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Nature Reviews Nephrology, ISSN 1759-5061, 08/2016, Volume 12, Issue 8, pp. 449 - 450
Journal Article
by Boor, P
NATURE REVIEWS NEPHROLOGY, ISSN 1759-5061, 01/2020, Volume 16, Issue 1, pp. 4 - 6
Once confined to the world of science fiction, advances in information technology, particularly in computational and storage resources, have enabled use of... 
UROLOGY & NEPHROLOGY
Journal Article
Metabolic Brain Disease, ISSN 0885-7490, 6/2018, Volume 33, Issue 3, pp. 961 - 967
Journal Article
Nature Genetics, ISSN 1061-4036, 09/2013, Volume 45, Issue 9, pp. 1067 - 1072
Journal Article
1998, Geschichte der deutschen Literatur von den Anfängen bis zur Gegenwart, ISBN 9783406441042, Volume Bd. 9, 1, xix, 825
Book
by Boor, P
NATURE REVIEWS NEPHROLOGY, ISSN 1759-5061, 08/2016, Volume 12, Issue 8, pp. 449 - 449
A new study reports an independent association between albuminuria and skin capillary rarefaction in a population-based cohort. These findings suggest that as... 
II DIABETIC-PATIENTS | UROLOGY & NEPHROLOGY | HYPERTENSION | TRANSCAPILLARY ESCAPE RATE | MICROALBUMINURIA | Complications and side effects | Blood circulation disorders | Care and treatment | Albuminuria | Diagnosis | Research | Risk factors
Journal Article
Expert Reviews in Molecular Medicine, ISSN 1462-3994, 2018, Volume 20, p. e1
Journal Article
Kidney International, ISSN 0085-2538, 07/2012, Volume 82, Issue 2, pp. 132 - 135
Most progressive and severe acute kidney diseases lead to renal fibrosis, for which we still lack effective treatment options. Prostaglandin E (PGE ) is one of... 
UROLOGY & NEPHROLOGY | RECEPTORS | KIDNEY | PROSTAGLANDIN E-2 | OBSTRUCTION | Dinoprostone - metabolism | Animals | Epithelial Cells - metabolism | Kidney Tubules - metabolism | Kidney Diseases - prevention & control | Receptors, Prostaglandin E, EP4 Subtype - metabolism
Journal Article
The Journal of Pathology, ISSN 0022-3417, 11/2012, Volume 228, Issue 3, pp. 286 - 299
Kidney diseases impart a vast burden on affected individuals and the overall health care system. Progressive loss of renal parenchymal cells and functional... 
Notch receptor | proliferation | inflammation | kidney diseases | fibrosis | tubular cells | chemotaxis | DIABETIC-NEPHROPATHY | PATHOLOGY | PODOCYTES | UNILATERAL URETERAL OBSTRUCTION | ATTENUATES RENAL FIBROSIS | ONCOLOGY | SIGNALING PATHWAY | DISEASE | MICE | MUTATIONS | EXPRESSION | DEVELOPING MOUSE KIDNEY | Cell Proliferation | Kidney - pathology | Humans | Receptors, Notch - genetics | Ureteral Obstruction - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Kidney - metabolism | Nephritis, Interstitial - metabolism | Nephritis, Interstitial - pathology | Jagged-2 Protein | Female | Membrane Proteins - metabolism | Receptors, Notch - deficiency | Kidney - physiopathology | Disease Models, Animal | Cell Line | Mice, Inbred C57BL | Nephritis, Interstitial - physiopathology | Rats | Receptors, Notch - physiology | Ureteral Obstruction - physiopathology | Mice, Knockout | Up-Regulation - drug effects | Transforming Growth Factor beta - pharmacology | Animals | Biopsy | Ureteral Obstruction - pathology | Fibrosis | Receptor, Notch3 | Signal Transduction - physiology | Mice | In Vitro Techniques | Receptor, Notch1 - physiology | Inflammation - physiopathology | Health care | Kidneys | Interstitial cells | Animal diseases | Activation | Chemotactic response | Ablation | Diseases | Monocytes | Receptors | Injury prevention | Collagen | Fibroblasts | Infiltration | Notch protein | Kidney diseases | Receptor mechanisms
Journal Article
Molecular Aspects of Medicine, ISSN 0098-2997, 02/2019, Volume 65, pp. 16 - 36
Journal Article