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by Chatrchyan, S and Khachatryan, V and Sirunyan, A.M and Tumasyan, A and Adam, N and Adam, W and Aguilo, E and Bergauer, T and Dragicevic, M and Erö, J and Fabjan, C and Friedl, M and Frühwirth, R and Ghete, V.M and Hammer, J and Hoch, M and Hörmann, N and Hrubec, J and Jeitler, M and Kiesenhofer, W and Knünz, V and Krammer, M and Krätschmer, I and Liko, D and Majerotto, W and Mikulec, I and Pernicka, M and Rahbaran, B and Rohringer, H and Rohringer, C and Schöfbeck, R and Strauss, J and Szoncsó, F and Taurok, A and Waltenberger, W and Walzel, G and Widl, E and Wulz, C.-E and Chekhovsky, V and Emeliantchik, I and Litomin, A and Makarenko, V and Mossolov, V and Shumeiko, N and Solin, A and Stefanovitch, R and Suarez Gonzalez, J and Fedorov, A and Korzhik, M and Missevitch, O and Zuyeuski, R and Bansal, M and Bansal, S and Beaumont, W and Cornelis, T and De Wolf, E.A and Druzhkin, D and Janssen, X and Luyckx, S and Mucibello, L and Ochesanu, S and Roland, B and Roland, C and Roland, G and Rougny, R and Selvaggi, G and Selvaggi, M and Staykova, Z and Van Haevermaet, H and Van Mechelen, P and Van Remortel, N and Van Spilbeeck, A and Blekman, F and Blyweert, S and DʼHondt, J and Devroede, O and Gonzalez Suarez, R and Goorens, R and Kalogeropoulos, A and Maes, M and Olbrechts, A and Tavernier, S and Van Doninck, W and Van Lancker, L and Van Mulders, P and Van Onsem, G.P and Villella, I and Clerbaux, B and De Lentdecker, G and Dero, V and Dewulf, J.P and Gay, A.P.R and Hreus, T and Léonard, A and Marage, P.E and Mohammadi, A and Reis, T and Rugovac, S and Thomas, S and Thomas, M and ... and CMS Collaboration and Los Alamos National Lab. (LANL), Los Alamos, NM (United States) and Argonne National Lab. (ANL), Argonne, IL (United States) and Fermi National Accelerator Lab. (FNAL), Batavia, IL (United States) and Lawrence Livermore National Lab. (LLNL), Livermore, CA (United States)
Physics Letters B, ISSN 0370-2693, 09/2012, Volume 716, Issue 1, pp. 30 - 61
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2015, Volume 112, Issue 19, pp. 6080 - 6085
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2010, Volume 107, Issue 46, pp. 19967 - 19972
Toll-like receptors (TLRs) trigger intestinal inflammation when the epithelial barrier is breached by physical trauma or pathogenic microbes. Although it has... 
Phenotypes | Inflammatory bowel diseases | Epithelial cells | Weight loss | Toll like receptors | Ligands | Inflammation | Mice | Colitis | Genetic mutation | Inflammatory bowel disease | Toll-like receptor signaling | Intestinal homeostasis | ENU mutagenesis | Epidermal growth factor receptor signaling | CHRONIC INTESTINAL INFLAMMATION | CROHNS-DISEASE | MULTIDISCIPLINARY SCIENCES | INJURY | ALPHA | intestinal homeostasis | inflammatory bowel disease | epidermal growth factor receptor signaling | INFLAMMATORY-BOWEL-DISEASE | TOLL-LIKE RECEPTOR-4 | INCREASED SUSCEPTIBILITY | SULFATE-INDUCED COLITIS | MUTATION | INNATE IMMUNITY | Dextran Sulfate | Adaptor Proteins, Vesicular Transport - deficiency | Glycoproteins - metabolism | Epiregulin | Amphiregulin | Intercellular Signaling Peptides and Proteins - metabolism | Receptor, Epidermal Growth Factor - metabolism | EGF Family of Proteins | Colitis - chemically induced | Toll-Like Receptors - metabolism | Metagenome - drug effects | Mice, Inbred C57BL | Epidermal Growth Factor - metabolism | Mutation - genetics | Hematopoietic System - cytology | Phenotype | Animals | Myeloid Differentiation Factor 88 - deficiency | Signal Transduction - drug effects | Colitis - metabolism | Lipopolysaccharides - pharmacology | Colitis - prevention & control | Myeloid Differentiation Factor 88 - metabolism | Ligands (Biochemistry) | Development and progression | Cellular signal transduction | Genetic aspects | Research | Properties | Human immunogenetics | Proteins | Genotype & phenotype | Cellular biology | Rodents | Mutation | Gene expression | Index Medicus | Biological Sciences
Journal Article
Nature Immunology, ISSN 1529-2908, 07/2012, Volume 13, Issue 7, pp. 642 - 650
Journal Article
Science, ISSN 0036-8075, 1/2012, Volume 335, Issue 6065, pp. 229 - 232
Innate immune responses are vital for pathogen defense but can result in septic shock when excessive. A key mediator of septic shock is tumor necrosis factor-α... 
Pathology | Splenocytes | Liver | REPORTS | Septic shock | Innate immunity | Immunoblotting | Infections | Gene expression regulation | Cellular immunity | Research universities | RHEUMATOID-ARTHRITIS | CELLS | MONOCYTOGENES | DISINTEGRIN | METALLOPROTEINASE | MULTIDISCIPLINARY SCIENCES | NECROSIS-FACTOR-ALPHA | MICE | INFECTION | MATURATION | FAMILY | ADAM17 Protein | Tumor Necrosis Factor-alpha - metabolism | Tumor Necrosis Factor-alpha - blood | Tumor Necrosis Factor-alpha - genetics | Molecular Sequence Data | Listeria monocytogenes - immunology | Shock, Septic - immunology | Lipopolysaccharides - immunology | Shock, Septic - metabolism | Base Sequence | Gene Deletion | Listeriosis - immunology | Carrier Proteins - chemistry | Cell Membrane - metabolism | Listeria monocytogenes - physiology | B-Lymphocytes - metabolism | Macrophages - immunology | Amino Acid Sequence | Cell Line | Listeriosis - metabolism | Macrophages, Peritoneal - immunology | Spleen - cytology | Immunity, Innate | Protein Transport | Carrier Proteins - genetics | ADAM Proteins - metabolism | Macrophages - metabolism | Animals | B-Lymphocytes - immunology | Carrier Proteins - metabolism | Macrophages, Peritoneal - microbiology | Listeriosis - pathology | Listeriosis - microbiology | Mice | ADAM Proteins - genetics | Macrophages, Peritoneal - metabolism | Prevention | Immune response | Listeria | Properties | Tumor necrosis factor | Index Medicus | Cell surface
Journal Article