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Diabetes, ISSN 0012-1797, 10/2011, Volume 60, Issue 10, pp. 2455 - 2456
Journal Article
Diabetes, ISSN 0012-1797, 10/2011, Volume 60, Issue 10, pp. 2455 - 2456
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e87067
Pancreatic beta-cell dysfunction is central to type 2 diabetes pathogenesis. Prolonged elevated levels of circulating free-fatty acids and hyperglycemia, also... 
OXIDATIVE STRESS | GENE | MULTIDISCIPLINARY SCIENCES | N-TERMINAL KINASE | FATTY-ACID | DEATH | ENDOPLASMIC-RETICULUM STRESS | DYSFUNCTION | ACTIVATING TRANSCRIPTION FACTOR-3 | PALMITATE | TYPE-2 | RNA, Small Interfering - genetics | Transcription Factor CHOP - genetics | Palmitic Acid - pharmacology | Caspase 9 - genetics | Caspase 9 - metabolism | Mitogen-Activated Protein Kinase 8 - antagonists & inhibitors | Apoptosis - drug effects | Diabetes Mellitus, Type 2 - genetics | Humans | Caspase 3 - metabolism | Mitogen-Activated Protein Kinase 9 - genetics | RNA, Messenger - metabolism | Mitogen-Activated Protein Kinase 10 - genetics | RNA, Messenger - antagonists & inhibitors | Caspase 3 - genetics | Apoptosis Regulatory Proteins - genetics | Mitogen-Activated Protein Kinase 8 - genetics | Cell Line | Signal Transduction | Mitogen-Activated Protein Kinase 9 - metabolism | Mitogen-Activated Protein Kinase 8 - metabolism | RNA, Messenger - genetics | Diabetes Mellitus, Type 2 - enzymology | Gene Expression Regulation | Rats | Glucose - pharmacology | Apoptosis Regulatory Proteins - metabolism | Mitogen-Activated Protein Kinase 10 - metabolism | Animals | Insulin-Secreting Cells - drug effects | Mitogen-Activated Protein Kinase 9 - antagonists & inhibitors | Diabetes Mellitus, Type 2 - pathology | Insulin-Secreting Cells - pathology | Mitogen-Activated Protein Kinase 10 - antagonists & inhibitors | Transcription Factor CHOP - metabolism | Insulin-Secreting Cells - enzymology | RNA, Small Interfering - metabolism | Type 2 diabetes | Pancreatic beta cells | RNA | Glucose | Fatty acids | Dextrose | Apoptosis | Oxidative stress | Transcription factors | Pathogenesis | JNK3 protein | Kinases | Palmitic acid | Proteins | Mitochondria | Hyperglycemia | Pancreas | Obesity | Immunoglobulins | Cytokines | Diabetes mellitus | Markers | c-Jun protein | Caspase | Exposure | Gene expression | Insulin | Beta cells | Cell lines | Isoforms | Caspase-9 | Insulin resistance | Diabetes | Endoplasmic reticulum | Endocrinology
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2015, Volume 10, Issue 2, p. e0117130
Journal Article
Journal Article
Acta Obstetricia et Gynecologica Scandinavica, ISSN 0001-6349, 11/2014, Volume 93, Issue 11, pp. 1109 - 1122
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 10/2019, Volume 496, p. 110524
Decreased insulin secretory capacity in Type 2 diabetes mellitus is associated with beta-cell dedifferentiation and inflammation. We hypothesize that prolonged... 
Beta-cell dysfunction | Histone modifications | Proliferation | Beta-cell dedifferentiation | Cytokines | Insulin secretion
Journal Article
Acta Obstetricia et Gynecologica Scandinavica, ISSN 0001-6349, 11/2014, Volume 93, Issue 11, pp. 1190 - 1197
ObjectiveSeveral studies have shown increased beta cell mass during pregnancy in both rodents and humans. Proliferation of existing beta cells seems to be the... 
neogenesis | Neurogenin‐3 | pancreas | beta cells | pregnancy | Neurogenin-3 | PROGENITORS | RAT PANCREAS | ISLETS | BETA-CELLS | INSULIN-SECRETION | ENDOCRINE | MOUSE | PROLIFERATION | OBSTETRICS & GYNECOLOGY | GROWTH | MICE | Nerve Tissue Proteins - blood | Humans | Basic H