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eBook
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 5, p. e19424
Background: Complex biological processes such as acute inflammation induced by trauma/hemorrhagic shock/(T/HS) are dynamic and multi-dimensional. We utilized... 
HEMORRHAGIC-SHOCK | SYSTEMIC INFLAMMATION | IMMUNE-RESPONSE | NECROSIS-FACTOR RECEPTORS | IN-SILICO | MULTIDISCIPLINARY SCIENCES | KERATINOCYTE-DERIVED CHEMOKINE | INJURY | SEVERE TRAUMA | PLASMA CYTOKINE LEVELS | ENDOTHELIAL GROWTH-FACTOR | Nitrates - blood | Multivariate Analysis | Shock, Hemorrhagic - complications | Wounds and Injuries - blood | Mice, Inbred C57BL | Tumor Necrosis Factor-alpha - blood | Wounds and Injuries - complications | Interleukin-10 - blood | Male | Inflammation - etiology | Inflammation - blood | Shock, Hemorrhagic - blood | Animals | Interleukin-6 - blood | Nitrogen Dioxide - blood | Mice | Principal Component Analysis | Cluster Analysis | Automobile drivers | Interferon | Inflammation | Interleukins | Biological response modifiers | Analysis | Multiplexing | Cluster analysis | Plasma | Interleukin | Discriminators | Multivariate analysis | Hemorrhage | Complexity | Interleukin 6 | Rodents | Surgery | Network analysis | IP-10 protein | Mathematical models | Modelling | Postoperative period | Cytokines | Narcotics | Principal components analysis | Cannulation | Interleukin 12 | Data processing | Interleukin 13 | Tumor necrosis factor-α | Gene expression | Clustering | Trauma | Biological activity | Nodes | Medicine | Studies | Interleukin 10 | Hypoxia | Nitrogen dioxide | Laboratory animals
Journal Article
Journal Article
PLoS Medicine, ISSN 1549-1277, 07/2017, Volume 14, Issue 7, p. e1002365
Background The immunosuppression and immune dysregulation that follows severe injury includes type 2 immune responses manifested by elevations in interleukin... 
IMMUNE-RESPONSE | MEDICINE, GENERAL & INTERNAL | ORGAN INJURY | CYTOKINES | SYSTEMIC INFLAMMATORY RESPONSE | INTERLEUKIN-33 | INNATE LYMPHOID-CELLS | SUPPRESSOR-CELLS | PROMOTES | IL-33 | EXPRESSION | Shock, Hemorrhagic - complications | Humans | Middle Aged | Wounds and Injuries - etiology | Male | Interleukin-5 - genetics | Young Adult | Lymphocytes - immunology | Wounds and Injuries - genetics | Immunity, Humoral | Aged, 80 and over | Adult | Female | Retrospective Studies | Disease Models, Animal | Mice, Inbred C57BL | Gene Expression Regulation | Wounds and Injuries - immunology | Interleukin-33 - blood | Interleukin-5 - immunology | Animals | Interleukin-33 - metabolism | Aged | Mice | Shock, Hemorrhagic - immunology | Lung - immunology | Cohort Studies | Immune response | Interleukins | Analysis | Neutrophils | Pulmonary function tests | Research | Wounds and injuries | Nosocomial infection | Respiratory function | Authorship | Laboratories | Interleukin | Infections | Activation | Immunity | Hemorrhage | Experiments | Interleukin 4 | Cell activation | Interleukin 5 | Rodents | Surgery | Interleukin 1 | Nosocomial infections | Activation analysis | Medical research | Lymphoid cells | Cytokines | CXCR2 protein | Leukocytes (neutrophilic) | Inflammation | Interleukin 13 | Tumorigenicity | Roles | Patients | Trauma | Asthma | Studies | Immunosuppression | Injury prevention | Hospitals | Lungs | Correlation analysis | Plasmas (physics)
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, p. e69520
Journal Article
Journal Article
细胞研究:英文版, ISSN 1001-0602, 2017, Volume 27, Issue 7, pp. 916 - 932
Pancreatic ductal adenocarcinoma (PDAC) driven by oncogenic K-Ras remains among the most lethal human cancers despite recent advances in modern medicine. The... 
ONCOGENIC KRAS | HMGB1 | RAGE | CIRCULATING NUCLEOSOMES | INTRAEPITHELIAL NEOPLASIA | INFLAMMATORY LIVER-INJURY | MOUSE MODELS | CHROMOSOMAL INSTABILITY | CELL-DEATH | CELL BIOLOGY | pancreatic cancer | TO-DUCTAL METAPLASIA | K-Ras | ANIMAL-MODELS | MOBILITY GROUP BOX-1 | histone | Pancreatic Neoplasms - metabolism | Prognosis | Oxidative Stress | Tumor Suppressor Proteins - antagonists & inhibitors | Humans | Carcinoma, Pancreatic Ductal - metabolism | Carcinogenesis - metabolism | HMGB1 Protein - genetics | Carcinoma, Pancreatic Ductal - genetics | Neoplasm Metastasis | Glycyrrhizic Acid - pharmacology | Interleukin-6 - blood | HMGB1 Protein - metabolism | Tumor Suppressor Proteins - genetics | Neoplasms, Experimental | Telomere - metabolism | Chromosomal Instability | HMGB1 Protein - antagonists & inhibitors | Genes, ras - genetics | Telomere - genetics | Tumor Suppressor Proteins - metabolism | Mice, Inbred C57BL | Carcinogenesis - genetics | Kaplan-Meier Estimate | Nucleosomes - metabolism | Pancreatic Neoplasms - genetics | Mice, Knockout | Animals | Mice | DNA Damage | Cohort Studies | Adenocarcinoma | Pathogenesis | DNA damage | Interleukin | Antibodies | Activation | Macrophages | Chromosome rearrangements | HMGB1 protein | Neoplasms | K-Ras protein | Genomic instability | Metastases | Interleukin 6 | Cell activation | Rodents | Tumorigenesis | Inhibition | Lesions | Pancreas | Chromosomes | Deoxyribonucleic acid--DNA | Glycyrrhizin | Advanced glycosylation end products | Stability | Secretion | Abnormalities | Leukocytes (neutrophilic) | Pharmacology | Inflammation | Glycosylation | Survival | Ablation | Pancreatic cancer | Neutralizing | Nucleosomes | Tumor suppressor genes | Intracellular | Histone H3 | Tumors | Cancer | Original
Journal Article
2006, 8th ed., ISBN 0071105182, xxiii, 1320
Book
Gastroenterology, ISSN 0016-5085, 05/2018, Volume 154, Issue 6, pp. S-1363 - S-1363
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2013, Volume 8, Issue 12, p. e79804
The translation of in vitro findings to clinical outcomes is often elusive. Trauma/hemorrhagic shock (T/HS) results in hepatic hypoxia that drives... 
GENE-EXPRESSION DATA | LIVER-INJURY | CHEMOKINES | MULTIDISCIPLINARY SCIENCES | TRAUMA-HEMORRHAGE | TRANSCRIPTION | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | ORGAN DAMAGE | MICE | HYPOXIA | TRANSFORMED HEPATOCYTES | Humans | Middle Aged | Hepatocytes - pathology | Male | Hepatocytes - metabolism | Case-Control Studies | Inflammation - blood | Cell Hypoxia | Shock, Hemorrhagic - mortality | Aged, 80 and over | Adult | Female | Inflammation - mortality | Wounds and Injuries - blood | Survival Rate | Biomarkers - blood | Chemokine CCL2 - blood | Interleukin-1alpha - blood | Hep G2 Cells | Mice, Knockout | Shock, Hemorrhagic - blood | Wounds and Injuries - mortality | Disease-Free Survival | Animals | Aged | Mice | Physiological aspects | Development and progression | Inflammation | Genetic aspects | Research | Chemokine receptors | Cluster analysis | Liver | Hemorrhage | Inflammatory diseases | Proteins | Lysates | Signal transduction | Ischemia | Rodents | Network analysis | Surgery | Physiology | Bioindicators | Mathematical models | Statistical analysis | Cytokines | Vector quantization | Principal components analysis | Data processing | Gene expression | Clustering | Trauma | Medicine | Studies | Hepatocytes | Computer applications | Technology assessment | Biomarkers | Ventilation | Hypoxia | Chemokines | Monocyte chemoattractant protein 1
Journal Article