAN GENOME | SET | SEQUENCE | DISEASE | GENETICS & HEREDITY | SPONTANEOUSLY HYPERTENSIVE RAT | MAP | Haplotypes | Animals | Rats, Inbred Strains - genetics | Recombination, Genetic | Databases, Genetic | Chromosome Mapping | Polymorphism, Single Nucleotide | Phylogeny | Genome | Quantitative Trait Loci | Rats - genetics | Linkage Disequilibrium | Chromosome mapping | Genetic aspects | Rats as laboratory animals | Single nucleotide polymorphisms | Identification and classification | Methods | Studies | Genetic disorders | Disease | Genomics | Rodents | Science | Genetics | Inbreeding | Amino acids | Laboratory animals | Drug resistance | Index Medicus | Genètica | Rates | Haplotip | Locus de caràcters quantitatius | Polimorfisme de nucleòtids simples | Bases de dades
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5. Full Text Direct quantitative trait locus mapping of mammalian metabolic phenotypes in diabetic and normoglycemic rat models
by Nicholson, Jeremy K and Wallis, Robert H and Baunsgaard, Dorrit and Sidelmann, Ulla Grove and Keun, Hector C and Gauguier, Dominique and Barton, Richard H and D'Amato, Lisa and Dumas, Marc-Emmanuel and Blancher, Christine and Wilder, Steven P and Argoud, Karène and Bihoreau, Marie-Thérèse and Fearnside, Jane F and Scott, James
Nature Genetics, ISSN 1061-4036, 05/2007, Volume 39, Issue 5, pp. 666 - 672
Characterizing the relationships between genomic and phenotypic variation is essential to understanding disease etiology. Information-dense data sets derived... 
CROSSES | GOTO-KAKIZAKI RATS | BROWN-NORWAY | METABONOMICS | GENETICS | DISEASE | GENETICS & HEREDITY | SYSTEMS | EXPRESSION | ASSOCIATION | GENOME | Diabetes Mellitus - genetics | Benzoates - chemistry | Biomarkers - analysis | Genome - genetics | Molecular Sequence Data | Rats | Sequence Analysis, DNA | Lod Score | Glucuronosyltransferase - genetics | Phenotype | Animals | Base Sequence | Nuclear Magnetic Resonance, Biomolecular | Molecular Structure | Quantitative Trait Loci | Metabolism - genetics | Genetic Linkage | Quantitative trait loci | Physiological aspects | Research | Genetic variation | Genotype & phenotype | Genetic markers | Diabetes | Genomics | Rodents | Proteomics | Index Medicus
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6. Full Text Familial breast cancer and DNA repair genes: Insights into known and novel susceptibility genes from the GENESIS study, and implications for multigene panel testing
: DNA repair genes and familial breast cancer
by Girard, Elodie and Eon-Marchais, Séverine and Olaso, Robert and Renault, Anne-Laure and Damiola, Francesca and Dondon, Marie-Gabrielle and Barjhoux, Laure and Goidin, Didier and Meyer, Vincent and Le Gal, Dorothée and Beauvallet, Juana and Mebirouk, Noura and Lonjou, Christine and Coignard, Juliette and Marcou, Morgane and Cavaciuti, Eve and Baulard, Céline and Bihoreau, Marie-Thérèse and Cohen-Haguenauer, Odile and Leroux, Dominique and Penet, Clotilde and Fert-Ferrer, Sandra and Colas, Chrystelle and Frebourg, Thierry and Eisinger, François and Adenis, Claude and Fajac, Anne and Gladieff, Laurence and Tinat, Julie and Floquet, Anne and Chiesa, Jean and Giraud, Sophie and Mortemousque, Isabelle and Soubrier, Florent and Audebert-Bellanger, Séverine and Limacher, Jean-Marc and Lasset, Christine and Lejeune-Dumoulin, Sophie and Dreyfus, Hélène and Bignon, Yves-Jean and Longy, Michel and Pujol, Pascal and Venat-Bouvet, Laurence and Bonadona, Valérie and Berthet, Pascaline and Luporsi, Elisabeth and Maugard, Christine M and Noguès, Catherine and Delnatte, Capucine and Fricker, Jean-Pierre and Gesta, Paul and Faivre, Laurence and Lortholary, Alain and Buecher, Bruno and Caron, Olivier and Gauthier-Villars, Marion and Coupier, Isabelle and Servant, Nicolas and Boland, Anne and Mazoyer, Sylvie and Deleuze, Jean-François and Stoppa-Lyonnet, Dominique and Andrieu, Nadine and Lesueur, Fabienne
International Journal of Cancer, ISSN 0020-7136, 04/2019, Volume 144, Issue 8, pp. 1962 - 1974
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7. Full Text The SAM domain of ANKS6 has different interacting partners and mutations can induce different cystic phenotypes
by Bakey, Zeineb and Bihoreau, Marie-Thérèse and Piedagnel, Rémi and Delestré, Laure and Arnould, Catherine and de Villiers, Alexandre d'Hotman and Devuyst, Olivier and Hoffmann, Sigrid and Ronco, Pierre and Gauguier, Dominique and Lelongt, Brigitte
Kidney International, ISSN 0085-2538, 08/2015, Volume 88, Issue 2, pp. 299 - 310
The ankyrin repeat and sterile α motif (SAM) domain–containing six gene ( ) is a candidate for polycystic kidney disease (PKD). Originally identified in the... 
renal pathology | ADPKD | genetic kidney disease | NEK8 | PROTEIN | RAT | SAMCYSTIN | POLYCYSTIC KIDNEY-DISEASE | NEPHRONOPHTHISIS | BICC1 | GENE | MOUSE MODEL | UROLOGY & NEPHROLOGY | BICAUDAL C | Kidney - embryology | Kidney - pathology | Humans | Loop of Henle - metabolism | Loop of Henle - pathology | Male | Mutation, Missense | Kidney - metabolism | Polycystic Kidney, Autosomal Dominant - genetics | Kidney Diseases, Cystic - genetics | Kidney Glomerulus - metabolism | Female | Nuclear Proteins - genetics | Kidney Diseases, Cystic - physiopathology | Podocytes - metabolism | Kidney Tubules, Collecting - pathology | Rats | Nuclear Proteins - metabolism | Ankyrin Repeat | Kidney Glomerulus - pathology | Cilia - metabolism | Mice, Inbred C3H | Polycystic Kidney, Autosomal Dominant - metabolism | Carrier Proteins - genetics | Homozygote | Kidney Tubules, Collecting - metabolism | Kidney Diseases, Cystic - metabolism | Phenotype | Animals | Carrier Proteins - metabolism | Mice | RNA-Binding Proteins - metabolism | Index Medicus
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8. Full Text EGR2 mutation enhances phenotype spectrum of Dejerine–Sottas syndrome
by Gargaun, Elena and Seferian, Andreea Mihaela and Cardas, Ruxandra and Le Moing, Anne-Gaelle and Delanoe, Catherine and Nectoux, Juliette and Nelson, Isabelle and Bonne, Gisèle and Bihoreau, Marie-Thérèse and Deleuze, Jean-François and Boland, Anne and Masson, Cécile and Servais, Laurent and Gidaro, Teresa
Journal of Neurology, ISSN 0340-5354, 7/2016, Volume 263, Issue 7, pp. 1456 - 1458
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1007/s00415-016-8153-9 
Neurology | Neurosciences | Medicine & Public Health | Neuroradiology | NEUROPATHY | MOTOR-NEURON DISEASE | VIALETTO-VAN-LAERE | CLINICAL NEUROLOGY | Hereditary Sensory and Motor Neuropathy - physiopathology | Hereditary Sensory and Motor Neuropathy - pathology | DNA Mutational Analysis | Early Growth Response Protein 2 - genetics | Hereditary Sensory and Motor Neuropathy - genetics | Humans | Child, Preschool | Female | Mutation - genetics
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9. Full Text Inhibition of comt with tolcapone slows progression of polycystic kidney disease in the more severely affected PKD/Mhm (cy/+) substrain of the hannover sprague-dawley rat
by Boehn, Susanne N.E and Spahn, Sonja and Neudecker, Sabine and Keppler, Andrea and Bihoreau, Marie-Thérèse and Kränzlin, Bettina and Pandey, Priyanka and Hoffmann, Sigrid C and Li, Li and Torres, Vicente E and Gröne, Hermann-Josef and Gretz, Norbert
Nephrology Dialysis Transplantation, ISSN 0931-0509, 08/2013, Volume 28, Issue 8, pp. 2045 - 2058
Background. Autosomal dominant polycystic kidney disease (ADPKD) is one of the most common human inherited diseases. Modifier genes seem to modulate the... 
Catechol-O-methyltransferase | Gene expression profiling | Modifier | Tolcapone | Polycystic kidney disease | catechol-O-methyltransferase | tolcapone | modifier | QUANTITATIVE TRAIT | CELL-PROLIFERATION | RENAL-DISEASE | MODEL | TRANSPLANTATION | PHYTOESTROGEN CONTENT | polycystic kidney disease | TUBULOINTERSTITIAL FIBROSIS | UROLOGY & NEPHROLOGY | gene expression profiling | SIGNAL-REGULATED KINASE | TRANSCRIPTION FACTOR | SEVERITY | Biomarkers - metabolism | Oligonucleotide Array Sequence Analysis | Apoptosis - drug effects | Humans | RNA, Messenger - genetics | Enzyme Inhibitors - pharmacology | Benzophenones - pharmacology | Rats | Male | Gene Expression Profiling | Polycystic Kidney Diseases - drug therapy | Reverse Transcriptase Polymerase Chain Reaction | Rats, Sprague-Dawley | Blotting, Western | Disease Progression | Immunoenzyme Techniques | Animals | Polycystic Kidney Diseases - pathology | Nitrophenols - pharmacology | Female | Catechol O-Methyltransferase Inhibitors | Cell Proliferation - drug effects | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Index Medicus | Original
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10. Full Text Untargeted metabolome quantitative trait locus mapping associates variation in urine glycerate to mutant glycerate kinase
by Cazier, Jean-Baptise and Kaisaki, Pamela J and Argoud, Kare and Blaise, Benjamin J and Veselkov, Kirill and Ebbels, Timothy M. D and Tsang, Tsz and Wang, Yulan and Bihoreau, Marie-Thérèse and Mitchell, Steve C and Holmes, Elaine C and Lindon, John C and Scott, James and Nicholson, Jeremy K and Dumas, Marc-Emmanuel and Gauguier, Dominique
Journal of Proteome Research, ISSN 1535-3893, 02/2012, Volume 11, Issue 2, pp. 631 - 642
With successes of genome-wide association studies, molecular phenotyping systems are developed to identify genetically determined disease-associated... 
metabonomics | genetics | glycerate kinase | quantitative trait locus | functional genomics | glycerate | NMR spectroscopy | BIOCHEMICAL RESEARCH METHODS | INSULIN-RESISTANT MICE | SYSTEMS BIOLOGY | Metabolomics | Phosphotransferases (Alcohol Group Acceptor) - deficiency | Male | Phosphotransferases (Alcohol Group Acceptor) - genetics | Lod Score | Metabolome - genetics | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Glyceric Acids - urine | Animals | Analysis of Variance | Chromosome Mapping - methods | Computer Simulation | Nuclear Magnetic Resonance, Biomolecular | Mice | Mice, Inbred BALB C | Genomics - methods | Quantitative Trait Loci | Index Medicus | Analytical chemistry | Chemical Sciences
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11. Full Text Genetic control of differential acetylation in diabetic rats
by Kaisaki, Pamela J and Otto, Georg W and McGouran, Joanna F and Toubal, Amine and Argoud, Karène and Waller-Evans, Helen and Finlay, Clare and Caldérari, Sophie and Bihoreau, Marie-Thérèse and Kessler, Benedikt M and Gauguier, Dominique and Mott, Richard
PLoS ONE, ISSN 1932-6203, 04/2014, Volume 9, Issue 4, pp. e94555 - e94555
Post-translational protein modifications such as acetylation have significant regulatory roles in metabolic processes, but their relationship to both variation... 
LONGEVITY | LIVER-MITOCHONDRIA | DEACETYLASE | MULTIDISCIPLINARY SCIENCES | STRESS | ACETYLOME | RODENT MODEL | SIRT3 | INSIGHTS | Species Specificity | Diabetes Mellitus, Type 2 - genetics | Diabetes Mellitus, Experimental - genetics | Male | Diabetes Mellitus, Type 2 - metabolism | Acetyltransferases - genetics | Pyrimidines - metabolism | Amino Acids - metabolism | Transcription, Genetic | Acetylation | Diabetes Mellitus, Experimental - metabolism | Fatty Acids - metabolism | Gluconeogenesis | Sirtuin 3 - genetics | Purines - metabolism | Liver - metabolism | Gene Expression Regulation | Rats | Citric Acid Cycle | Polymorphism, Genetic | Animals | Sequence Analysis, RNA | Proteomics | Glycolysis | Protein Processing, Post-Translational | Pentose Phosphate Pathway | Care and treatment | Analysis | Genetic aspects | Diabetes | Diagnosis | Research | Transcription | Liver | Homeostasis | Genomes | Single-nucleotide polymorphism | Kinases | Metabolic syndrome | Proteins | Spectrometry | Mitochondria | Pathways | Metabolites | Rodents | Post-translation | Genetics | Inbreeding | Chromosomes | Bioinformatics | Deoxyribonucleic acid--DNA | Enzymes | Nutrition | Genetic control | Nucleotide sequence | Diabetes mellitus | Genetic diversity | Gene expression | Studies | NAD | Musculoskeletal system | Lysine | Metabolic disorders | Index Medicus | Life Sciences | Human health and pathology | Deoxyribonucleic acid | DNA
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12. Nouveaux outils de cartographie génétique et génomique pour les études des traits complexes chez le rat
by Gauguier, Dominique and Bihoreau, Marie-Thérèse
Medecine/Sciences, ISSN 0767-0974, 05/2008, Volume 24, Issue 5, pp. 545 - 547
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13. Full Text Broad-ranging natural metabotype variation drives physiological plasticity in healthy control inbred rat strains
by Pontoizeau, Clément and Fearnside, Jane F and Navratil, Vincent and Domange, Céline and Cazier, Jean-Baptiste and Fernández-Santamaría, Cristina and Kaisaki, Pamela J and Emsley, Lyndon and Toulhoat, Pierre and Bihoreau, Marie-Thérèse and Nicholson, Jeremy K and Gauguier, Dominique and Dumas, Marc E
Journal of Proteome Research, ISSN 1535-3893, 04/2011, Volume 10, Issue 4, pp. 1675 - 1689
Maintaining homeostasis in higher organisms involves a complex interplay of multiple ubiquitous and organ-specific molecular mechanisms that can be... 
cardiometabolic syndrome | single nucleotide polymorphism | natural variation | metabonomics/metabolomics | metabolite-set enrichment analysis | NMR spectroscopy | pathway analysis | METABONOMICS | GENETIC-ANALYSIS | COMMUNITY VIEW | MOUSE | BIOCHEMICAL RESEARCH METHODS | GUT MICROBIOTA | QUANTITATIVE TRAIT LOCI | METABOLISM | MICE | HYPERTENSION | EXPRESSION | Rats - physiology | Phenotype | Animals | Humans | Metabolome | Nuclear Magnetic Resonance, Biomolecular | Male | Metabolic Networks and Pathways - physiology | Rats, Inbred Strains | Metabolomics - methods | Rats - metabolism | Cluster Analysis | Index Medicus | Metabolomics | Biochemistry, Molecular Biology | Rats | Life Sciences | Metabolic Networks and Pathways | Biomolecules
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