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Journal of Neurochemistry, ISSN 0022-3042, 07/2008, Volume 106, Issue 1, pp. 475 - 485
Recent findings suggest that hypercholesterolemia may contribute to the onset of Alzheimer’s disease‐like dementia but the underlying mechanisms remain... 
cholesterol | low density lipoprotein receptor | beta‐site APP cleaving enzyme 1 | amyloid precursor protein processing | neuroinflammation | working memory | Neuroinflammation | Low density lipoprotein receptor | Working memory | Beta-site APP cleaving enzyme 1 | Amyloid precursor protein processing | Cholesterol | BETA-SECRETASE | NEURODEGENERATIVE DISEASE | MICROGLIAL ACTIVATION | LEARNING-DEFICITS | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | HYPERCHOLESTEROLEMIC RABBITS | TRANSGENIC MODEL | NEUROSCIENCES | MOUSE MODEL | RADIAL-ARM MAZE | beta-site APP cleaving enzyme 1 | DENSITY-LIPOPROTEIN RECEPTOR | Alzheimer Disease - etiology | Dietary Fats - metabolism | Memory Disorders - physiopathology | Neurons - pathology | Memory Disorders - metabolism | Brain - metabolism | Encephalitis - physiopathology | Encephalitis - metabolism | Up-Regulation - physiology | Amyloid beta-Protein Precursor - metabolism | Neurons - metabolism | Disease Models, Animal | Receptors, LDL - genetics | Alzheimer Disease - physiopathology | Dietary Fats - adverse effects | Cytokines - metabolism | Amyloid beta-Peptides - biosynthesis | Brain - physiopathology | Mice, Inbred C57BL | Cholesterol - metabolism | Peptide Fragments - biosynthesis | Mice, Knockout | Amyloid Precursor Protein Secretases - metabolism | Animals | Maze Learning | Memory Disorders - etiology | Aspartic Acid Endopeptidases - metabolism | Alzheimer Disease - metabolism | Hypercholesterolemia - complications | Hypercholesterolemia - physiopathology | Mice | Memory, Short-Term | Hypercholesterolemia - metabolism | Encephalitis - etiology | Hypercholesterolemia | Tumor necrosis factor | Amyloid beta-protein | Short-term memory | Alzheimer's disease | Neurology | Biochemistry | Alzheimers disease | Rodents | Memory | Index Medicus | BACE1 | APP processing | LDLR
Journal Article
Behavioural Brain Research, ISSN 0166-4328, 2011, Volume 221, Issue 2, pp. 610 - 622
Memantine is a partial NMDA receptor antagonist that has been shown to improve learning and memory in several animal models, and is approved for the treatment... 
Cholinergic neuron | BDNF | Radial arm maze | Brain derived growth factor | Inflammation | Novel object recognition | Alzheimer's disease | Hippocampus | OBJECT RECOGNITION MEMORY | ALZHEIMERS-DISEASE | NEUROTROPHIC FACTOR | AMYLOID PRECURSOR PROTEIN | NEUROSCIENCES | MESSENGER-RNA LEVELS | NERVE GROWTH-FACTOR | BASAL FOREBRAIN NEURONS | MOUSE MODEL | NMDA RECEPTOR ANTAGONIST | BEHAVIORAL SCIENCES | TRANSGENIC MICE | Excitatory Amino Acid Antagonists - therapeutic use | Chromosomes, Mammalian - genetics | Motor Activity - drug effects | Male | Brain - metabolism | Mice, Mutant Strains | Brain-Derived Neurotrophic Factor - metabolism | Trisomy - pathology | Memantine - administration & dosage | Cholinergic Fibers - drug effects | Memory Disorders - pathology | Disease Models, Animal | Down Syndrome - pathology | Memantine - pharmacology | Drug Administration Schedule | Excitatory Amino Acid Antagonists - administration & dosage | Excitatory Amino Acid Antagonists - pharmacology | Maze Learning - drug effects | Cholinergic Fibers - pathology | Brain - drug effects | Animals | Memory Disorders - drug therapy | Recognition (Psychology) - drug effects | Brain - pathology | Down Syndrome - drug therapy | Mice | Memantine - therapeutic use | Down Syndrome - psychology | Nerve Degeneration - drug therapy | Methyl aspartate | Neurosciences | Neurons | Analysis | Memory | Down syndrome | Nervous system agents | Amyloid beta-protein | cholinergic neuron | Alzheimer’s disease | hippocampus | inflammation | novel object recognition | radial arm maze | brain derived growth factor
Journal Article
Annals of Biomedical Engineering, ISSN 0090-6964, 07/2017, Volume 45, Issue 7, p. 1697
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1007/s10439-017-1859-8 With... 
Drugs | Phenols | Drug delivery systems | Estradiol | Menopause | Vehicles | Encapsulation | Therapy | Ovariectomy | Hyperplasia | Cognitive ability | Central nervous system | Risk reduction | 17β-Estradiol | Pharmacology | Hydrophobicity | Chemical compounds | Molecular weight | Polylactide-co-glycolide | Carriers | Engineering | Sex hormones | Uterus | Glycolic acid | Attenuation | Hormone replacement therapy | Endometrium
Journal Article
03/2015, ISBN 1493921592
Book
Neuromethods, ISSN 0893-2336, 2015, Volume 94, pp. 3 - 36
Journal Article
Neuromethods, ISSN 0893-2336, 2015, Volume 94, pp. 421 - 427
Journal Article
Annals of biomedical engineering, ISSN 0090-6964, 7/2017, Volume 45, Issue 7, pp. 1697 - 1709
With menopause, circulating levels of 17β-estradiol (E2) markedly decrease. E2-based hormone therapy is prescribed to alleviate symptoms associated with... 
polymer | (3–10): 17β-estradiol | PLGA | cognition | nanoparticle | drug delivery | microparticle
Journal Article
Neuromethods, ISSN 0893-2336, 2015, Volume 94, pp. 441 - 449
Journal Article
Neuromethods, ISSN 0893-2336, 2015, Volume 94, pp. 451 - 454
Journal Article
Neuromethods, ISSN 0893-2336, 2015, Volume 94, pp. 407 - 410
Journal Article
Neuromethods, ISSN 0893-2336, 2015, Volume 94, pp. 411 - 419
Journal Article
Journal Article
Endocrinology, ISSN 1945-7170, 01/2019, Volume 160, Issue 2, pp. 461 - 462
Journal Article
PLoS genetics, ISSN 1553-7404, 04/2019, Volume 15, Issue 4, pp. e1008108 - e1008108
RASopathies are a family of related syndromes caused by mutations in regulators of the RAS/Extracellular Regulated Kinase 1/2 (ERK1/2) signaling cascade that... 
Phenotypes | Neurosciences | Funding | Astrocytes | Glial fibrillary acidic protein | Olig2 protein | Forebrain | Extracellular signal-regulated kinase | Nervous system | Cognition | Noonan's syndrome | Glial stem cells | Progenitor cells | Defects | Fear conditioning | Neurological diseases | Consortia | Autism | Myelination | Life sciences | Anxiety | Mutation | Supervision | Locomotor activity
Journal Article