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1992, 2, CRC Press revivals, ISBN 9780849361098, 297
Hepatocyte and Kupffer Cell Interactions presents a comprehensive discussion of historical and recent information regarding this diverse field of research. The... 
Pathophysiology | Liver cells | Liver | Kupffer cells | Immunology | Gastroenterology | Cell Biology
Book
The New England journal of medicine, ISSN 1533-4406, 07/2018, Volume 379, Issue 4, pp. 315 - 326
Journal Article
Critical care medicine, ISSN 0090-3493, 08/2013, Volume 41, Issue 8, pp. 2008 - 2014
Journal Article
PloS one, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, pp. e69520 - e69520
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 02/2013, Volume 110, Issue 9, pp. 3507 - 3512
Journal Article
Cell research, ISSN 1001-0602, 2017, Volume 27, Issue 7, pp. 916 - 932
Pancreatic ductal adenocarcinoma (PDAC) driven by oncogenic K-Ras remains among the most lethal human cancers despite recent advances in modern medicine. The... 
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Pancreatic Neoplasms - metabolism | Prognosis | Oxidative Stress | Tumor Suppressor Proteins - antagonists & inhibitors | Humans | Carcinoma, Pancreatic Ductal - metabolism | Carcinogenesis - metabolism | HMGB1 Protein - genetics | Carcinoma, Pancreatic Ductal - genetics | Neoplasm Metastasis | Glycyrrhizic Acid - pharmacology | Interleukin-6 - blood | HMGB1 Protein - metabolism | Tumor Suppressor Proteins - genetics | Neoplasms, Experimental | Telomere - metabolism | Chromosomal Instability | HMGB1 Protein - antagonists & inhibitors | Genes, ras - genetics | Telomere - genetics | Tumor Suppressor Proteins - metabolism | Mice, Inbred C57BL | Carcinogenesis - genetics | Kaplan-Meier Estimate | Nucleosomes - metabolism | Pancreatic Neoplasms - genetics | Mice, Knockout | Animals | Mice | DNA Damage | Cohort Studies | Adenocarcinoma | Pathogenesis | DNA damage | Interleukin | Antibodies | Macrophages | Chromosome rearrangements | HMGB1 protein | Neoplasms | K-Ras protein | Genomic instability | Metastases | Interleukin 6 | Cell activation | Rodents | Tumorigenesis | Inhibition | Lesions | Pancreas | Chromosomes | Deoxyribonucleic acid--DNA | Glycyrrhizin | Advanced glycosylation end products | Stability | Abnormalities | Leukocytes (neutrophilic) | Pharmacology | Inflammation | Glycosylation | Survival | Ablation | Pancreatic cancer | Neutralizing | Nucleosomes | Tumor suppressor genes | Intracellular | Histone H3 | Tumors | Cancer | Index Medicus | pancreatic cancer | HMGB1 | RAGE | histone | K-Ras | Original
Journal Article
American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, ISSN 0363-6119, 12/2009, Volume 297, Issue 6, pp. 1670 - 1680
Journal Article