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1989, 2. ed. ampliata., Collana di filosofia, ISBN 9788878266025, 144
Book
NATURE COMMUNICATIONS, ISSN 2041-1723, 07/2019, Volume 10, Issue 1, pp. 3196 - 15
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 2014, Volume 20, Issue 6, pp. 599 - 606
Journal Article
EMBO reports, ISSN 1469-221X, 06/2019, Volume 20, Issue 6, p. n/a
The antiviral response is a tightly regulated process that follows specific patterns of timing and duration. The type I interferon‐induced antiviral pathway is... 
YAP | TAZ | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | Stat2 protein | Translocation | Transcription | Gene expression | Nuclear transport | Proteins | Signal transduction | Signaling | Interferon | Autoimmune diseases | Stat1 protein | Protein interaction | Dimerization
Journal Article
Methods in Molecular Biology, ISSN 1064-3745, 2016, Volume 1379, pp. 21 - 29
microRNAs are 22-nucleotide-long double-strand small RNAs, able to modulate gene expression at posttranscriptional level, degrading mRNA and/or impairing... 
Fresh tissues | RNA extraction | Mesothelioma | MicroRNA | Thymoma | HNSCC | Neoplasms - prevention & control | MicroRNAs - isolation & purification | MicroRNAs - genetics | Chemical Fractionation - methods | Chemoprevention | Humans | Index Medicus
Journal Article
The EMBO Journal, ISSN 0261-4189, 05/2015, Volume 34, Issue 9, pp. 1214 - 1230
Journal Article
Scientific Reports, ISSN 2045-2322, 02/2017, Volume 7, Issue 1, pp. 43013 - 43013
Combined MAPK/PI3K pathway inhibition represents an attractive, albeit toxic, therapeutic strategy in oncology. Since PTEN lies at the intersection of these... 
LUNG-CANCER | METASTATIC MELANOMA | SIGNALING PATHWAYS | RAS/RAF/MEK/ERK | MULTIDISCIPLINARY SCIENCES | DEREGULATION | PI3K/PTEN/AKT/MTOR | COMBINATION | NF-KAPPA-B | MAPK PATHWAY | STEM-CELL POPULATION | Neoplastic Stem Cells - cytology | TOR Serine-Threonine Kinases - metabolism | Neoplastic Stem Cells - drug effects | Humans | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | TOR Serine-Threonine Kinases - antagonists & inhibitors | Janus Kinase 1 - metabolism | PTEN Phosphohydrolase - antagonists & inhibitors | Neoplastic Stem Cells - metabolism | RNA Interference | Janus Kinase 1 - genetics | Female | Pyrimidinones - pharmacology | MAP Kinase Kinase Kinases - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | PTEN Phosphohydrolase - genetics | Everolimus - pharmacology | PTEN Phosphohydrolase - metabolism | MAP Kinase Kinase Kinases - metabolism | Drug Synergism | Animals | Mice, Nude | Cell Line, Tumor | Mice | Protein Kinase Inhibitors - pharmacology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Pyridones - pharmacology | RNA, Small Interfering - metabolism | TOR protein | Stat3 protein | MAP kinase | Janus kinase | mRNA | Tumor cell lines | PTEN protein | Cancer | 1-Phosphatidylinositol 3-kinase | Index Medicus
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 05/2017, Volume 18, Issue 5, p. 961
Journal Article
Journal of Molecular Medicine, ISSN 0946-2716, 6/2012, Volume 90, Issue 6, pp. 667 - 679
Journal Article
2016, ISBN 9782889199617
TP53 gene mutations are present in more than half of all human cancers. The resulting proteins are mostly full-length with a single amino acid change and are... 
mouse models | mutant p53 | dominant netagive | therapies | Oncogenic addiction | gain of function
eBook