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Journal of cell science, ISSN 0021-9533, 11/2018, Volume 131, Issue 21, p. jcs218313
The pro-inflammatory cytokine interleukin-15 (IL15) and its receptor a (IL15RA) participate in the regulation of musculoskeletal function and metabolism.... 
Myokine | IL15RA | Mitochondria | Microscopy | OPA1 | IL15 | Ultrastructure | Interleukin | Fatigue | Cardiolipin | Fiber type | HOMEOSTASIS | SUBPOPULATIONS | PROTEOME | AUTOPHAGY | INTERLEUKIN-15 RECEPTOR | CELL BIOLOGY | SKELETAL-MUSCLE | IL-15R-ALPHA DEFICIENCY | ENDURANCE EXERCISE | MODULATION | 132
Journal Article
Cancer Research, ISSN 0008-5472, 07/2017, Volume 77, Issue 13, pp. 3391 - 3405
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2015, Volume 308, Issue 1, pp. H39 - H48
Pan S, Wang N, Bisetto S, Yi B, Sheu SS. Downregulation of adenine nucleotide translocator 1 exacerbates tumor necrosis factor -mediated cardiac inflammatory... 
Adenine nucleotide translocator | Sepsis | Inflammation | Cytokine | cytokine | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | HEART-FAILURE | DILATED CARDIOMYOPATHY | KAPPA-B | MYOCARDIAL DEPRESSION | adenine nucleotide translocator | inflammation | ENDOTHELIAL-CELLS | SEPTIC SHOCK | MITOCHONDRIAL DYSFUNCTION | PERIPHERAL VASCULAR DISEASE | TNF-ALPHA | EXPRESSION | sepsis | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Mitochondria, Heart - metabolism | Reactive Oxygen Species - metabolism | Mitochondria, Heart - pathology | Myocytes, Cardiac - immunology | Male | NF-kappa B - metabolism | Inflammation - blood | Inflammation - metabolism | Transfection | RNA Interference | Time Factors | Inflammation Mediators - metabolism | Membrane Potential, Mitochondrial | Mitochondrial Swelling | Interleukin-6 - metabolism | Genes, Reporter | Disease Models, Animal | Cell Line | Adenine Nucleotide Translocator 1 - genetics | Interleukin-6 - genetics | Down-Regulation | Mice, Inbred C57BL | Adenine Nucleotide Translocator 1 - metabolism | Inflammation - immunology | Mitochondria, Heart - immunology | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | NF-kappa B - genetics | Lipopolysaccharides - pharmacology | Inflammation - genetics | Myocytes, Cardiac - metabolism | Signaling and Stress Response
Journal Article
American Journal of Physiology, ISSN 0363-6135, 01/2015, Volume 308, Issue 1, p. H39
  Inflammation contributes significantly to cardiac dysfunction. Although the initial phase of inflammation is essential for repair and healing, excessive... 
Mitochondria | Matrix | Cardiovascular disease | Inflammatory diseases | Cells | Tumors
Journal Article
American Journal of Physiology: Heart and Circulatory Physiology, ISSN 0363-6135, 01/2015, Volume 308, Issue 1, pp. H39 - H39
Inflammation contributes significantly to cardiac dysfunction. Although the initial phase of inflammation is essential for repair and healing, excessive... 
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 06/2016, Volume 95, pp. 308 - 322
We investigated the mechanism of selective ascorbate-induced cytotoxicity in tumor cells, including Hep G2 cells, compared to primary hepatocytes. H O... 
Glucose oxidase | Hydrogen peroxide | Sorafenib | Hep G2 | Vitamin C | Hepatocellular carcinoma | Ascorbate | Synergy | Calcium homeostasis | Mitochondrial membrane potential | Mitochondrial membranepotential | Hydrogenperoxide | VITAMIN-C PHARMACOKINETICS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLUTATHIONE-PEROXIDASE | PANCREATIC-CANCER | INTRACELLULAR HYDROGEN-PEROXIDE | RAF/MEK/ERK PATHWAY | SUPPLEMENTAL ASCORBATE | CELL-DEATH | REDOX REGULATION | TERMINAL HUMAN CANCER | SUPPORTIVE TREATMENT | ENDOCRINOLOGY & METABOLISM | Niacinamide - analogs & derivatives | Apoptosis - drug effects | Humans | Liver Neoplasms - drug therapy | Ascorbic Acid - administration & dosage | Mitochondria - metabolism | Mitochondria - drug effects | Hep G2 Cells | Hydrogen Peroxide - metabolism | Niacinamide - administration & dosage | Drug Synergism | Carcinoma, Hepatocellular - drug therapy | Phenylurea Compounds - administration & dosage | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Liver Neoplasms - pathology | Cell Proliferation - drug effects | Carcinoma, Hepatocellular - metabolism | Oxidases | Hepatoma | Metastasis | Glucose | Antineoplastic agents | Cells | Dextrose | Antimitotic agents | Chemotherapy | Cell death | Analysis | Gastrointestinal diseases | Kidney diseases | Cancer | Medical research | Medicine, Experimental | Peroxidase | synergy | sorafenib | vitamin C | glucose oxidase | hydrogen peroxide | mitochondrial membrane potential | ascorbate | calcium homeostasis | hepatocellular carcinoma
Journal Article
Cancer Research, ISSN 0008-5472, 02/2016, Volume 76, Issue 3 Supplement, pp. B10 - B10
Journal Article
Cancer Research, ISSN 0008-5472, 04/2013, Volume 73, Issue 8 Supplement, pp. LB-133 - LB-133
Journal Article
Journal Article
American Journal of Physiology, ISSN 0363-6135, 12/2013, Volume 305, Issue 12, p. H1736
  Ca+ influx to mitochondria is an important trigger for both mitochondrial dynamics and ATP generation in various cell types, including cardiac cells.... 
Musculoskeletal system | Mitochondria | Calcium | Cardiomyocytes | Gene expression | Cells
Journal Article
American Journal of Physiology: Heart and Circulatory Physiology, ISSN 0363-6135, 12/2013, Volume 305, Issue 12, pp. H1736 - H1736
Ca+ influx to mitochondria is an important trigger for both mitochondrial dynamics and ATP generation in various cell types, including cardiac cells.... 
ATP
Journal Article
American Journal of Physiology (Consolidated), ISSN 0002-9513, 01/2015, Volume 308, Issue 1, p. H39
Inflammation contributes significantly to cardiac dysfunction. Although the initial phase of inflammation is essential for repair and healing, excessive... 
Purines | Adenylic acid | Tumor necrosis factor | Health aspects
Journal Article
The American Journal of Physiology, ISSN 0002-9513, 12/2013, Volume 305, Issue 6, p. H1736
[Ca.sup.+] influx to mitochondria is an important trigger for both mitochondrial dynamics and ATP generation in various cell types, including cardiac cells.... 
Calcium channels | Heart cells | Physiological aspects | Genetic aspects | Research | Gene expression | Adenosine triphosphate
Journal Article
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