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Lancet Oncology, The, ISSN 1470-2045, 2013, Volume 14, Issue 6, pp. 472 - 480
Summary Background Various human cancers have ALK gene translocations, amplifications, or oncogenic mutations, such as anaplastic large-cell lymphoma,... 
Hematology, Oncology and Palliative Medicine | LUNG-CANCER | FUSION | GENE | ONCOLOGY | ADOLESCENTS | RESISTANCE | ALK KINASE | ONCOGENIC MUTATIONS | IDENTIFICATION | ACTIVATING MUTATIONS | CHEMOTHERAPY | Pyrazoles - therapeutic use | Age Factors | Humans | Child, Preschool | Pyridines - pharmacokinetics | Infant | Male | Antineoplastic Agents - therapeutic use | Lymphoma, Large-Cell, Anaplastic - enzymology | Antineoplastic Agents - administration & dosage | Protein Kinase Inhibitors - adverse effects | Molecular Targeted Therapy | Young Adult | Pyridines - adverse effects | Neoplasms - genetics | Time Factors | Antineoplastic Agents - adverse effects | Biomarkers, Tumor - metabolism | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Female | Antineoplastic Agents - pharmacokinetics | Biomarkers, Tumor - antagonists & inhibitors | Pyrazoles - pharmacokinetics | Child | Lymphoma, Large-Cell, Anaplastic - genetics | Pyrazoles - adverse effects | Pyridines - therapeutic use | Protein Kinase Inhibitors - pharmacokinetics | Drug Administration Schedule | Neoplasms - enzymology | Treatment Outcome | Receptor Protein-Tyrosine Kinases - metabolism | Disease Progression | Neoplasms - drug therapy | Protein Kinase Inhibitors - administration & dosage | Drug Resistance, Neoplasm - genetics | Maximum Tolerated Dose | Receptor Protein-Tyrosine Kinases - genetics | Protein Kinase Inhibitors - therapeutic use | Lymphoma, Large-Cell, Anaplastic - drug therapy | Adolescent | Biomarkers, Tumor - genetics | Lymphoma, Large-Cell, Anaplastic - pathology | Mutation | Neoplasms - pathology | Consortia | Pediatrics | Lymphomas | Safety and security measures | Lung cancer, Non-small cell
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